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Post by aqt on Apr 5, 2010 11:20:09 GMT -5
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Post by aqt on Apr 5, 2010 11:22:22 GMT -5
quote: Anglopwr A form of "quasi-life" that is thought by most biological scientists to be the precursor to life on Earth. A protocell is similar to the modern cell in many ways; they are both primarily composed of proteins, they both divide, and they both have a cell membrane. However, protocells do not have any organelles, and their metabolism processes are still unknown. Because of this, protocells are not classified as cells and so are described as "quasi-life". Protocells were thought to have formed about 4 billion years ago, when the Earth had a reducing atmosphere composed of such gases as ammonia, methane, water vapor, and hydrogen. It is thought that an electric spark from lightning or ultraviolet rays from the sun "fused" these chemicals into amino acids, which then formed into protocells. This process was re-created in the laboratory with positive results. Stanley Miller ran an electric discharge through a beaker filled with the gases from the primitive reducing atmosphere (described above). Amino acids and other biological compounds started to form at the bottom of the flask. A few years later, Sidney Fox heated a solution of amino acids, and artificial protocells were formed.everything2.com/title/protocell
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Post by aqt on Apr 5, 2010 11:24:02 GMT -5
en.wikipedia.org/wiki/Sidney_W._FoxSidney Walter Fox (24 March 1912 - 10 August 1998) was a Los Angeles-born biochemist responsible for unique discoveries in the autosynthesis of protocells. [edit] Professor In 1943 Fox was granted his first academic position at Iowa State College. In 1955 Fox assumed the directorship of the Oceanographic Institute at Florida State University. Shortly thereafter he published–with Joseph Foster—his first textbook. Beginning in 1964, Fox served as director of the Institute for Molecular and Cellular Evolution (IMCE) at the University of Miami. During this time, his laboratory was involved in studying some of the first moon rocks brought back by the Apollo missions. After more than three decades in Florida, Fox moved to Southern Illinois University in 1989, and then on to the University of South Alabama in 1993.
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Post by aqt on Apr 5, 2010 11:28:33 GMT -5
D. A Protocell Evolves 1. Before the first true cell arose, there would have been a protocell or protobiont. 2. A protocell would have a lipid-protein membrane and carry on energy metabolism. 3. Fox showed that if lipids are made available to microspheres, lipids become associated with microspheres producing a lipid-protein membrane. 4. Oparin demonstrated a protocell could have developed from coacervate droplets. a. Coacervate droplets are complex spherical units that spontaneously form when concentrated mixtures of macromolecules are held in the right temperature, ionic composition, and pH. b. Coacervate droplets absorb and incorporate various substances from the surrounding solution. c. In a liquid environment, phospholipid molecules spontaneously form liposomes, spheres surrounded by a layer of phospholipids; this is called the "membrane-first" hypothesis. d. A protocell could have contained only RNA to function as both genetic material and enzymes. 5. If a protocell was a heterotrophic fermenter living on the organic molecules in the organic soup that was its environment, this would indicate heterotrophs preceded autotrophs. a. A heterotroph is an organism that cannot synthesize organic compounds from inorganic substances and therefore must take in preformed organic compounds. b. An autotroph is an organism that makes organic molecules from inorganic nutrients. 6. If the protocell evolved at hydrothermal vents, it would be chemosynthetic and autotrophs would have preceded heterotrophs. 7. The first protocells may have used preformed ATP, but as supplies dwindled, natural selection would favor cells that could extract energy from carbohydrates to transform ADP to ATP. 8. Since glycolysis is a common metabolic pathway in living things, it evolved early in the history of life. 9. As there was no free O2, it is assumed that protocells carried on a form of fermentation. 10. The first protocells had a limited ability to break down organic molecules; it took millions of years for glycolysis to evolve completely. 11. Fox has shown that a microsphere has some catalytic ability; Oparin found that coacervates incorporate enzymes if they are available in the medium. E. A Self-Replication System Evolves 1. In living systems, information flows from DNA ? RNA ? protein; it is possible that this sequence developed in stages. 2. The RNA-first hypothesis suggests that the first genes and enzymes were RNA molecules. a. These genes would have directed and carried out protein synthesis. b. Ribozymes are RNA that acts as enzymes. c. Some viruses contain RNA genes with a protein enzyme called reverse transcriptase that uses RNA as a template to form DNA; this could have given rise to the first DNA. 3. The protein-first hypothesis contends that proteins or at least polypeptides were the first to arise. a. Only after the protocell develops complex enzymes could it form nucleic acids from small molecules. b. Because a nucleic acid is complicated, the chance that it arose on its own is minimal. c. Therefore, enzymes are needed to guide the synthesis of nucleotides and then nucleic acids. www.biologycorner.com/bio3/evolution/ch19.html
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Post by aqt on Apr 5, 2010 11:31:43 GMT -5
