Stem Cells? Ok, another guess - they used HeLa cells in the manufacture of this or they knew they were already inside of us and would be incorporated into Morgellons? We did some pretty heavy research on the HeLa a couple of months ago, I've seen two researchers since then say that HeLA are a part of Morgellons. HeLa cells are immortal and I'm saying that what is inside of us - our spheres are too. This diagram below shows their placement in our artifacts:
it opened a lot of doors for me - we now have names for some of the artifacts we've been seeing and proves some of the theory that we have proposed is most likely happening, such as; we have HeLa cells, photos to show this aspect later.
"Fig 2. A Chlamydia trachomatis elementary body (EB) in the process of entry into a HeLa cell. Tannic acid stained to enhance visualisation of chlathrin. Note the clearly demarcated outer envelope of the EB and the surrounding membrane (m) of the vacuole. Also note how small the clathrin coated pit (ccp) is compared to the chlamydial endosome. The bar represents 0.1 microns."
We now have a name for our 'carbon-looking balls', 'ccp':
That's very interesting, Sky... the CDC publishing poetry, too. I don't understand the exact technicality of genetics too much but 'get' what you're saying. They are manipulating our genes and chromosomes, playing God with our bodies, and people are clueless this is even happening.
I love the post you wrote about how man is 2 chromosomes away from the ape - but, they consider us to be the same as the ape - basically evolutionists/atheists are in control of the Gov., according to them God didn't make man different than the other phylums... That was a great post, I wish you'd re-post it?
carbon balls ..............bucky balls............... if they have clathrin triskelion.
carbon bucky balls.
We don't know yet... we need some DNA/RNA, Molecular Biology testing done... there's an awful lot of carbon-looking artifacts in the mix but then I read yesterday where this Glomus fungus takes in carbon... a carbon vacuum? It looks like to me the bucky ball technology is being used... AND... even the organisms are defecating a bucky ball, too. I think fullerenes are now perpetual in nature!
These 'bucky balls' are inside of the fungus gnat larvae @100x. These are exact to what Carnicom is showing that contain hemoglobin:
The same ones are inside of our nematodes, Urine at 600x:
I suspect that all of the Morgellons organisms are defecating these, they are everywhere in our samples. Then one of the Glomus species has a carbon ball that resembles this - the Gi. Margarita.
It seems like this version of the SSB movie is different from what I remember, it seems that parts of it have been cut out from the original?
I remember noting in a wordpad document all of the terminology that I wanted to study as the movie was playing and then I posted all of these factors in a MDR thread, I remember specifically the movie referring to a 'satellite cell'... that looks similar to this:
Buckyballs were suggested by Teller to clean up the environment in 1990.
Beginning of the Kyoto stuff, the znos used to clean up water, the speroids of polystyrene
doe/genomes to life put microbes and radiolaren, which eat radiation into waterways.
these then combine with other microbes and they became in super radiative microbes.
Deinacoccos which eats radiation was used by Craig Venter to put in artificial DNA.
Did you believe Freitas, when he said the sphere was not perfect to be a nanobot?
but if it was a molecular bot. maybe it would fit the criteria.
This is more than just a natural thing, it is programmed, it is use of sacred geometry like the sea shell, the shape of the daisy, the shape of lichen, the shape of dicty, the shape of hydra the shape of clatrhin mimicked in bucky ball.
You see how they can become artificial?
mimics nature and makes artificial. Nature is the model, then along comes molecular models and nanomodes and then quantum dots.
Hey Sky, I just saw the movie you posted on how the vesicles are formed and made a comment that I think I have photos of the inside parts shown at 1:28.
I wrote Frietas, let me find that info...
This is from a blog article that I wrote back in October 2009 called "Micelles Inside of Us?". At this time, Carnicom had just published the same "black carbon balls" in his blood literature photos. Carnicom was finding hemoglobin, I was checking the artificial blood routes, studying respirocytes, Frietas is an expert in respirocytes:
"Micelles Inside of Us?
