I am so sorry you had to go through this, and for others and more coming, you said it right, my friend. Stay safe. We see the pain, they do not and we care. When they are out in the cold, and can't find their way home, shall we be there to assist, You bet! the difference between them and us. We would even help them, with a wary eye of course!
......."but you put your trust in whatever you want to call your higher power and I guarantee you that you will not feel the pain. Not only that, but you will be called upon to help those who do and you will do it. It's just that simple.
We are the enlightened and we are chosen to make a difference and we are going to do just that."
Cowden protocol: Magnesium>?? According to Dr. Stephen Fry, magnesium feeds the protozoan in Morgellons, FL1953 Protomyxzoa, so, may help Lymes but not Morgellons. The spirochete may be destroyed but not the myxzoa.
We do need something though to break down the films forming the filaments. NAC does work as paste to breakup the biofilm and callous. Waiting for finds on how NAC works internally.
Cowden protocol: Magnesium>?? According to Dr. Stephen Fry, magnesium feeds the protozoan in Morgellons, FL1953 Protomyxzoa, so, may help Lymes but not Morgellons. The spirochete may be destroyed but not the myxzoa.
We do need something though to break down the films forming the filaments. NAC does work as paste to breakup the biofilm and callous. Waiting for finds on how NAC works internally.
Seems like a catch 22 doesn't it? the critters take our magnesium and B vitamins (and prolly tons of other things yet to be determined) which makes us sicker. Then we take supplements to replenish and that feeds them.
What we need is a stealth nutrient that only benefits us and not the organism. Can't they design a trojan horse vitamin that when the organism injests it, it dies? I think they can.
I think they can find a "trojan horse" that could work. All they have to find is the genetic makeup of the organisms and the biofilm. All these libraries with all these genes from everything, and the dna, they should be able to target the organism, by its genome. It surely can be genomed. If the the human can be, than certainly there should be a way to genome the organism, but, no one will do it. However, Doc Fry, is looking in that direction, as I think some others are too.
it should be quite easy, unless, it is part chemical reaction and transposons with inorganic material. Then, the inorganic would have to be identified as well. I wonder if the Diffusion/Perfusion is a process, in the human body. Actually, creating the organsims, and that is why they may be different organisms, from a protocell or multicell that perform any function.
A couple of scientists think that diffusion and perfusion have something to do with the cells that are infiltrated, with a process of some kind. Even so, I would think that chemists, and physicists and biologists could narrow this down. But, maybe pharma does not want too. Wait until it is a full fledged syndrome, then they will look at it, for their "coffers" of course. So, pharma, in my mind, is holding off, till they have the surveys done of what their "created" little organisms do.
diffusion Diffusion and the Human Body The dependence of life processes on diffusion mechanisms could not be more prevalent. Diffusion occurs throughout the human body, and without it, cells and body tissue could not get important nutrients for survival, the eyes would dry out, and many medicines could not be absorbed into the body.
The digestion of food in the stomach is only the first step in delivering important nutrients to living cells, which need energy to operate and nutrients to build more cells. The main system of transporting nutrients to the body's cells is through the blood stream. But once a nutrient arrives at a needy cell, it is still inside a blood vessel while the cell lies outside. The lack of nutrients inside the cell, and between the cell and the blood vessel, creates a concentration gradient between the blood vessel and the cell. Due to the lower concentration in the cell, the nutrient diffuses through the blood vessel wall and into the cell.
Every organ in the body needs oxygen in order to survive, and the eyes are no different. However, the eyes lack a great number of blood vessels (which carry oxygen that diffuses into cells by the process above), and must have an extra supply of oxygen. The atmosphere provides the extra needed oxygen, which is consumed by the eye by the diffusion of oxygen through the cornea, the hard outer covering on the eye. In places with a very dirty atmosphere, oxygen concentration may be low, and the eye can dry out. You may have noticed this if you have ever been in a dust storm or had smoke from a fire blowing into your eyes.
From severe illness to a common headache, medicines are universally used to alleviate pain or cure sickness around the world. For medicines taken orally as pills, the medicine must somehow find its way into the bloodstream. Once in the stomach, if the pill capsule is a time release mechanism, the medicine must first diffuse out of the capsule. Once in the stomach, the medicine from the pill is absorbed into the lining of the stomach and then into the bloodstream, both processes of diffusion. For some medicines, the capsule cannot provide enough long term dosage, or the medicine does not last long enough because it is absorbed too quickly. Another possibility is the IV, but for most people, the IV is not quite a feasible day-to-day method of medicine intake. A solution to this problem, one that has seen increasing use over the past few years is the transdermal patch.
The transdermal patch consists of an adhesive layer that attaches it to the skin, and a reservoir that holds the medicine. The medicine must first diffuse out of the reservoir and onto the skin, and then through the skin and into the bloodstream. Since diffusion through the skin is a much slower process than diffusion through the stomach lining and into the bloodstream, and, since the patch reservoir is capable of holding a greater quantity of medicine than a pill capsule, the transdermal patch offers a method for increased dosage over a prolonged period of time. Click here for a pictorial representation of a transdermal patch. www.sv.vt.edu/classes/MSE2034_NoteBook/MSE2034_kriz_NoteBook/diffusion/apps/body.html -------------------------------------------------------- Perfusion: perfusion /per·fu·sion/ (-zhun) 1. the act of pouring over or through, especially the passage of a fluid through the vessels of a specific organ. 2. a liquid poured over or through an organ or tissue.
per·fu·sion (pr-fyzhn) n. 1. The act of perfusing. 2. The injection of fluid into a blood vessel in order to reach an organ or tissues, usually to supply nutrients and oxygen.
tissue perfusion the circulation of blood through the vascular bed of tissue. ineffective tissue perfusion (specify type) (renal, cerebral, cardiopulmonary, gastrointestinal, peripheral) a nursing diagnosis accepted by the North American Nursing Diagnosis Association, defined as a state in which an individual has a decrease in oxygen resulting in failure to nourish the tissues at the capillary level. medical-dictionary.thefreedictionary.com/perfusion =================================
This could be the diffusion from the Protocell. Diffusion is normal in body chemistry, but if the protocell which could form anything, any organism would depend on how the body reacts to the diffusion process. More and more it appears a "protocell", a programmed protocell, is what is going on.