Man-made protocell hosts DNA synthesis The researchers showed that a mixture of a fatty acid, its corresponding fatty alcohol, and the glycerol ester of the fatty acid, all combined to spontaneously assemble into vesicles that allowed sugars and modified nucleotides to pass into the vesicle. To see if the nucleotide could polymerise inside the protocell without the aid of enzymes, the researchers placed a short, simple DNA template 15 bases long within the vesicles. They then added the complementary nucleotide to the medium containing the protocells. 'We saw the monomers diffuse into the cell, bind to the template and spontaneously polymerise,' says Szostak. 'The new chain grew to its full length.' www.rsc.org/chemistryworld/News/2008/June/04060803.asp
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Post by aqt on Apr 5, 2010 11:33:41 GMT -5
adsabs.harvard.edu/abs/2007APS..MARD38001RA simple yet tightly coupled catalytic cooperation between genes, metabolism, and container forms the design underpinnings of our protocell, which is a minimal self-replicating molecular machine. Experimentally, we have recently demonstrated this coupling by having an informational molecule (8-oxoguanine) catalytically control the light driven metabolic (Ru-bpy based) production of container materials (fatty acids). This is a significant milestone towards assembling a minimal self-replicating molecular machine
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Post by aqt on Apr 5, 2010 11:36:27 GMT -5
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Post by aqt on Apr 5, 2010 11:37:54 GMT -5
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Post by skyship on Apr 7, 2010 0:43:47 GMT -5
aqt, like this one? Astronomers are now able to detect planets orbiting stars other than the Sun where life may exist, and living generations could see the signatures of extra-terrestrial life being detected. Should it turn out that we are not alone in the Universe, it will fundamentally affect how humanity understands itself - and we need to be prepared for the consequences. royalsociety.org/Event.aspx?id=1887or this one? seems they are preparing for something? royalsociety.org/2010-The-detection-of-extra-terrestrial-life/so is this why we need protocells?................. whooooooaaaaaaaaaaaaaaaa! The originals? Think we already have them? ==================================== skyship
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Post by skyship on Apr 7, 2010 0:47:58 GMT -5
Note this article; as possibe changes in telomeres. and the scientist involved. Haven't we read about mr. wine before? mmmmmm "Human herpesvirus 6 (HHV-6) infects nearly 100 percent of humans in early childhood, and the infection then lasts for the rest of a person's life. Now, a team led by Peter Medveczky, MD, a professor in the Department of Molecular Medicine at the University of South Florida (USF), has discovered that in some individuals, HHV-6 causes such a permanent infection by inserting or "integrating" its DNA into human chromosomes. From this harbor, the viral DNA cannot be eliminated by the immune system.".................... www.sciencedaily.com/releases/2010/03/100308151055.htmthen..................................look at this.........................could be our kinase change. ========================= Induced alteration of the cellular stress fiber cytoskeleton by a varicella zoster virus protein kinase.abstracts.iovs.org/cgi/content/abstract/45/5/1630out of respect for their request, I cannot post the article, but it is very revealing. and not just one type, there are others, 2 others. will post on that in next post. skyship
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Post by skyship on Apr 7, 2010 1:48:00 GMT -5
this show 3 types of this virus. and one protein kinase was identified. www.ncbi.nlm.nih.gov/pmc/articles/PMC247706/pdf/jvirol00068-0468.pdf========================= Lessons to be learned from varicella-zoster virus.Abstract Varicella-zoster virus (VZV) is an alphaherpesvirus responsible for two human diseases: chicken pox and shingles. The virus has a respiratory port of entry. A fter two successive viremias, it reaches the skin where it causes typical lesions. There, it penetrates the peripheral nervous system and it remains latent in dorsal root ganglia. It is still debatable whether VZV persists in neurons or in satellite cells. During latency, VZV expresses a limited set of transcripts of its immediate early (IE) and early (E) genes but no protein has been detected. Mechanisms of reactivation from ganglia have not been identified. However, dysfunction of the cellular immune system appears to be involved in this process. The cell-associated nature of VZV has made it difficult to identify a temporal order of gene expression, but there appears to be a cascade mechanism as for HSV-1. The lack of high titre cell-free virions or recombination mutants has hindered so far the understanding of VZV gene functions. Five genes, ORFs 4, 10, 61, 62, and 63 that encode regulatory proteins could be involved in VZV latency. ORF4p activates gene promoters with basal activities. ORF10p seems to activate the ORF 62 promoter. ORF61p has trans-activating and trans-repressing activities. The major IE protein ORF62p, a virion component, has DNA-binding and regulatory functions, transactivates many VZV promoters and even regulates its own expression. ORF63p is a nuclear IE protein of yet unclear regulatory functions, abundantly expressed very early in infection. We have established an animal model of VZV latency in the rat nervous system, enabling us to study the expression of