Believing that Carnicom is finding that we have an artificial blood component in the carbon spheres that we are seeing in the red wine samples, within the frass of the fungus gnat, and in the fungal growth and which appears to be the main carbon sphere involved in Morgellons, and that they might be a respirocyte, I emailed the main expert authority on Respirocytes, Robert Frietas, Jr., with the following question:
I am a Medical Researcher and seeing that you are an expert in Respirocytes and was wondering if you could identify the microscopic photo below as one?
Thanking you in advance for your time.''
(and sent him a link to this photo below:)
Dr. Frietas responds back with:
''No, those look more like micelles, with slightly lumpy edges; the profile edge of a respirocyte would appear perfectly smooth at this magnification.
Best wishes, Robert A. Freitas Jr.''"
The article goes on to explain that micelles are used in water remediation.
We know the nature of some of our lesions has been referred to as like a 'cone' that is embedded in the skin. That some seem to be funnel or tunnel-like and with a root system, we can't easily remove them, in fact - they are almost impossible to remove.
I'm seeing where this is a particular trait of the mycorrhiza fungi called Arbuscular mycorrhiza or "AM fungi".
"An arbuscular mycorrhiza (plural mycorrhizae or mycorrhizas) is a type of mycorrhiza in which the fungus penetrates the cortical cells of the roots of a vascular plant.
Arbuscular mycorrhizae (AMs) are characterized by the formation of unique structures such as arbuscules and vesicles by fungi of the phylum Glomeromycota (AM fungi). AM fungi help plants to capture nutrients such as phosphorus and micronutrients from the soil. It is believed that the development of the arbuscular mycorrhizal symbiosis played a crucial role in the initial colonisation of land by plants and in the evolution of the vascular plants.
It has been said that it is quicker to list the plants that do not form mycorrhizae than those that do. This symbiosis is a highly evolved mutualistic relationship found between fungi and plants, the most prevalent plant symbiosis known, and AM is found in 80% of vascular plant families of today."
Here's what an "AM" stock photo looks like:
I'm saying our lesion 'cones' resemble this nodule above from which the branches are forming down into our skin. The only problem is... this "AM" is only supposed to happen in plants.
"Arbuscular mycorrhizas are called this because they have developed a specific structure called an "arbuscule". Although some kinds of soil fungi can be associated with plant and animal disease, the fungi that form arbuscular mycorrhizas belong to a group of soil fungi that can be very beneficial. The name "mycorrhiza" means "fungus root" and this is derived from the close association of the fungi with plant roots - in fact, arbuscular mycorrhizal fungi cannot complete their life cycle unless they are connected to plant roots.
Generally, it is not possible to grow these fungi without the support of the plant. The reasons for this are still largely unknown."
Well... what happened? My ear is the leaf on a plant? It's riddled with the species within this group that causes the formation of the unique structures called "arbuscules".
If we look at the patents we can see where the scientists 'souped up' this arbuscules formation process. When the particles sprouted out of my head that landed on my body and ear... they were highly programmed to burrow deep into my skin to create the arbuscules and form a hyphaed root system from them.
Why did this website remove it's two first videos explaining what this web site is all about a month or so before the health care passage?? My take is that insurance comp. know this is happening to people and want a "bailout" in advance of all the claims sure to come!! I always wondered why they had obama and hilllary there on their videos, why would the germans care, now it seems quite clear. beammeup
If so, one would be able to obtain a lawyer and sue the government,( if it weren't for the laws that they already have in place saying that they can carry out biological warfare testing on the people of this country without informed consent.) How convenient. THEY LEFT NO STONE UNTURNED, BELIEVE ME.
The WHO refers to one of my illnesses as myalgic encephalomyelitis The USA refers to the same illness as Chronic Fatigue Syndrome....why you ask?
because the US created the mycoplasma which causes myalgic encephalomyelitis/cfs and because the US wants to belittle my illness by making me refer to it as chronic fatigue syndrome.... people would look at me and say..."Oh, so, you're tired all the time?"
I would reply yes, tired all the time....they would say yeah, so am I.................Which diminished the suffering I was enduring.
If I told them I had Myalgic Encephalomyelitis, they would take a few steps back from me....like I was contagious....NOONE had heard of Myalgic Encephalomyelitis....even my first medical doctor...I printed the information and took it to him. He had no idea what it was......He told me I didn't have Encephalitis.... :Pduh!