PACE, is the programmed artificial cell Evolution.
I am focusing on this right now. Just look at the cell, it has all kinds of components, yet it is artificial. If this diffused into say, our cytoskeletal cells, it could make a different kind of form, depending on which type cell or filament it went after, like microfibrils, intermediate fibers, or microtubules, actins etc.
It could effect us differently especially if chemical based. Which filament would it target? And could form a new type of organism, if the right DNA is put in the artificial cell. Could be distributed in the environment, like mobile cell element, just floats around.
These are "embedded" IT. Some say the filaments we have seem to have some kind of intelligence. Well, cells do to. So, I think they miss the process, like diffusion and perfusion and then these things create certain forms, by what they mutate. =========================
I am trying to find out what this protocell is made of. They say it is PNAs (peptide nucleuc acids, that act like RNA and DNA, and catalipids, which would involve the liposomes or the cell membranes.
This has to do with the channels or spaces in the body. So, the use of this is like a secondary system, a backup like system, but with the power to transmit some kind of signal, could be electrical magnetisim, or it could be phereomones, which are a natural process, but the cell would be artificial going into a natural membrance.
"Many of the properties of bilayer membranes composed of simple single-chain amphiphiles seem to be well-suited for a potential role as primitive cell membranes. However, the spontaneous formation of membranes from such amphiphiles is a concentration-dependent process in which a significant critical aggregate concentration (cac) must be reached. Since most scenarios for the prebiotic synthesis of fatty acids and related amphiphiles would result in dilute solutions well below the cac, the identification of mechanisms that would lead to increased local amphiphile concentrations is an important aspect of defining reasonable conditions for the origin of cellular life. Narrow, vertically oriented channels within the mineral precipitates of hydrothermal vent towers have previously been proposed to act as natural Clusius−dickel thermal diffusion columns, in which a strong transverse thermal gradient concentrates dilute molecules through the coupling of thermophoresis and convection. "..............
They are talking of hydrovents, with life and inorganic substances coming up diffusing into the ocean floor environment, Origin of Life, but they have created "protocells" of some sort for humans. However, if in human this is difused into the body liguids, then it would be in the "fluid channels of the body".
but, it looks like thermal (heat or cold) would be necessary as chemical reaction for this to take place. Even so, most doctors should realize if these are being used (the programmed artificial cells)and would look for abnormal findings, but, they are not looking.
Last Edit: Nov 17, 2012 21:18:16 GMT -5 by skyship
What are they doing exactly? I think the Royal Society needs to answer some burning questions! about "microfluidic cells" these can penetrate cell membranes, like most new DNa drugs. This diffusion on the lattice? and there is what is called the "microtrabecular" lattice in the extra cellular matrix of the human body, in the fluid areas, or between cells, like in serum, or water areas of cell and body. this lattice is "filament like" or hydrogel like, sometimes like geletin, but loosely connected.
Evolutionary self-organization in complex fluids
This paper explores the ability of molecular evolution to take control of collective physical phases, making the first decisive step from independent replicators towards cell-like collective structures. We develop a physical model of replicating combinatorial molecules in a ternary fluid of hydrocarbons, amphiphiles and water. Such systems are being studied experimentally in various laboratories to approach the synthesis of artificial cells, and are also relevant to the origin of cellular life. The model represents amphiphiles by spins on a lattice (with Ising coupling in the simplest case), coupled to replicating molecules that may diffuse on the lattice and react with each other.
,,,,,:It does appear that part of the functionality of a protocell is to provide a strong control of the mobility of genetic information. "..............So, this artificial cell could "read" and move genetic information? it seems!
They are at Academia's door, they are in the labs, almost, and pushing for patents on drugs, never tested, small groups, patent the creation by the students under thumb of hidden new tech science, and students, profs get no credit for the outcome. Academia let them in the door. Wrong move!
He was the one who started the reptilian conspiracy according to what Jesse Ventura found out and confronted him with. Well, ya can't be perfect on everything. But, they may be reptilian like.He does explain the concept of being "reptilian like", rigid, ritualistic etc. , Like what the Cathers knew. (gnostic:knowledge). Yet, they to me, were what one would call "open hearted". and yet appreciated the teachings of the Bible. Especially John the Baptist, and "little John" who wrote Revelations. So to me, they understood the "spirits of darkness" and the spirits of light and goodness" and knew who the "bringer of the false light was". To me, they were more than gnostics. They believed "through the heart".
A lot of what he writes is true about the Illuminati etc. I think he is trying to explain that one has to understand what perception is. We have to get back to the heart, I so agree. And to me that is where we find God as well, even though he thinks what is written in "those pages" as he says, is a lie. I do not think it all a lie, just parts, that make no sense, but then that could be my perception of what I think I am reading.
But, the creative side of life, is who we are. What we know is ourselves and no one else, that is what makes us unique, and that is where the "good light" is, in the heart, and for those who believe in God, that is also where Jesus resides, if we but ask him in and unchain the heart, let him in and then keep the light burning so the chains are not shackled around him and he is suppressed. We need to get out of the gut feelings-instant reaction, to the place where we know we come from. To thine own self be true, sort of thing.