viral mRNA and protein expression during latency, and yielding results similar to those found in humans. This model is beginning to shed light on the molecular events in VZV persistent infection and on the regulatory mechanisms that maintain the virus in a latent stage in nerve cells. tinyurl.com/yk26vtu======================== VZV gene 66 protein kinase and actin accumulation, disorganization in the cytoplasma. US3 protein kinases in HSV and PRV as well. What concerns me is the cytoskeleton rearrangement in cells. ======================== Cytoskeletal rearrangements and cell extensions induced by the US3 kinase of an alphaherpesvirus are associated with enhanced spreadAbstract The US3 protein is a viral kinase that is conserved among the Alphaherpesvirinae. Here, we show that US3 of the swine alphaherpesvirus pseudorabies virus causes dramatic alterations in the cytoskeleton, resulting in the formation of long actin- and microtubule-containing cell projections in infected and transfected cells. Analysis with a GFP-labeled virus showed that multiple virus particles move inside the projections toward the tip. GFP-labeled virus could also be found in the cytoplasm of neighboring cells that were in contact with the projections. In addition, projection formation could be inhibited by using the actin-stabilizing drug jasplakinolide and could be induced by using the Rho kinase inhibitor Y27632. Analyzing the effect of these drugs on intercellular virus spread indicated that the observed US3-induced alterations in the host cytoskeleton are associated with enhanced intercellular virus spread, thereby suggesting a previously undescribed aspect of alphaherpesvirus spread. * cytoskeleton * herpes * projections ..."Some viruses have been reported to exploit the host cytoskeleton in often very fascinating ways to facilitate important aspects of their life cycle like entry, egress, and intercellular spread. The polyomavirus simian virus 40 interacts with actin and microtubules to facilitate virus transport to the nucleus (1, 2), and vaccinia virus interacts with microtubules and induces the formation of actin tails to allow efficient egress and intercellular spread in the presence of virus-neutralizing antibodies (3–9). In addition, members of the herpesviruses, adenoviruses, and retroviruses have been shown to use dynein-dependent microtubule-associated transport for efficient delivery of the viral genome at the nucleus"... www.pnas.org/content/102/25/8990.full============================= US3 kinase and cdc42? ? This was brought up way back when the info on Morgellons started to unfold, I do not forget! the cdc42 was involved in tests that many had at beginning. remember that? anyone? ============== Expression of herpes simplex virus type 2 US3 affects the Cdc42/Rac pathway and attenuates c-Jun N-terminal kinase activationBACKGROUND: Although the US3 gene product of herpes simplex virus (HSV) has been identified as a serine/threonine protein kinase (PK), the functions are poorly understood. RESULTS: We found that US3 PK of HSV-2 induced disruption of actin filaments, cell rounding and accumulation of binucleate cells in HEp2 cells. Cell rounding was abrogated by expression of either kinase-dead forms of US3 PK or a mutant protein lacking the acidic cluster in the kinase regulatory domain. Co-expression of dominant active forms of Cdc42/Rac inhibited cell rounding, suggesting that a signal transduction pathway involving Cdc42/Rac may play a role in the morphological changes induced by the kinase. Cdc42 and Rac, members of the Rho family of small GTPases, function as molecular switches changing actin cytoskeletal organization, influencing transcription and controlling apoptotic cell death. By computed homology search, we noticed significant similarities between US3 PK and p21-activated kinase (PAK), which is activated by the Cdc42 or Rac. We also found that the expression of US3 suppressed the activation of c-Jun N-terminal kinase (JNK), a kinase that is downstream of PAK. CONCLUSIONS: These observations suggest that the US3 PK affects the Cdc42/Rac pathway and can act as an upstream suppressor of JNK in the stress-activated signalling pathway. www.psrast.org/junkdna.htm====================== they did not realize that JNK if activated gives us the diseases we were merely carriers of , like mitochondrial diseases? This means, Lil sissy, that the JNK, is suppressed, meaning what keeps us from getting diseases etc, has been dismantled, in other words, they are put to rest when in the past they have kept us healthy, because we NATURALLY adjusted to whatever was in our autoimmune and autorecessive genes. what has been labeled JNK or junk genes. dang......................Harvard is giving it away, what has been and is being done........................... ============================== "Junk DNA" Over 98 percent of DNA has largely unknown function By Jaan Suurkula M.D. Summary Presently, only the function of a few percent of the DNA is known, the rest has been believed to be useless garbage, commonly called "Junk DNA" by molecular biologists. =-00=0=0=0=0=0=0=0=0=0=0=0=0=0= Increasing evidence is now indicating that this DNA is not "junk" at all. Especially, it has been found to have various regulatory roles. This means that this so-called "non-coding DNA" influences the behavior of the genes, the "coding DNA", in important ways.However, the knowledge is still very incomplete about this DNA. And there is little knowledge about the relationship between non-coding DNA and the DNA of genes. Without this knowledge it is completely impossible to foresee and control the effect of artificial insertion of foreign genes.