Mycoplasma was bioweapons testing
Morgellons is a bioweapons illness
sky, fermentation was the origin
mycoplasma fermentans......it's the one most people carry...and one of the most dangerous.
The Main Human Mycoplasma Pathogens Pathogen / Implicated Disease (1-6)
Mycoplasma genitalium Arthritis, chronic nongonococcal urethritis, chronic pelvic inflammatory disease, other urogenital infections and diseases, infertility, AIDS/HIV
Mycoplasma fermentans Arthritis, Gulf War Syndrome, Fibromyalgia, Chronic Fatigue Syndrome, Lupus, AIDS/HIV, autoimmune diseases, ALS, psoriasis and Scleroderma, Crohn's and IBS, cancer, endocrine disorders, Multiple Sclerosis, diabetes
Mycoplasma salivarium Arthritis, TMJ disorders, Eye and ear disorders and infections, gingivitis, periodontal diseases including even cavities.
Mycoplasma hominis and Ureaplasma urealyticum Pelvic inflammatory disease, infertility, non-gonococcal urethritis, vaginitis, cervicitis, amnionitis, pyelonephritis, post-partum septicemia, neonatal pneumonia, neonatal conjunctivitis, Reiter's syndrome, peritonitis, wound infections (C-section), low birth weight infants, and premature rupture of membranes . Mycoplasma pneumonia Pneumonia, asthma, upper and lower respiratory diseases, heart diseases, leukemia, Steven-Johnson syndrome, polyarthritis or septic arthritis, CNS disorders and diseases, urinary tract infections, Crohn's and Irritable Bowel Syndrome, Guillain-Barr syndrome, polyradiculitis, encephalitis, and septic meningitis, autoimmune diseases.
Mycoplasma incognitus and Mycoplasma penetrans AIDS/HIV, urogenital infections and diseases, Autoimmune disorders and diseases
Mycoplasma pirum Urogenital infections and diseases, AIDS/HIV
Mycoplasma faucium, M. lipophilum and M. buccale Diseases of the gingival crevices and respiratory tract
I think mycoplasma is a BIG part of Morgellon's disease.
Oh, and uh, btw.......you girls rock!! I can't keep up.
I am working 2 jobs....because I can!!!!!!!!! I am physically, mentally and emotionally able!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!
will read/review when able and attempt to catch up with you all
Hi Aqt, I just saw your lovely photo on another thread, you look like the picture of health! I have received some bioset treatment pre-evident Morgellons, I used to go to a Naturopath and that particular progressive Chiropractor on a regular basis. They did a lot of strange things to me... lol - and a lot of things that didn't necessarily make sense to me - BUT... I always felt better after their treatments - tell us about the bioset procedure?
The key to discovery, I believe is in the "Arbuscular" aspect of this disease. If it were not for these spores/fibers being like missles seeking out a target and digging into the skin... this disease would look totally different. I believe I know who owns the patent, I have not looked in detail, I believe it was first invented in EU and incorporated into a later patent. We can speculate on who to litigate all day long - the proof is in the pudding - we have to be tested with more advanced equipment to verify our concerns, then apply this data to the product/s. At least we are hopefully narrowing our search?
I am not an attorney but believe the people have inherent rights, I believe it is the 4th Amendment? that our Government could be violating by allowing molecular testing of products and pathogens on the people without their knowledge? It might be possible to become a dual resident/citizen of another country and sue as a citizen of that country? When the full truth of what Morgellons is - is known, it will most likely take litigation to get the attention of our Government and the Medical Professionals to make drastic changes within the system unless our Government can quickly step up and do the right thing and make the proper corrections.
Sky and I are trying to get some people to look at the first few pages of this thread to get their attention about what we believe is going on inside our disease. I'm not sure how many people are currently on this site but, I can tell you that there are dis-information 'plants' already amongst us. There will be people to follow that will come in here and try to discredit everything we have said and attack us, such as this "FrankN", "FrankyB" (Wed, 27 Dec 2006) person did with TT and ran him out. There is not one piece of information about our disease that TT has left behind that discredits him. The core - here and now - be warned, we will need to protect each other. Moderators, I ask that you lock this thread if it becomes attacked, our intention is to get our words into the hands of someone who can decipher this information and help us.