So, I believe what he is saying is a fine line, but, one can add the 'true light" we have been given, rather than the one that "the light bringer" brought, like LUCIFERIN GENES etc..Bioilluminance from quantum dots etc...... So, there is more to what he says, I believe. I do not throw the Bible out, after all, it is a book, written by those who recorded events, so is "Hamlet", so is "Gone With The Wind", since whoever wrote these books, is human, then there may be some misunderstandings, yet these books are read over and over. Why? Because each time we read them or passages of them, we learn something new and the more we understand the human author.
Our perceptions have changed, and we see with new eyes. Which means we are operating from the heart, our own hearts which we have opened to let this "true light in". What happens is it becomes overshadowed by the darkness, pulling us into the low energy forces. We have to "pull ourselves back up again to the heart".
Great way to explain it as he has, just don't agree about his take on the Bible.
My 3 cents worth, still believing that it is not religion or dogma, but, all heart related, and the scriptures are meaningful, which is why we never turn away from others who are seeking comfort, love and caring in our Morgie communities. Our hearts know and understand our fellow sufferers, wherever they come from. That is unconditional!
Happy Thanksgiving everyone! We have much to be thankful for! We have free minds. We have free choice We have "free spirits" because we know where the light comes from. We have freedom, if we open our heart doors. We have light, if we take our shades off. We all have the capacity to love, and reach out to others, because we have hearts. This is what the dark knights cannot fathom.
The parent calls their children together and announces that one child was born special and the other kids were not. The special child would be treated with great favor, while the others must work tirelessly to compensate for being not-special.
Such a family's design results in nothing but trouble - division, confusion, dysfunction, rivalry, strife, control.
Before that parent came to town, all families - of all types - lived side by side - thriving in spiritual freedom.
But when that parent arrived, everything changed - for all families - even if they didn't live anywhere near them.
It spread like a dark cloud blanketing the planet, helped by salespeople and front men, sometimes called the sorcerer-kings, who moved in and took over - making the rules, collecting the money, and causing the suffering we have seen throughout history. And three distinct warring groups have carried that legacy forward.
But we deserve it - we're not-special - some of us even thrash ourselves. The parent requires perpetual adoration, appeasement, and sacrifices of - and by - the not-special ones.
My uncle made what is called the ultimate sacrifice - he died in war. He gave his life for a cause foisted upon him since birth.
The family's heartbreak was so deep that it carved holes in their hearts - voids that could never be filled - which carried down to through generations, changing lives. His sacrifice changed everything. This is common for the not-special commoners.
It's the ultimate sacrifice because heart energy is the most potent of all.
The sorrow and grief are infinite, while the monsters (movie - Monsters, Inc.) harvest the endless supply of energy created by that tragedy. Multiply that by many families and sacrifices throughout time, while the cauldron is continually stirred.
Neo, we are batteries - being drained and recharged until we wear out.
In finance, we are held in slavery by a system called monetized debt. In spirit, the not-special kids are also held in a system of debt. The special kids have a rule (literally) that they don't charge each other interest, while the not-special kids pay and pay and pay. And just who are they paying?
At its base, it's the same system, applied to different scenarios, by the same monsters. They have no debt because they concocted the system - and laugh at the not-special huddled masses.
If what we've been taught is true, then even monsters would be fearful of that parent - but they're not. Besides, if a parent was good, why would anyone need to fear them, the way we've been taught to?
Fear is the number one tool of the monsters - generated in our lower brain - the R-complex. Ditto, the batteries.
Were stories about that parent developed for this purpose? Or maybe they were changed long ago, for this purpose? Regardless, there's no denying they include contradictory, schizophrenic, psychopathic death and destruction, blood sacrifice, and using the not-special children as pawns.
So here we are - corruption of absolutely everything - with endless war, strife, etc. But that's OK. We've known (been programmed to believe) this would all happen, and some can't wait till the showdown of billions. Wow, what an energy harvest that will be!
Is that corruption so vast because we were tricked into believing in the wrong parent, while we've been feeding his ravenous appetite, giving him more and more strength? If the good parent cared or had power, why would he allow his kids to be treated this way for so long?
As a parent yourself, would you set up your family like that parent? If that "parent" told you to take the life of your child or neighbor or whatever, would you?
Why is this story readily accepted in religion, yet is so wrong to us in our hearts? Hearing voices in your head is fine for religion, but called schizophrenic in society. The sorcerer-kings now can even put those voices in your head electronically. It's our fault. We let them grow to this immense strength, by accepting what we were told - because the parent frowns on questioning by the not-special children.
We're told that we have but one parent. But that parent had only one son. No, we are all children - it's why we call him father. Wait - no, he had only one child, who cried out why have you forsaken me?
Wasn't that blood sacrifice supposed to pay the debt for all? Oh wait - no - yes - no - yes. Nope, we're in deeper debt doo-doo than ever.
Is the confusion on purpose? Not to pick on one group - I promise - but just as an example - why does the Vatican own two of the world's best telescopes - named Lucifer 1 and Lucifer 2?
There are volumes of information about those kinds of disconnects, but our lifelong programming masks our clear view - while the Lucifer scopes have the best views in the world - which aren't shared with anyone (including NASA) until they're picked through and modified. What are they looking for, anyway?
And after being told repeatedly that ETs don't exist, now the Pope tells us that ETs do not need absolution of original sin, because they have none. Apparently they're special kids, too. His latest statement is that Jesus wasn't born when we were told he was. Geesh, maybe he'll even tell us the truth - that we've been celebrating Saturnalia and not Christ's birth. (Big energy harvesting time, loved especially by the marketers and bankers.)
What did Michelangelo know? Why did he put horns on his Moses statue - resting comfortably in Rome? Look it up in Wiki, and you'll see that no one can even agree on that, let alone any other part of this incredible story.
Then there's that confounded woman who ate that apple. They didn't even have apples there, at that time. But she placed women into extra-deep debt, who have remained there ever since.