This is a very important reason why genetic engineering is unsuitable for commercial application. It is still at a stage of early experimentation with very incomplete understanding about its consequences. According to the ethical standards of sound science, the products of such experimentation should be strictly contained in labortories, especially as released DNA may spread indefinitely in an uncontrollable way. ......."More than 98 percent of all DNA, was called "Junk DNA" by molecular biologists, because they were unable to ascribe any function to it. They assumed that it was just "molecular garbage". If it were "junk", the sequence of the "syllables", i.e. the nucleotides in DNA should be completely random.However it has been found that the sequence of the syllables is not random at all and has a striking resemblance with the structure of human language (ref. Flam, F. "Hints of a language in junk DNA", Science 266:1320, 1994, see quote below). Therefore, scientists now generally believe that this DNA must contain some kind of coded information. But the code and its function is yet completely unknown. It has been reported that the sequences of this unknown DNA are inherited and that some repetitive patterns in it seem to be associated with increased risk for cancer. Also, the DNA has been found to mutate rapidly for example in response to cancer. It has been speculated that this DNA may contribute to the regulation of cellular processes. Haig H. Kazazian, Jr., chairman of genetics at the University of Pennysylvania has recently found reasons to suspect they may be a key force for the development of new species during evolution. He thinks this DNA may be essential for increasing the plasticity of the hereditary substance.Published at this website in May 1997." www.psrast.org/junkdna.htmI am onto this now, so the molecular biologists just went ahead any way. Now WE KNOW WHERE TO FOCUS! skyship
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Post by skyship on Apr 7, 2010 2:34:58 GMT -5
More snippets from the link: www.psrast.org/junkdna.htmcheck this out............................ ====================== .........Russian research adds a quantum physics perspective Recently, experimental results by Gariaev et al indicate that some, and perhaps important, aspects of genetic regulation are mediated at a quantum level. Moreover, in this respect they suggest that non-coding "Junk DNA" plays a crucial role, see * "The DNA-wave Biocomputer" (an MS Word document) and * "Crisis in Life Sciences. The Wave Genetics Response". Excerpt: "It appears that the languages we were looking for, are, in fact, hidden in the 98%, "junk" DNA contained in our own genetic apparatus [4]. The basic principle of these languages is similar to the language of holographic images [5] based on principles of laser radiations of the genetic structures [6] which operate together as a quasi-intelligent system, as in [3] It particularly important to realize that our genetic devices actually perform real processes which supplement the triplet model of the genetic code."............. ............."Conclusion The idea that a major part of our DNA is "garbage" ignored the fact that a key feature of biological organisms is optimal energy expenditure. To carry enormous amounts of unnecessary molecules is contrary to this fundamental energy saving feature of biological organisms. Increasing evidence are now indicating many important functions of this DNA, including various regulatory roles. This means that this so-called non-coding DNA influences the behavior of the genes, the "coding DNA", in important ways. Still there is very little knowledge about the relationship between non-coding DNA and the DNA of genes. This adds to other factors making it impossible to foresee and control the effect of artificial insertion of foreign genes" www.psrast.org/junkdna.htmmore from link at end of artcle: appendix; :Appendix JUNK DNA- May Not Be Junk After All (Quoted from Gene exchange no 2, 1996) In another reminder that we may not understand the full ramifications of genetic engineering, Science magazine recently reported new work on the function of genetic material*. Scientists have long been puzzled by the fact that fully 97% of the DNA in human cells does not code for proteins and appears to consist of meaningless sequences. The possibility that this apparently useless DNA has some as yet unknown function continues to tantalize scientists. The Science article reports on a paper suggesting that the non-coding 97% of the DNA, commonly referred to as junk DNA, might have a function. The authors of the paper employed linguistic tests to analyze junk DNA and discovered striking similarities to ordinary language. The scientists interpret those similarities as suggestions that there might be messages in the junk sequences, although its anyone s guess as to how the language might work. * F. Flam, Hints of a language in junk DNA, Science 266:1320, 1994.: ============================== so, do you think DNa is floating all around us? from the Aerosol genetic program? skyship
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Post by skyship on Apr 7, 2010 3:02:40 GMT -5