Yes, Aqt, I appreciate you being here and helping us to keep this thread streamlined so that a busy person, such as a doctor, scientist, etc., could come here and not find a thousand off-topic subjects to wade through. Let's keep it in the theme of molecular microbiology, what's possibly happening with our cells... on a cellular level, etc.?
I want to catch this thread up to some of my recent research, that I think pertains - to get us up-to-date with where some of us (think we) are:
Unfortunately, we don't have much to go with - we have to take what's out there by the scientists and doctors with equipment are telling us that they are getting from their experiments, lab results, etc., which takes us to Carnicom's latest report. By studying and building on his information, I was lead into new discoveries that I believe ties into the information that TT left behind...
"2. The chlamydia-like structure would appear on the surface to be a bacterial form. Chlamydia (esp. Chlamyida pneumonia) has been suggested as one target candidate because of numerous parallels in morphology, biological characteristics and symptomology that are in accordance with my study of that particular organism. But we must also notice that from the beginning, I have specifically used the term "chlamydia-like" , and not Chlamydia, for two good reasons:
a) No absolute and proper means of identification at the required level has come forth from any source.
b) Certain characteristics of the organism DO NOT fit the Chlamydia genus, especially with regard to chemical and thermal stresses that have been placed on the organism during various testing procedures."
Chlamydiae have long been known as obligate intracellular bacteria which cause a wide variety of disease in animals and humans [see: Chlamydial infections].
Chlamydiae were recognised as having a very wide host range while being closely related with each other, forming a common evolutionary lineage quite distinct from all other known bacteria. However, as Figure 1 shows, this perception has changed quite dramatically in recent years as a result of three main findings:
the isolation of a 'chlamydia-like' organism from an aborted bovine foetus
the discovery and ultra-structural analysis of several endosymbionts of free-living amoebae
the description of a conspicuous bacterium growing as a contaminant in a cell culture within "cytoplasmic phagosomes".
"Electron microscopic studies of these new obligate intracellular bacteria indicated that they possessed a life cycle very similar to the unique and characteristic chlamydial developmental cycle, although they have an additional crescent body growth stage associated with prolonged incubation and possibly characteristic of the Parachlamydiaceae.
Elementary and crescent bodies are released into the extracellular medium within vesicles or after amoebal lysis. Comparative ribosomal RNA sequence analysis identified them as being close but distinct living relatives of the previously described chlamydiae belonging to the Chlamydiaceae. However some of the differences between them suggested a hidden diversity of chlamydia-like bacteria which awaited discovery."
We can see in the diagram the diversity of changes that were noted to take place from 1996 to 2002.
"Parachlamydiaceae, which naturally infect amoebae, form a sister taxon to the Chlamydiaceae on the basis of the Chlamydia-like cycle of replication and 80% to 90% homology of ribosomal RNA genes. Because intra-amoebal growth could increase the virulence of some intracellular bacteria, Parachlamydiaceae may be pathogenic. Arguments supporting a pathogenic role are that Chlamydia pneumoniae, a well-recognized agent of pneumonia, was shown to infect free-living amoebae and that another member of the Chlamydiales, Simkania negevensis, which has 88% homology with Parachlamydia acanthamoebae, has caused pneumonia in adults and acute bronchiolitis in infants. The recent identification of a 16S rRNA gene sequence of a Parachlamydiaceae from bronchoalveolar lavage is additional evidence supporting potential for pathogenicity.
"Although Legionella was the first pathogen demonstrated to multiply and persist in amoebae (18), several other fastidious intracellular bacterial pathogens, including Chlamydia pneumoniae (19), Mycobacterium avium (20), Listeria monocytogenes (21), and an Ehrlichia-like organism (22), may infect free-living amoebae.