Boy, that was one expensive piece of fruit, and that's one dysfunctional parent, no matter what stick you measure it by! (Um, unless it's a special stick.)
See it for what it is. Take the good parts, but recognize the ones that aren't. Listen to your heart, because it knows the truth. Would you call CPS on that parent?
If they did they would use these genes: what do ya know there is a heat shock protein in there.
An 86-year-old female patient from northeast Mexico presented with diffuse lepromatous leprosy (DLL). Sequence analysis of four genes (rrs, rpoB, sigA, and hsp65) from the skin biopsy specimen identified “Mycobacterium lepromatosis.” This is the first independent confirmation of a case of DLL due to M. lepromatosis.
sounds familiar to our skin, and then the white patches are left behind. My skin turned purple after this last onslaught I had.
"generalized dermatosis characterized by diffuse infiltration of the skin, predominantly in the face, with thickening of the superciliary region and ears plus an accompanying absence of eyebrows and eyelashes. In the rest of the body, the skin had an atrophic “cigarette paper”-like appearance and diminished or absent body hair.
The neurological examination revealed extensive areas of dysesthesia. The areas of diffuse lepromatous leprosy (DLL) associated with Lucio's phenomenon were characterized by a purpuric appearance, with necrosis and ulceration in some of the areas, leading to sloughing in acral sites such as fingers and toes. "........
..."Many acid-fast bacilli were seen"......
..." diverse range of clinical manifestations. Recently, Han et al. (4, 5) have established the existence of “Mycobacterium lepromatosis,” a closely related but distinct species, as a causative agent of DLL in two patients of Mexican origin in Arizona, who succumbed to the disease. Those authors have further investigated the existence of M. lepromatosis in leprosy patients in Mexico and have claimed that M. lepromatosis, particularly the DLL form, is the predominant cause of leprosy there ".........
So, you might have something there, Krits. The mycobacterium is diff from mycoplasm or microplasma, so this could be a bacterium rather that the plasma type or could be the new type. or diverse one.
....." Upon analysis of the sequences of the rpoB gene flanking the rifampin resistance-determining region (RRDR), there were multiple mismatches with the M. leprae sequence but there was 100% identity with the corresponding sequences of M. lepromatosis FJ924."...... sothis Fj924 m. lepromatosis?
The way the skin looks after a Morgellons onslaught, appears related to this.So, there are different species of this:
....."To confirm this identification, partial sequences of three other genes (hsp65, rrs, and sigA) were determined using the primers described by Han et al. (4) together with additional sets of primers that could amplify the sigA sequences from both the M. leprae and M. lepromatosis species "..........
....."M. leprae and M. lepromatosis also needs further investigation."..
rrs, rpoB, sigA, and hsp65) M. leprae M. lepromatosis
From rpoB named: M. lepromatosis FJ924. the M. lepromatosis? So the others remain to be identified: rrs, sigA, and hsp65
Bioagents: they say some very nasty things have been sprayed on Mexico. Why? Who really are the "terrorists". I believe they can be found in our own DARPA and other US terrorist organisation. They have infiltrated the "scientific biological community". Sneaky....heh?
....." Journal Article: Probable zoonotic leprosy in the southern United States.
ABSTRACT: In the southern region of the United States, such as in Louisiana and Texas, there are autochthonous cases of leprosy among native-born Americans with no history of foreign exposure. In the same region, as well as in Mexico, wild armadillos are infected with Mycobacterium leprae. Whole-genome resequencing of M. leprae from one wild armadillo and three U.S. patients with leprosy revealed that the infective strains were essentially identical. Comparative genomic analysis of these strains and M. leprae strains from Asia and Brazil identified 51 single-nucleotide polymorphisms and an 11-bp insertion-deletion. We genotyped these polymorphic sites, in combination with 10 variable-number tandem repeats, in M. leprae strains obtained from 33 wild armadillos from five southern states, 50 U.S. outpatients seen at a clinic in Louisiana, and 64 Venezuelan patients, as well as in four foreign reference strains. The M. leprae genotype of patients with foreign exposure generally reflected their country of origin or travel history.
However, a unique M. leprae genotype (3I-2-v1) was found in 28 of the 33 wild armadillos and 25 of the 39 U.S. patients who resided in areas where exposure to armadillo-borne M. leprae was possible. This genotype has not been reported elsewhere in the world. Wild armadillos and many patients with leprosy in the southern United States are infected with the same strain of M. leprae. Armadillos are a large natural reservoir for M. leprae, and leprosy may be a zoonosis in the region. (Funded by the National Institute of Allergy and Infectious Diseases and others.). "..........
Locating these: rrs, sigA, and hsp65 defin. involved with "thiol esters"............. that is what I am finding. just the beginning.