I do not think that molecular biologists or any of these artificial folks ever thought of the importance of all dna in a particular person. It was all general. so since it is junk they know we have it. They are using it, can be coded.......... What we as Morgellon people need to do is look at diseases in our family. Alz, als, muscular dystrophy, ms, mitochondrial diseases, autorecessive diseases, If we are carriers of certain diseases, our bodies often adjust and to manifest these diseases in us, because of JNK dna, but, those cannot be read like normal dna, they have adapted to the disease, and become recessive, not active in carrier, but would be active in our children. Common sense tells one to be wary, know the genetic manifestations, how it works, etc. What does recessive mean, etc. But, when it is labeled JNK, you and I know the technology today, if they can put gfp in us, they can follow the jnk as well. so, we are experiments...........we are tracked..........because ................... YOU KNOW WHAT? ?? GOD DON'T MAKE ANY JUNK! He gives us the brains, and the knowledge, to prevent disease, naturally we have adjusted, and we are alive today BECAUSE of our JUNK DNA. Why fix what was already fixed? And some of us still have ancient memory, ancient genes. However, we know it too is being used as a weapon. So, The selfish gene? Yes, but you didn't see the need, the reason you call it selfish, is because you do not have it! So you label the gene itself as selfish, when You Mr. D were part of the plan. To integrate perfect genes into near perfect bodies, yourselves! ==================== "Selfish" genes In describing genes as being "selfish", the author does not intend (as he states unequivocally in the work) to imply that they are driven by any motives or will—merely that their effects can be accurately described as if they were. The contention is that the genes that get passed on are the ones whose consequences serve their own implicit interests (to continue being replicated), not necessarily those of the organism, much less any larger level. This view explains altruism at the individual level in nature, especially in kin relationships: when an individual sacrifices its own life to protect the lives of kin, it is acting in the interest of its own genes. Some people find this metaphor entirely clear, while others find it confusing, misleading or simply redundant to ascribe mental attributes to something that is mindless. For example, Andrew Brown has written:" en.wikipedia.org/wiki/The_Selfish_Gene========================= That is assuming the carrier has no brains. Evolution happens, we evolve by use of jnk dna, simple as that. We do not become frogs, or elephants, or artificial pogrammes! That is the aim! for you nasty programmers who want to be scientists! It is only Philosophy to them, folks! Because they have hidden and stolen all the cures for themselves! To be used for experiments so that the masters in this earthly joint want immortal genes, which can only be PLASTIC, false genes, dead genes, ............... Just wait till our ship comes in, there will not be room for the lot of you molecular false lying "pie in the sky" BRATS! skyship
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Post by skyship on Apr 7, 2010 3:11:49 GMT -5
Studies have been done on this jnk dna: Recent studiesSuch observations as above have spurred an extensive research into "Junk DNA" in recent years, some of which is briefly presented below. Various important roles of "Junk DNA" have been discovered in recent years. In June 2004 a team at Harvard Medical School (HMS) reported, that they have, in a yeast, found a "Junk DNA" gene that regulates the activity of nearby genes. While common genes work by giving rise to proteins, this gene works by just being switched on. Then it blocks the activity of an adjacent gene. Quote: "In a region of DNA long considered a genetic wasteland, HMS researchers have discovered a new class of gene."... "The researchers have evidence that the new gene, SRG1, works by physically blocking transcription of the adjacent gene, SER3. They found that transcription of SRG1 prevents the binding of a critical piece of SER3's transcriptional machinery." Source: "Junk DNA Yields New Kind of Gene", Focus, Harvard Medical School, June 4 2004.
Some studies have found that noncoding DNA plays a vital role in the regulation of gene expression during development (Ting SJ. 1995. A binary model of repetitive DNA sequence in Caenorhabditis elegans. DNA Cell Biol. 14: 83-85.), including: * development of photoreceptor cells (Vandendries ER, Johnson D, Reinke R. 1996. Orthodenticle is required for photoreceptor cell development in the Drosophila eye. Dev Biol 173: 243-255.), * the reproductive tract (Keplinger BL, Rabetoy AL, Cavener DR. 1996. A somatic reproductive organ enhancer complex activates expression in both the developing and the mature Drosophila reproductive tract. Dev Biol 180: 311-323.), and * the central nervous system (Kohler J, Schafer-Preuss S, Buttgereit D. 1996. Related enhancers in the intron of the beta1 tubulin gene of Drosophila melanogaster are essential for maternal and CNS-specific expression during embryogenesis. Nucleic Acids Res 24: 2543-2550.). Over 700 studies have demonstrated the role of non-coding DNA as enhancers for transcription of proximal genes. This includes a/o:[/b]
* eosinophil-derived neurotoxin (EDN) and eosinophil cationic protein (ECP) (Tiffany HL, Handen JS, Rosenberg HF. 1996. Enhanced expression of the eosinophil-derived neurotoxin ribonuclease (RNS2) gene requires interaction between the promoter and intron. J Biol Chem 271: 12387-12393), * the variable region of the rearranged immunoglobulin mu (IgM) gene (Jenuwein T, Forrester WC, Fernandez-Herrero LA, Laible G, Dull M, Grosschedl R. 1997. Extension of chromatin accessibility by nuclear matrix attachment regions. Nature 385: 269-272.; Nikolajczyk BS, Nelsen B, Sen R. 1996. Precise alignment of sites required for mu enhancer activation in B cells. Mol Cell Biol 16: 4544-4554), * the alpha-globin gene (Bouhassira EE, Kielman MF, Gilman J, Fabry MF, Suzuka S, Leone O, Gikas E, Bernini LF, Nagel RL. 1997. Properties of the mouse alpha-globin HS-26: relationship to HS-40, the major enhancer of human alpha-globin gene expression. Am J Hematol 54: 30-39), * the activin beta A subunit gene (Tanimoto K, Yoshida E, Mita S, Nibu Y, Murakami K, Fukamizu A. 1996. Human activin betaA gene. Identification of novel 5' exon, functional promoter, and enhancers. J Biol Chem 271: 32760-32769).