Extensive study of the ecology of Legionella pneumophila has confirmed empirical observations of its predilection for growth in hot water tanks and its localization in sediment (23). Rowbotham described the ability of L. pneumophila to multiply intracellularly within protozoa (18) and suggested that free-living amoebae could be a reservoir for Legionella species (24). As amoebae are common inhabitants of natural aquatic environments and water systems (25,26) and are resistant to extreme temperatures, pH, and osmolarity conditions while encysted (27), the Legionella reservoir is important. Growth of free-living amoebae at high temperatures (44°C to 53°C) was observed more frequently for strains isolated from hot-water tanks (mainly Hartmanella vermiformis) than for those isolated from moist sanitary areas (mainly Acanthamoeba, Naegleria, and Valkhampfia species) (26). This great tolerance of cysts and species-dependent thermotolerance of trophozoites could account for the difficulty in eliminating Legionellae from water systems (28). The resistance of Acanthamoeba spp. cysts to various disinfecting solutions (29–31) complicates the eradication of free-living amoebae. Moreover, a wide variety of Enterobacteriaceae have increased resistance to chlorination when ingested by Tetrahymena pyriformis (32). Thus, free-living amoebae could readily act as Trojan horses for bacterial endosymbionts (33,34).
The relationship between Legionellaceae and free-living amoebae, which serves as a model for other endosymbionts such as Parachlamydiaceae, is not restricted to the role of reservoir. Indeed, Acanthamoeba strains were found to produce Legionella-containing vesicles, which may be agents of transmission of legionellosis. The risk of transmission may be underestimated by plate count methods (35). In addition, Legionellae grown inside amoebae were more virulent (36,37) , more motile (24), and more resistant to biocides (38) than are bacteria cultured in axenic media. The entry of Legionellae into monocytes was found to be enhanced by the intra-amoebal growth environment (39). In addition, intra-amoebal growth of L. pneumophila was shown to induce an antibiotic-resistant phenotype, while Legionellae cultured in broth did not (40). Similarly, M. avium living within Acanthamoeba had greater resistance to rifabutin, clarithromycin, and azithromycin than did strains living in macrophages (41). This finding could result from decreased uptake of antibiotics into the amoebae, an inactivation of the compound within amoebae, or a change in the bacterial phenotype. Replication of bacteria in amoebae was found not only to affect the bacterial host (through increased potential for spread, resistance to biocides and antibiotics, and acquisition of virulence traits) but also to enhance the pathogenicity of the free-living amoebae (42).
Parachlamydiaceae family was proposed (44) that forms a sister taxon to the Chlamydiaceae, as it has a Chlamydia-like cycle of replication and 80% to 90% homology of ribosomal RNA genes. This family comprises two genera, of which the type strains are Parachlamydia acanthamoebae (17) and Neochlamydia hartmanellae (45).
The ecologic loci and prevalence of the Parachlamydiaceae are unknown, but the latter could be underestimated, as this fastidious gram-negative bacteria was recovered only by amoebal cultures, a procedure not performed routinely on clinical samples. Moreover, these Chlamydia-like organisms have potential for widespread dissemination, as they are mostly endosymbionts of Acanthamoeba, a free-living amoeba with worldwide distribution (27).
Strains of Parachlamydiaceae
Nine strains of Parachlamydia have been described (Table). The first, P. acanthamoebae, was identified within Acanthamoeba BN9, an amoeba recovered from the nasal mucosa of a female volunteer (17). Its 16S rRNA sequence had 88.2% homology with Simkania negevensis and 87% homology with Chlamydophila pneumoniae(17). The second, Berg17 endosymbiont, also isolated from the nasal mucosa of a female volunteer, seems to have an rRNA signature similar to that of the Bn9 endosymbiont, as demonstrated by the binding of the Bn9658 hybridization probe designed for in situ identification of P. acanthamoebae (17). The third, Hall’s coccus, was found in an Acanthamoeba isolated from water taken from a humidifier in a case of humidifier-associated fever in Vermont (16). Its 16S rRNA gene sequence had >99% similarity with that of Bn9 endosymbiont and 86% to 87% with those of four Chlamydia species (16). Two additional Parachlamydiaceae, UWE1 and UWE25, were also found to infect Acanthamoeba. Both amoeba strains were recovered from soil samples from Washington State (46). A sixth strain, UWC22 endosymbiont, infected an Acanthamoeba recovered from infected corneal tissues (46). TUME1 endosymbiont was found in an amoeba recovered from municipal sewage sludge in Germany (46). The eighth strain, Neochlamydia hartmannellae, is the only strain of Parachlamydiaceae isolated from Hartmanella vermiformis. It did not grow on Acanthamoeba sp. or Naegleria, and its 16S rRNA gene sequence had only 92% homology with that of P. acanthamoeba and varied from 91.6% to 97.1% with the four latter endosymbionts of Acanthamoeba (45). The last one, CorvenA4, could not be isolated. Only its 16S rRNA sequence was retrieved from a respiratory sample (47).