...."4. Genes and Its Variants Associated with Leprosy Types and Immunological Reactions4.1. Complement Component 3 (C3, 19p13.3-p13.2)Human complement component forms ester linkages with hydroxyl groups. Cleavage of C3 exposes a reactive short-lived thioester moiety in C3b, which covalently attaches to amine and carbohydrate groups on the target surface. This initial tagging is quickly amplified on foreign cells but is immediately regulated on human cells. Moreover, the reactivity of the thioester moiety to specific carbohydrates might lead to preferential opsonization of foreign particles and represent a basic pattern recognition mechanism [36, 37]. In an extrinsic protease pathway, plasmin, thrombin, elastase, and plasma kallikrein can also cleave and activate C3. Furthermore, target-bound MBL can activate C3 independently of mannan binding lectin serine protease 2 (MASP-2), C2, or C4 in vitro. The highly abundant C3 can act as a systemic surveillance protein that has few endogenous ligands but becomes transformed into one of the most versatile binding partners upon activation to C3b. The remaining C3a is a powerful chemoattractant that guides neutrophils, monocytes, and macrophages toward sites of complement activation and induces TLR activation in antigen presenting cells . Phenolic glycolipid 1, a major component of M. leprae cell wall and activated complement component C3, are exposed on the cell membrane of M. leprae-infected human dendritic cells. C3 costimulate naïve T cells via CD46 thus induce the differentiation of IL-10-secreting regulatory T cells. In this way, M. leprae subverts host natural immunity to provoke an adaptive response that favors bacillary survival . In populations from Ethiopia and Mali, it was found that mean C3 serum concentration was lower in leprosy patients (0.88 mg/mL in Ethiopic and 0.73 mg/mL in patients from Mali) when compared with nonleprosy subjects (1.1 mg/mL, both populations) being the lowest C3 levels in each population found in BL patients (0.62 mg/mL) and in TT patients (0.81 mg/mL) from Mali and Ethiopic populations, respectively. Otherwise C3 phenotypes designated according to electrophoretic mobility FF (fast-fast), FS (fast-slow), and SS (slow-slow) were not associated with leprosy. Correlation of both variables were not reported ."............
so the creation of the MACROMOLECULES are from m. leprae genes involving 'thiol esters". So this was the consequence of creating these supermacromolecules?
''''...."The present invention is directed to heat shock proteins from Mycobacterium leprae as well as their encoding polynucleotides and vectors and host cells containing these polynucleotides. These heat shock proteins and their encoding polynucleotides are useful in detection of Mycobacterium leprae. In addition, the heat shock protein can be used as an adjuvant in a pharmaceutical composition containing an antigen to induce or enhance the immune response against the antigen. Further, the heat shock protein may be used to treat atopic conditions or as a vaccine against Mycobacterium leprae. Alternatively, the heat shock protein can be used to form a fusion protein with an antigen to induce or enhance the immune response against the antigen.
So, could be in air, as vaccinate the air? right? sure? So, make a vaccine that has the leprosy in it, and then disperse into population, even though they do not have leprosy, but, this will surely give it to them. right. The trouble with vaccines? ........ the can heal or kill, right?
So, vaccine for a bacterium, like phages. Phages could be put in the air, like little machines. right?
C3b: Thioester moiety. The Movie Coraline comes to mind, they do put this in literature......strange way to tell us...though... I think it is all right here folks, the worm, the protozoan, the thioester, the c3 the complements to our dna. How nice. Add some proteins here and there from all kinds of creatures, and those proteins become the newly classified "nano junk dna", I would rather keep my natural "junk DNA"! Creates pseudogenes, or false genes, not natural at all, but propogate what is desired, right?
The mammalian complement system mediates inﬂammation by generating anaphylatoxins to elicit chemotaxis and cell activation, and by promoting phagocytosis, degranulation and cell lysis. Itsmain functions include host defence against microorganisms, elimination of immune complexes and apoptotic cells, and facilitating adaptive immune responses 1,2 . The pivotal complement factor C3 is the convergence point for the classical, lectin and alternative pathways of complement activation. C3 (187 kDa) emerged over 700 million years ago3 and belongs to the a2-macroglobulin (a2M) family.
These large (approximately 1,400–1,800 residue) proteins are characterized by homologous sequence features, including a unique thioester motif and a central, highly variable part that is functionally important 4 . Its members, such as the complement factors C3, C4 and C5, the proteinase inhibitor a2M and the insect and nematode thioester-containing proteins (TEPs), have important roles in the immune response in metazoans, considerably pre-dating the emergence of immunoglobulins 5
Key word is Thioester: The TED. The unit has some subunits:
Activation of C3 The orientation of the TED domain with the thioester pointing inwards seems to be essential for maintaining the protective TED– MG8 interactions in native C3. TED is buttressed in this position by interactions with MG2, MG8 and CUB (Fig. 4c). Cleavage of C3 at Ser 726–Arg 727 removes the ANA domain (which becomes the anaphylatoxin C3a) and yields an activated C3b with an exposed and reactive thioester
Complement: a key system for immune surveillance and homeostasis
Nearly a century after the significance of the human complement system was recognized, we have come to realize that its functions extend far beyond the elimination of microbes. Complement acts as a rapid and efficient immune surveillance system that has distinct effects on healthy and altered host cells and foreign intruders. By eliminating cellular debris and infectious microbes, orchestrating immune responses and sending ‘danger’ signals, complement contributes substantially to homeostasis, but it can also take action against healthy cells if not properly controlled. This review describes our updated view of the function, structure and dynamics of the complement network, highlights its interconnection with immunity at large and with other endogenous pathways, and illustrates its multiple roles in homeostasis and disease. A hidden connection is stronger than an obvious one. —Heraclitus of Ephesus (535–475 BC)
Yes, in the water, you are so correct, my friend. That dang thioester, a magic key, heh? in the soil, of course....... It was the magic pruvate in the Alchemy of Black Magic?
This pruvate contaminates and destroys, and then the real cure for the environment will be distributed for whoever is left standing, right? That would be the sanitization of the earth.
So only pure angels are left, sorta like Cain delivering the crops, right? All over again........
Except, they aren't pure, and we know it! They still have not learned!
The real Cain descendents or the lost tribes know better and they accept their plight, and yet reach for the light as well as we all do. It is harder for them, but they know the difference as well as the "scribes and pharisees" do, but, they are in the churches following protocol, or dogma, and not their hearts, where the light is. Why the Revelations are revealed to the churches, who don't really tell the truth, they hide it under a bushel. Once they wake up to their dishonesty, and preach the "spirit" then they will be okay. Why are they so blind, they cannot see? We do!