Over 60 studies have demonstrated the role of non-coding DNA as silencers for suppression of transcription of proximal genes. Such silencer genes include a/o:
* apolipoprotein A-II gene (Bossu JP, Chartier FL, Fruchart JC, Auwerx J, Staels B, Laine B. 1996. Two regulatory elements of similar structure and placed in tandem account for the repressive activity of the first intron of the human apolipoprotein A-II gene. Biochem J 318: 547-553.), * the osteocalcin gene (Goto K, Heymont JL, Klein-Nulend J, Kronenberg HM, Demay MB. 1996. Identification of an osteoblastic silencer element in the first intron of the rat osteocalcin gene. Biochemistry 35: 11005-11011), * the 2-crystallin gene (Dirks RP, Kraft HJ, Van Genesen ST, Klok EJ, Pfundt R, Schoenmakers JG, Lubsen NH. 1996. The cooperation between two silencers creates an enhancer element that controls both the lens-preferred and the differentiation stage-specific expression of the rat beta B2-crystallin gene. Eur J Biochem 239: 23-32).
Some studies indicate that non-coding DNA regulate translation of proteins. This includes a/o
* the Lipoprotein Lipase gene (Ranganathan G, Vu D, Kern PA. 1997. Translational Regulation of Lipoprotein Lipase by Epinephrine Involves a Trans-acting Binding Protein Interacting with the 3' Untranslated Region. J Biol Chem 272: 2515-2519) * glutathione peroxidase and phospholipid-hydroperoxide glutathione peroxidase genes (Bermano G, Arthur JR, Hesketh JE. 1996. Role of the 3' untranslated region in the regulation of cytosolic glutathione peroxidase and phospholipid-hydroperoxide glutathione peroxidase gene expression by selenium supply. Biochem J 320: 891-895), * the luteinizing hormone/human chorionic gonadotropin receptor gene (58. Lu DL, Menon KM. 1996. 3' untranslated region-mediated regulation of luteinizing hormone/human chorionic gonadotropin receptor expression. Biochemistry 35: 12347-12353), * the thyrotropin receptor gene (Kakinuma A, Chazenbalk G, Filetti S, McLachlan SM, Rapoport B. 1996. BOTH the 5' and 3' noncoding regions of the thyrotropin receptor messenger ribonucleic acid influence the level of receptor protein expression in transfected mammalian cells. Endocrinology 137: 2664-2669), * the interleukin 1 type I receptor gene (Ye K, Vannier E, Clark BD, Sims JE, Dinarello CA. 1996. Three distinct promoters direct transcription of different 5' untranslated regions of the human interleukin 1 type I receptor: a possible mechanism for control of translation. Cytokine 8: 421-429)
Russian research adds a quantum physics perspective
Recently, experimental results by Gariaev et al indicate that some, and perhaps important, aspects of genetic regulation are mediated at a quantum level. Moreover, in this respect they suggest that non-coding "Junk DNA" plays a crucial role, see * " The DNA-wave Biocomputer" (an MS Word document) and * "Crisis in Life Sciences. The Wave Genetics Response". Excerpt: "It appears that the languages we were looking for, are, in fact, hidden in the 98%, "junk" DNA contained in our own genetic apparatus [4]. T he basic principle of these languages is similar to the language of holographic images [5] based on principles of laser radiations of the genetic structures [6] which operate together as a quasi-intelligent system, as in [3] I t particularly important to realize that our genetic devices actually perform real processes which supplement the triplet model of the genetic code."www.psrast.org/junkdna.htm========================= so, we can tackle some of these or go straight to the Molecular scientists for the most current DNA thefts and alteration and substitutions and integrative artificial crap. skyship
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Post by skyship on Apr 7, 2010 3:42:29 GMT -5
More from the study on Wave DNA; DNA Changes and Psychic Consciousness Words, Wave Genetics and the Grammar of SpiritualityShare Article | Feb 1, 2009 Mary Desaulniers Russian scientists' discovery of the wave information character of DNA points to a recovery of lost spirituality. Recent discoveries in the nature of DNA have overthrown the idea that man is genetically fixed. The Human Genome Project has uncovered two interesting facts about human genes: a) man has 30,000 genes( only 300 more than a mouse) in the human DNA; b) only 10% of the human DNA is used for coding and reproduction of proteins; the rest –approximately 90%-- is known as junk DNA which scientists have considered garbled and redundant. However, the Russian Branch of the Human Genome Project , headed by renowned Dr. Pjotr Garjajev, has uncovered the linguistic and psychic capabilities of junk DNA, ushering in a re-evaluation of the human gene in terms of words, wave genetics and a grammar of spirituality.DNA Changes and WordsAccording to the findings of the Russian Scientists, the genetic code follows the same rules found in human languages. By modulating certain frequency sound patterns on a laser ray, they are able to influence DNA frequency and genetic information. The most interesting aspect of their discovery is that simple words and phrases can work just as well as laser beams. Man can literally reprogram his genetic blueprint through words--which explains why affirmations and hypnosis can have powerful effects on mind and body. DNA Changes and Wave GeneticsThis discovery also points to the significance of sound frequencies and vibrations in the origin of human life and the possibility that creation was generated by waves of consciousness. The Phantom DNA effect is a case in point: the energy field of a DNA sample remains detectable by laser light even when the