Rationale for Potential Pathogenicity
Intra-amoebal growth may increase the virulence of some intracellular bacteria (39), prompting concern that other intracellular bacteria recovered from amoebae, such as the Parachlamydiaceae, could be pathogenic. Indeed, a bacterium able to survive exposure to the lytic enzymes of amoebal phagolysosomes would probably also survive the lytic activity of macrophages. This hypothesis is supported by the fact that mutants of Legionella that have similar cytotoxic defects and intracellular replication in mammalian macrophages and protozoa have been isolated (48), suggesting a common adaptive mechanism to the intracellular environment. Moreover, Parachlamydia can adapt to mammalian cells, as demonstrated by successful passage from an amoebal host to Vero cells (a monkey cell line) (17). Additional arguments in favor of a pathogenic role of the Parachlamydiaceae are that Chlamydia pneumoniae, a well-recognized agent of pneumonia, was shown to infect free-living amoebae (19) and that another member of the Chlamydiales, Simkania negevensis (49,50) , which has 88% homology with P. acanthamoebae (46), has been shown to cause pneumonia in adults and acute bronchiolitis in infants (51,52).
Strong evidence that some Parachlamydiaceae could be pathogenic came from the identification of Hall’s coccus in an amoeba isolated from the source of an outbreak of humidifier-associated fever in the United States, as well as related serologic studies (16).
In addition, a patient with adult Kawasaki syndrome was found to have a fourfold rise in antibody titer to P. acanthamoebae (54). A possible relationship between a previous respiratory infection and Kawasaki syndrome has already been reported (57,58) . Thus, the role of Parachlamydia in the pathogenesis of Kawasaki syndrome should be explored further.
As Parachlamydia could potentially be resistant to lytic macrophages enzymes for years, it could enhance chronic inflammatory disease or chronic pathogenic mechanisms, such as the one leading to vascular damage. A role of Parachlamydiaceae in the pathogenesis of arteriosclerosis is suggested by the presence in an abdominal aneurysm specimen of a Chlamydia-like strain that had a sequence closer to that of P. acanthamoebae than to Chlamydia, Chlamydophila, and Simkaniaceae (56). Some serologic studies have suggested that Chlamydophila pneumoniae could play a role in the pathogenesis of arteriosclerosis (59,60) , although this observation was not confirmed in other studies (61,62) .
Based on this rationale, one may hypothesize that some Parachlamydiaceae could cause pneumonia. Thus, patients with nosocomial or community-acquired pneumonia of unknown etiology should ideally receive an extensive diagnostic work-up, including testing for Parachlamydia. In addition, patients with arteriosclerosis and Kawasaki disease or other infectious syndromes of unknown etiology should perhaps be tested for Parachlamydia. As Parachlamydia strains were all identified within free-living amoebae, recent history of swimming in ponds, rivers, or swimming pools might prompt a specific diagnostic approach.
No diagnostic tool is commercially available. Because of the fastidious nature of Parachlamydiaceae, molecular biology is probably the easiest and cheapest diagnostic approach. Serologic testing is also promising; however, it requires antigen and a laboratory capable of performing amoebal coculture.
The role of Parachlamydia sp. as an emerging pathogen needs to be confirmed. In view of the genetic diversity of the Parachlamydiaceae (46), their phylogeny needs to be elucidated, as the various species could be associated with species-specific pathogenicity. Search for additional Parachlamydia strains in hospital water systems could help define potential nosocomial exposures."
Parachlamydia spp. and Related Chlamydia-like Organisms and Bovine Abortion
"Figure. A) Immunohistochemical analysis of a bovine placenta positive by PCR for Parachlamydia acanthamoebae, showing a positive brown-red granular reaction within trophoblastic epithelium. Antigen detection was conducted with a polyclonal antibody against Parachlamydia spp. (3-amino-9-ethylcarbazole/peroxidase method, hematoxylin counterstain). B) Transmission electron micrograph of bovine placenta positive by PCR and immunohistochemical analysis for P. acanthamoebae, showing 7 cocci-shaped bacteria in an inclusion with morphologic features similar to those of Chlamydia-like organisms (12).