New type Leprosy? Infection with environmental mycobacteria in cutaneous lesions in leprosy patients, have been reported6 and could explain the DNA identiﬁed by Han et al. Alternatively, the suggestion that diffuse lepromatous leprosy is caused by a bacterium that is very closely related to M. leprae but clearly different from the type strain requires further study to authenticate this claim.
Wow....... HERE IS THE PROBLEM: It cannot be grown in culture!
ESTABLISHING CAUSATION OF DISEASE An important milestone in establishing the ‘Germ Theory’ in medicine was the application of Henle’s postulates to establish the causal relationship between a microbe and a disease. Henle’s work was later reﬁned by his student Robert Koch, to become known as Henle-Koch’s postulates. The postulates state that the following criteria have to be met for an agent to be established as the cause of an infectious disease; (1) the agent must be present in every case of disease (recognising asymptomatic carriers can exist), (2) the agent must be isolated from lesions in pure culture, (3) the agent must be inoculated into a susceptible host reproducing the disease and (4) the agent must be recovered from the lesions. With the leprosy bacillus the steps requiring growth of the agent have been impossible to meet because the bacterium cannot be grown in culture. The same is apparently true of the agent FJ924 though only a few attempts at cultivation have been made. As a surrogate for cultivation, DNA obtained from bacteria found in leprosy lesions have been repeatedly shown to be essentially identical to the type strain of M. leprae. 7,8 In addition, M. leprae has been isolated from human lesions, passaged in mice and armadillos and recovered from infected tissues. These passaged bacteria have been shown to cause characteristic granulomatous lesions with nerve invasion in the armadillo, the hallmark of M. leprae infection in man. Similar studies have not been forthcoming from Han’s group using the agent FJ924. These kinds of studies are essential to establish the linkage between the suspect agent from which DNA has been isolated from a small number of patients and the claim that the causative agent is separate and distinct from M. leprae.
So, it is in the soil and water, has to be in the air, viral nanoparticle form, So fire just might kill it then. Get some horseradish, some hot peppers, some garlic, salt and gold manna. We got it then. Again rrS and SigA in this new leprosy!
Maybe this is where the spirochaete come in: a new species of Leptospirosis. The rrS gene from this. So, is this what is in the the new Leprosy as well? fails to be cultured as well?
Pathogenic Leptospira spp. present in the blood of patients with leptospirosis during the first week of symptoms can be detected using culture or PCR. A proportion of patients who are positive by PCR are negative by culture. Leptospira spp. are fastidious bacteria, and we hypothesized that a false-negative culture result may represent infection with a distinct bacterial subset that fail to grow in standard culture medium. Methods
We evaluated our hypothesis during a prospective study of 418 consecutive patients presenting to a hospital in northeast Thailand with an acute febrile illness. Admission blood samples were taken for Leptospira culture and PCR. A single tube nested PCR that amplified a region of the rrs gene was developed and applied, amplicons sequenced and a phylogenetic tree reconstructed. Results
39/418 (9%) patients were culture-positive for Leptospira spp., and 81/418 (19%) patients were culture-negative but rrs PCR-positive. The species associated with culture-positive leptospirosis (37 L. interrogans and 2 L. borgpetersenii) were comparable to those associated with culture-negative, PCR-positive leptospirosis (76 L. interrogans, 4 L. borgpetersenii, 1 unidentified, possibly new species). ...."Leptospirosis is an acute febrile illness caused by pathogenic species belonging to the genus Leptospira [1,2]. This zoonotic disease has a worldwide distribution but is most common in tropical and subtropical regions and has the greatest impact on public health in developing countries [1-4]. Disease is maintained by chronic carrier hosts that excrete the organism into the environment, and infection in man results from direct contact with infected animals or indirect contact with a contaminated environment "..........
25 species involved in rrS???is this the PRIMARY UNIT? or subunit with the other 3 SigA, rpoB, and hsp65 (from the original leprosy). So it sounds like m. tuberculosis is in here too.
The PCR assay described here was designed to speciate Leptospira spp. associated with culture-negative infection, and increases the number of cases for whom the causative species can be identified. This assay also represents a potentially useful epidemiological tool in settings where culture is not available. It is not clear why culture-negative leptospirosis is so common among our patient population, but the ready availability of over the counter antibiotics and the frequency with which these are purchased represents a plausible explanation that could be tested in future studies.
Well well........ so if lepto is involved, different from what we saw before on this, there was no assoc with leprosy in our other researches, that I know of. So this lepto was created as new species, evidently.
Here is a breakdown of all spirochetes and the lepto are in there too.
Fig. 1. Taxonomy of Spirochetes. The figure shows a simplified version of taxonomy of spirochetes focusing on the clinically important genera. Bacteria specifically mentioned in the text are boxed; the diseases caused by them or associated with their presence are shown in red.
" Fig. 2. Membrane architecture of Gram-negative bacteria in comparison to spirochetes. Gram-negative bacteria possess two outer membranes and a small layer of peptidoglycan. The outer membrane is formed by LPS. Spirochetes also exhibit two membranes, but the outer membrane is not attached to the peptidoglycan layer, and the periplasmic space contains the flagellum. The outer membrane contains several lipoproteins, and glycolipids, in particular LPS, have been repeatedly described to be present as well. Note that LPS has been ruled out for Borrelia and Treponema, but has been shown in Leptospira"...... ars.els-cdn.com/content/image/1-s2.0-S0171298507001404-gr2.jpg
right in the membrane cells: They call parts of humans now Architecture.. Cell membrane architecture Peptidoglycan liposaccharides glycolipids lipoproteins etc.
So, looking at the ARCHITECTURE of MORGELLON CREATURE.........well......
Lanl............oh yeah...............created the cell.......ah hah....................yup, lets see how did it get in the water? mmmmm Remember the breakin at Los Alamos National Lab. where is that located really? now. What was lost computer information or was it the dang species? Lost like in spread around, like in released into environment on purpose. Then these labs joined the universities.