physical sample is removed. At a fundamental level, man is pure energy. In Wave Genetics, the junk DNA functions at a rich infrastructure level of super codes and wave communication, realized in material form as crystalline structures—dynamic gene-holograms in liquid crystals of the chromosome continuum. What this model suggests is that the human gene is part of larger holograms (multiverse) of wave information reality. Hyper-communication, in the form of remote sensing, remote healing and telepathy, is definitely a part of the human protocol. DNA Changes and the Grammar of SpiritualityIs it possible that the linguistic structures of the human genome point to a form of universal speech, a grammar of spirituality? William A Tiller in Psychoenergetic Science: A Second Copernican-Scale Revolution (Walnut Creek, CA: Pavior Publishing, 2007) certainly thinks so. Man, he explains, is composed of a personality body self and a “rich infrastructure ...in subtle bodies” that lead to ”greater human performance and expanded capabilities”(89). The purpose of life is building this infrastructure into a grammar of spirituality. Michael Cremo in Human Devolution: a Vedic Alternative to Darwin's theory (Badger, CA: Torchlight Publishing, 2003) claims that man originated billions of years ago in waves of consciousness, but has “devolved” into an ego-driven modern counterpart , alienated from the group consciousness and spirituality of his ancestors. Group consciousness can bring about the healing of the earth. If weather can be influenced by Schumann’s frequencies, man, who is capable of resonating at these frequencies, can through group consciousness bring about changes that dissolve violence and restore earth to its natural balance. alternativespirituality.suite101.com/article.cfm/dna_changesskyship
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Post by lilsissy on Apr 7, 2010 7:26:35 GMT -5
fascinating
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Post by skyship on Apr 7, 2010 15:11:59 GMT -5
I am looking into that jnk business, seems it includes viruses dna, not just human dna. I am wondering how much of that JNK has been altered already, due to mingling of yeast genes especially.
Okay, Kammy called it on the Bac, which I did see at one time as the LUCA, the last universal common ancestor in Woese's , tree of life the archaea the bacteria and eukaroytes, or as the recreated progenitor of life, the nucleus of the cell.
Now, viruses belong in there, and the replicating or reproducing factor comes down to the Us3 in the VZV Protetin kinase 66.
This US3 has:
8 amino acids an enzyme is produced autophosphorylation takes place means replicates phosphorylation automatically 68,000 molecular weight NH2-Tyr-Cys-Lew-pro-Leu-Phe-Gln-Gln-Lys-COOH From Alphaadenovirus package
However, this is from a eukaryotic species.
The proteins themselves that make up this nuclear core.
Baculoviruses are used to infect plants to alter genomic structure, however a virus is need to break into the plant cell.
This US3 is from the mix in this particular baculovirus. the Herpes is in Chickens and Turkeys, Ducks, and plants.
This is a polypetide double strand as far as I can determine.Marek's disease or Mardivirus.========================= The Protein Encoded by the US3 Orthologue of Marek's Disease Virus Is Required for Efficient De-Envelopment of Perinuclear Virions and Involved in Actin Stress Fiber BreakdownMarek's disease virus (MDV) encodes a protein exhibiting high amino acid similarity to the US3 protein of herpes simplex virus type 1 and the gene 66 product of varicella-zoster virus. The MDV US3 orthologue was replaced with a kanamycin resistance gene in the infectious bacterial artificial chromosome clone BAC20. After transfection of US3-negative BAC20 DNA (20{Delta}US3), the resulting recombinant 20{Delta}US3 virus exhibited markedly reduced growth kinetics. Virus titers on chicken embryo cells were reduced by approximately 10-fold, and plaque sizes were significantly smaller (65% reduction) compared to parental BAC20 virus. The defect of the US3-negative MDV was completely restored in a revertant virus (20US3*) expressing a US3 protein with a carboxy-terminal FLAG tag. Electron microscopical studies revealed that the defect of the 20{Delta}US3 mutant to efficiently spread from cell to cell was concomitant with an accumulation in the perinuclear space of primarily enveloped virions in characteristic vesicles containing several virus particles, which resulted in reduced numbers of particles in the cytoplasm. The formation of these vesicles was not observed in cells infected with either parental BAC20 virus or the 20US3* revertant virus. The role of the MDV US3 protein in actin stress fiber breakdown was investigated by visualizing actin with phalloidin-Alexa 488 after infection or transfection of a US3 expression plasmid. Addition of the actin-depolymerizing drug cytochalasin D to cells transfected or infected with BAC20 resulted in complete inhibition of plaque formation with as little as 50 nM of the drug, while concentrations of nocodazole as high as 50 µM only had a relatively minor effect on MDV plaque formation. The results indicated that the MDV US3 serine-threonine protein kinase is transiently involved in MDV-mediated stress fiber breakdown and that polymerization of actin, but not microtubules, plays an important role in MDV cell-to-cell spread.jvi.asm.org/cgi/content/abstract/79/7/3987================================ fibers we see are actin, and other cell skeleton fibers, some from plasma, some from the incorporated double stranded eurkaryotic dna.