"Parachlamydia acanthamoebae is a Chlamydia-like organism that easily grows within Acanthamoeba spp. Thus, it probably uses these widespread free-living amoebae as a replicative niche, a cosmopolite aquatic reservoir and a vector. A potential role of P. acanthamoebae as an agent of lower respiratory tract infection was initially suggested by its isolation within an Acanthamoeba sp. recovered from the water of a humidifier during the investigation of an outbreak of fever. Additional serological and molecular-based investigations further supported its pathogenic role, mainly in bronchiolitis, bronchitis, aspiration pneumonia and community-acquired pneumonia. P. acanthamoebae was shown to survive and replicate within human macrophages, lung fibroblasts and pneumocytes. Moreover, this strict intracellular bacterium also causes severe pneumonia in experimentally infected mice, thus fulfilling the third and fourth Koch criteria for a pathogenic role.
Consequently, new tools have been developed for the diagnosis of parachlamydial infections. It will be important to routinely search for this emerging agent of pneumonia, as P. acanthamoebae is apparently resistant to quinolones, which are antibiotics often used for the empirical treatment of atypical pneumonia. Other Chlamydia-related bacteria, including Protochlamydia naegleriophila, Simkania negevensis and Waddlia chondrophila, might also cause lung infections. Moreover, several additional novel chlamydiae, e.g. Criblamydia sequanensis and Rhabdochlamydia crassificans, have been discovered and are now being investigated for their human pathogenicity."
The Spheres are the bacterial Chlamydia - Intracellular of Chlamydia can live, one of many:
**In this photo, I believe the images that look like flying bats represent the S. negevensis aspect, something similar might look like 'dragons'?... as seen in someone's spheres? S. negevenis has 'sisters', which I haven't looked at yet.
Simkania negevensis is within the Parachlamydiaceae family, let's look at it closer:
"Evidence for the Presence of Simkania negevensis in Drinking Water and in Reclaimed Wastewater in Israel"
Simkania negevensis is a recently discovered chlamydia-like intracellular microorganism which has been associated with bronchiolitis in infants and with community-acquired pneumonia in adults; a high seroprevalence of antibodies to the microorganism has been found in various population groups. S. negevensis can be grown in various cell lines as well as in free-living amoebae such as Acanthamoeba polyphaga. In this study, evidence for the existence of Simkania or Simkania-like microorganisms in drinking water and in reclaimed wastewater is presented for the first time. Detection of the microorganism was made possible by the development of a specific and sensitive filter membrane immunoassay and was confirmed by PCR detection of microbial DNA in the water samples. The common presence of S. negevensis in water sources together with the high seroprevalence of antibodies to it and early age of acquisition of infection may implicate water as a source of infection. The possible significance of this finding for public health and for municipal water testing and treatment needs to be further examined."
I believe I just uncovered some very interesting information over the past few Parachlamydia posts, I wanted to bring out the highlights into one post in laymen's terms so that we can possibly use this information to help us better understand what is most likely happening. Please feel free to help me look, this is a vast subject:
Chlamydia is a bacterial sphere which HOLDS 'others', such as Simkania negevensis, etc. which are also called "chlamydia-like intracellular microorganisms", it appears that this genus is ever-evolving and new discoveries are being made. That, as of 2009, new tools were being developed for the diagnosis of parachlamydial infections, that prior to then it was stated in 2002, there were no diagnostic tests for this type of infection, as per the PubMed articles above. We should be asking to be tested for and seek out diagnostic labs and doctors that can test for parachlamydial infections.
It's possible that some of our 'blob' and other images are what are known as "free living amoeba", which I'm looking at and seem possible, will post later.
We see that S. negevensis (SN) can live inside of free-living amoebae such as Polyphaga acanthamoeba (PA). So, we have various intracellular microorganisms, S. negevensis, as an example can be inside of the chlamydia (CL) 'sphere', which all can live inside of an amoeba / "blob".