Okay so DOE, LANL, and PACE all work together.
The macro molecular minimal protocell people: All together one big happy Fam.
this shows this as membrane protein 341aa and did find the 451aa before, so this must be one of the proteins in the Morgellon Architecture............ Complicated SOB. so, this could be the on/off operator or switcher who knows, seems a switch has power to turn the dna on and off.
Rv0885: UNKNOWN Rv0885, (MTCY31.13), len: 340 aa. Conservedhypothetical protein, equivalent to O33063|MLCB57_18possible transmembrane protein from Mycobacterium leprae(341 aa), FASTA score: (83.9% identity in 341 aa overlap).Also similar except in C-terminus to T35630 probablemembrane protein from Streptomyces coelicolor (312 aa). experimental evidence, no additional detailsrecorded
If they remade the evolutionary minimal cell or have made one to replace our own would it be in this multicellular group. And this is by the Broad Institute who are involved in using art/synthetic stuffs.
Salpingoeca rosetta. beginning of animal cell? N. King?
But, could pick up whatever bacteria it encodes? so would depend on genetic makeup.
The ability to synthesize biological constructs on the scale of the organisms we observe unaided is probably one of the more outlandish, yet recurring, dreams humans have had since they began to modify genes. This review brings together recent developments in synthetic biology, cell and developmental biology, computation, and technological development to provide context and direction for the engineering of rudimentary, autonomous multicellular ensembles. Keywords
Can we build synthetic, multicellular systems by controlling developmental signaling in space and time?
Using biological machinery to make new, functional molecules is an exciting area in chemical biology. Complex molecules containing both ‘natural’ and ‘unnatural’ components are made by processes ranging from enzymatic catalysis to the combination of molecular biology with chemical tools. Here, we discuss applying this approach to the next level of biological complexity — building synthetic, functional biotic systems by manipulating biological machinery responsible for development of multicellular organisms. We describe recent advances enabling this approach, including first, recent developmental biology progress unraveling the pathways and molecules involved in development and pattern formation; second, emergence of microfluidic tools for delivering stimuli to a developing organism with exceptional control in space and time; third, the development of molecular and synthetic biology toolsets for redesigning or de novo engineering of signaling networks; and fourth, biological systems that are especially amendable to this approach.
Full-size image (62 K)
Figure 1. A cartoon illustrating chemical microinterface for controlling, in real time, concentration of morphogens across developing tissue. Morphogens are delivered with high spatial and temporal resolution (blue arrows). Their effect is read out using integrated fluorescent reporters (green arrows), and dosing of morphogens is adjusted using feedback control mechanisms to achieve the desired differentiation and growth of tissue.
emphasizes the large number of different membrane surfaces. In clockwise order, beginning from the upper left: (a) Giant unilamellar vesicles and blebs; (b) networks of giant vesicles connected by lipid microtubules; (c) ruptured GUV's on solid supported bilayers; (d) membrane nanodiscs containing transmembrane proteins; (e) supported lipid bilayers analyzed by NanoSIMS; (f) ruptured cell membranes on solid supports; (g) Bilayers tethered to a solid support containing ion channels; (h) vesicles tethered to a supported lipid bilayer by DNA; (i) visual representation of multi-scale simulations.
If Morgellons new name is Neurodermatitis, then would it be caused by the creation of the Neurome?
Neurome is the totality of neurological genes, RNA, proteins, cells, and their links that form the most complex information processing network on Earth. Neurome project collects the above data and build DBs to serve on the net openfreely.
gs. 10–13. Metopus hasei, a common ciliate in insufficiently aerated soils in vivo (10, 11), in the scanning electron microscope (12), and after silver impregnation (13). 10–12: M. hasei is about 100 μm long and has caudal cilia (arrows), which were not preserved in the SEM; 13: Metopid ciliates lack mitochondria and have many rod-shaped methanogenic bacteria in the cytoplasm (arrowheads). AZM: adoral zone of membranelles, MA: macronucleus.
Aldrich must be in the billions: More here and seems they create macromolecules dendrites, and the like as they say. Def are nanomaterials.
"Sigma-Aldrich offers a wide selection of dendritic macromolecules, including dendrimers, dendrons, and hyperbranched polymers. Available dendrimer types include: poly(amidoamine), also known as PAMAM, or STARBURST® dendrimers; phosphorous dendrimers; polylysine dendrimers; and polypropylenimine (PPI) dendrimers"
Dendrons are wedge-shaped sections of dendrimers with reactive focal point functions, useful as macromolecular building blocks. Hyperbranched polymers are inexpensive dendritic macromolecules that can be used for many applications (conjugate scaffolds, macromolecular chemistry, polymer additives) that are tolerant of imperfect branching and polydispersity.
And these branch. and the sigmafactor is what replicates them. I do not know if this is the name given because it it created by Sigma or it just is the replicating macromolecule. Hyperbranched polymers would fit description of the branched needle type forms which seem to have pointed ends an branch from a main strand, It seems that mycoplasmas and microbacteriums, mixed bacteriums microbes, etc are used as models for the synthetic brands. however are they also used as a way to get the synthetic polymers into the human system? If mutations are desired, a virus would have to be present. Or a phage used as a foreign DNA transposon, rather than an eater of bacteria. What virus could introduce the foreign material and be used in a phage system? The only ones I find, is from b. subtilis bacteriophage 029 or the Phi174 from 50s. and these walked right into the H5N1. This RNAP or RNA polymer sigma factor, with the ribosome as the replicator could be the source. This could create all kinds of things.
RNA polymer sigma factor from Sigma Aldrich?
E-COLI is the replicator............... dang......... it can stay, even though it is to do its job in the body and leave. So, Entamoeba-coli could be the protozoan? rather than what we call e-coli or it is the replicator form to bring in the macromolecules?Entamoeba coli, not Escherichia coli which "thrives on beach sand" ==================
The ability to identify amoeba based on nuclear structure in iodine wet mount preparations is limited. Cysts of the amoeba are usually granular. Cysts of the mature pathogenic E. histolytica contain four nuclei while those of the non-pathogenic E. coli contain up to eight nuclei. " www.provlab.ab.ca/webbug/parasite/artifact/ecoli.htm ============
Human elements - Charcot-Leyden Crystals Charcot-Leyden Crystals Trichrome stain x 1000 Charcot-Leyden crystals are a breakdown product of eosinophils. They are found in feces but may also occur in sputum and tissues. Please note they are non-specific findings and are not synonymous with parasitic infections. 51 - Charcot Leyden Crystals x1000 56 - Charcot-Leyden crystal - large - Trichrome stain x 1000 Charcot-Leyden Crystals are present in people with tissue-invading parasitic infections or in individuals with various kinds of allergic conditions. Crystals stain red in trichrome-stained fecal smear.
Root hairs & other plant elements
These are fuzz from peaches seen frequently in stools. They can be confused with Strongyloides or other nematode larvae by the inexperienced eye. The refractile core should not be mistaken for the well differentiated esophagus or intestine of a nematode larva. 4 - Plant hair - phase contrast x 100 Plant hairs - phase contrast x 100 Although one end of the hair may be tapering and smoothly rounded as a nematode larvae, the other end usually is blunt and irregular a feature not seen in a larval parasite. Root hairs are usually straight, but when flexed (on the left), can be interpreted as larvae. The approximate lengths and widths of the hair (250 - 350 mm x 10-15 mm) are consistent with the sizes of Strongyloides and other nematode larvae. 6 - Plant hair Plant hairs Phase contrast x 1000 69 Plant hair Spore Trichrome Stain x 1000 This artifact could be mistaken for a Giardia trophozoite. Note the lack of the two anteriorly placed nuclei, the dark stained median body and axonemes. The long tail-like object is not like the typical flagellum of Giardia. This object appears to be a spore that is germinating.
Well. as usual Freud was only centered on using physiology as a neurosis. Time to see the truth!
"Definition of CHARCOT-LEYDEN CRYSTALS : minute colorless crystals that occur in various pathological discharges and especially in the sputum following an asthmatic attack and that are thought to be formed by the disintegration of eosinophils Biographical Note for CHARCOT-LEYDEN CRYSTALS Char·cot , Jean–Martin (1825–1893), French neurologist. One of the fathers of modern neurology, Charcot created the greatest neurological clinic of his time. An eminent clinician and pathologist as well as a neurologist, he practiced the method which correlates the moribund patient's symptoms with the lesions discovered during the autopsy. He was the first to describe the disintegration of ligaments and joint surfaces, the condition now known as Charcot's joint or Charcot's disease, caused by tabes dorsalis. He did pioneering work on the determination of the brain centers responsible for specific nervous functions. He demonstrated the clear relationship between psychology and physiology, and his work on hysteria and hypnosis stimulated Sigmund Freud, one of his students, to pursue the psychological origins of neurosis. Leyden, Ernst Viktor von (1832–1910), German physician. Leyden was a professor of medicine at the University of Berlin and a renowned neurologist. In 1869 he described the crystals found in the sputum of bronchial asthma patients. The crystals had already been described by Charcot in 1853, and consequently they are associated with the names of both men.
When people name things after themselves, they tell us nothing of the species. Charcot-Leyden are two people who named something after themselves, which tell us nothing! Alzheimers disease. named after A. Alzheimer, Now ALS Arterial lateral sclerosis tells us what it is. Lymes Disease found in Lyme, Conn(named after a place) tells us nothing about what this is: a spirochete disease.
See what I mean. So, behind the names, one has to dig where the science was lost.
Alzheimers is about amyloids, plaque etc. Just a tag given. Like Morgellons, Masquelons would be more appropiate even, because it masks so many things. But, it involves plaque, it involves, fungus, it involves algae, it involves a formed organism, it has plant hairs, it has inorganic materials, like telechelic polymers, PEGs, AEGs, polyvinyl thiophenes. Nanoparticles, building blocks, macromolecules. Not so in the original Morgellons. Or was this Alchemical experiment done before? After all, Sir Thomas Browne understood Alchemy. How often has the Great Work of Alchemy involving the genome been done? Was it done with nanoparticles before, a previous alchemical experiment on humanity?
Last Edit: Dec 12, 2012 14:09:20 GMT -5 by skyship
Charcot-Leyden crystals: Slender crystals shaped like a double pyramid with pointed ends, formed from the breakdown products of eosinophils and found in feces, sputum, and tissues: indicative of an immune response that may have parasitic or nonparasitic causes.
CLINICAL MANIFESTATIONS Allergic bronchopulmonary aspergillosis occurs in patients with preexisting asthma (particularly glucocorticoid-dependent asthma) or cystic fibrosis and causes intermittent episodes of wheezing, pulmonary infiltrates from transient bronchial plugging, sputum and blood eosinophilia, low-grade fever, and brownish or greenish flecks in the sputum. These flecks contain Aspergillus hyphae, thick mucus, eosinophils, and Charcot-Leyden crystals. Some patients with repeated exacerbations develop central bronchiectasis and progressive loss of pulmonary function.
tash: Hi skizit, I have watched all your videos on youtube and cant thank you enough for all you have educated me on. I cry for you alot and a bit for me. I was wanting to send you photos of what is raining down everyday here in Australia in hope you can tell me
Dec 11, 2019 23:28:22 GMT -5
tash: not sure where to send them as hush mail and rocket mail bounced back
Dec 11, 2019 23:30:03 GMT -5