P. acanthamoebae (PA) was shown to survive and replicate within human macrophages (MA). I believe we are seeing macrophages (photos later) in our specimens...
We also know that our disease is producing a biofilm (BF) which is probably another protective coating for these organisms.
S. negevensis can be inside of Chlamydia which both can live inside of P. acanthamoebae which can be inside of human macrophages, all covered in a biofilm. I believe this is a good example of what is happening inside of Morgellons with the complexity being the addition of more organisms and nested variables. Morgellons appears to be a certain nested matrix of specific organisms that we all have in common with the addition of 'others', in some, to cause different symptoms.
I'm thinking that the key is that we have to kill our pathogens in layers with the outside layer being attacked first?
I have noted from photographs that the biofilm (BF) appears to be the 'womb' from which anything can be seen inside of.
The algae/fungus/bacteria/amoeba byproducts produce or = BF
BF = MA <-+ PA <-+ CL <-+ SN
So, if the CL is creating the BF, it is nested inside of the MA and PA layers. Let us hope that the BF is being created by the fungal/algae or amoebae element? We know that some have had success in removing the biofilm aspect.
It might help to know if Chlamydia or P. acanthamoebae are known to produce a biofilm? (Someone looked and Chlamydia does form a biofilm.)
We might want to look at this statement to find clues inside the contagiousness or the life cycle of Morgellons?
"Chlamydia has a very unique life-cycle, in which in alternates between a non-replicating, infectious elementary body, and a replicating, non-infectious reticulate body. The elementary body is the dispersal form of the pathogen, and is analogous to the spore. The bacterium induces its own endocytosis upon contact with potential host cells.
Once inside a cell the elementary body germinates as the result of interaction with glycogen, and converts to its vegetative, reticulate form. The reticulate form divides every 2-3 hours, and has an incubation period of about 7-21 days in its host."
We have talked about the gycogen aspect, we need to look at this again or closer?
"1 : resembling a net or network; especially : having veins, fibers, or lines crossing <a reticulate leaf>
2 : being or involving evolutionary change dependent on genetic recombination involving diverse interbreeding populations"
I believe I can show photos of exocytosis, Exocytosis is:
"Exocytosis (ek-soh-sy-TOH-sis; from Greek ἔξω "out" and English cyto- "cell" from Gk. κύτος "receptacle") is the durable process by which a cell directs the contents of secretory vesicles out of the cell membrane. These membrane-bound vesicles contain soluble proteins to be secreted to the extracellular environment, as well as membrane proteins and lipids that are sent to become components of the cell membrane."
We need to look at the "Types" and the "Steps" of Exocytosis in the above link closer?
"Endocytosis is the process by which cells absorb molecules (such as proteins) from outside the cell by engulfing it with their cell membrane. It is used by all cells of the body because most substances important to them are large polar molecules that cannot pass through the hydrophobic plasma membrane or cell membrane. The process opposite to endocytosis is exocytosis."
morgaliens: Has anyone caught the wire growth on microscope camera?
May 25, 2019 17:46:19 GMT -5
EXPERT LIES !: EXPERT, you should be ashamed of yourself.
Jun 15, 2019 4:32:25 GMT -5
EXPERT LIES !: Yinan Zhao, Yujing Sha, Qian Lin, Yijun Zhong, Moses O Tade, and Zongping Shao . Facile Conversion of Commercial Coarse-Type LiCoO2 to Nanocomposite-Separated Nanolayer Architectures as a Way for Electrode Performance Enhancement. ACS Applied Materials
Jun 15, 2019 4:33:00 GMT -5
morgaliens: Is anyone else taking either legal stimulant medication or speed/methamphetamine?
Jun 21, 2019 12:58:55 GMT -5
morgaliens: If so, PLEASE contact me @ firstname.lastname@example.org!
Jun 21, 2019 12:59:38 GMT -5
morgaliens: I'm very interested in the relationship between Morgellons and stimulants.
Jun 21, 2019 12:59:56 GMT -5
emmymack84: I take adderall
Aug 28, 2019 21:51:15 GMT -5
Tainthatnice: I take those a couple times a year.
Sept 5, 2019 23:18:34 GMT -5