A good example is the fiber on Karen's finger, the blue, black and red fibers are types of coded, gfp, rfp, the black could be artificial dna, the red could be ragged red fibers, from actin break down.
Odd colored fibers are marker proteins.
US3 is the protein kinase, this is a charged particle, either from the virus itself or from inorganic construction of a nucleotide from the protocell?
BAC20:============================= Actin Rearrangement-Inducing Factor of Baculoviruses Is Tyrosine Phosphorylated and Colocalizes to F-Actin at the Plasma MembraneInfluenza virus RNA polymerase is a multifunctional and multisubunit enzyme consisting of three viral P proteins, PB1, PB2, and PA. We have previously shown that radioactive 8-azido GTP (8-N3 GTP) was photo-crosslinked specifically to the PB1 subunit. Here we confirmed the specific crosslinking of PB1 using oxidized GTP and further identified the GTP analogue-binding domains after proteolytic cleavage of the crosslinked PB1 with V8 protease. The cleavage pattern of PB1 was determined by analysis of the amino-terminal proximal sequence of fragments generated in the presence of increasing concentrations of V8 protease. The GTP-crosslinking was identified in three fragments: two adjacent fragments, P6 starting from residue 179 and P11b starting from residue 298; and the third fragment, P11c, starting from residue 458. Thus, we propose that two GTP-binding sites exist in the PB1 subunit, i.e., the amino terminal-proximal site I located at the boundary between P6 and P11b, and the carboxy terminal-proximal site II on P11c fragment. The locations of GTP-binding sites I and II are close to those of sequence motif A and motif D, respectively, conserved among RNA-dependent RNA polymerases. Of the two fragments forming site I, the crosslinking of 8-N3 GTP is higher to P11b, while that of oxidized GTP is higher to P6, suggesting that the ribose and guanine moieties of GTP bound in this binding pocket face P6 and P11c, respectively. From the V8 concentration-dependent change in proteolytic cleavage pattern, it is likely that the two GTP-binding sites on PB1 protein are located on structurally different domains. The existence of two GTP-binding sites is discussed in relation to the binding sites for substrates, primers, and products. jb.oxfordjournals.org/cgi/content/abstract/122/3/627============================= very close to the H1N1 and avian virus?............
The V8 protease, the US3 has eight amino acids.........========================== The structure of a universally employed enzyme: V8 protease from Staphylococcus aureusAbstract: V8 protease, an extracellular protease of Staphylococcus aureus, is related to the pancreatic serine proteases. The enzyme cleaves peptide bonds exclusively on the carbonyl side of aspartate and glutamate residues. Unlike the pancreatic serine proteases, V8 protease possesses no disulfide bridges. This is a major evolutionary difference, as all pancreatic proteases have at least two disulfide bridges. The structure of V8 protease shows structural similarity with several other serine proteases, specifically the epidermolytic toxins A and B from S. aureus and trypsin, in which the conformation of the active site is almost identical. V8 protease is also unique in that the positively charged N-terminus is involved in determining the substrate-specificity of the enzyme.PDB reference: 1qy6 Keywords: V8 protease. scripts.iucr.org/cgi-bin/paper?S090744490302599X================================= Okay, will try to put together so far, the identifications. They are here.
But, hard to find, as Kammy can attest to.
Lil sissy, note from above article that the change in dna most likely takes place in the pancreas.
Trypsin, cytosol, mucin, all of it.
more on next post.
If we Morgellies can unify in a sense of spirit around the world we can lick this thing! Prayer in mass, lifting up our spirits to on high, looking up, not down, reaching out, we can do this.
At least expose it!
skyship
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Post by skyship on Apr 7, 2010 15:29:56 GMT -5
Kammy called it on the Baculovirus.
Aqt called it on the kinase.
Jill showed how the molyb. can work to break it up and expel it.
Lil sissy got the gfp.
Karen got the photo of the crystal
Bannanny as well, and the observations
Angmorg got the pattern formations.
Dr. Wymore is getting the dna blasted of sorts.
Dr. Harvey had the fungal connection.
Dr. Staninger had the proteomic and the molecular machine link to high tech.
Carnicom saw link between 3 Kingdoms.
So, here we are and I know I have missed some, but, we all have contributed.
Will try to compose a concise report.
We have the viral Varicella zoster Virus in a Alphaadenovirus The bacteria artificial chromosome bac 20 We have the kinase US3 Protocell? The construction of nickel and iron core. Protocell core?
skyship
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Post by beammeup on Apr 7, 2010 15:33:26 GMT -5
Does anyone know why they removed the original page with the first two videos??? beammeup
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Post by skyship on Apr 7, 2010 15:50:28 GMT -5
Because it showed it EXACTLY!
I am trying to reconstruct what I saw when I first saw it! Seems so long ago.
On video, said things were like many other things.
So, cyano - like CB001 trypanasoma - like, the kinase from protozoan of or trypanasomatid protein/ gene from squid- gfp? feather-like from bird, chicken, turkey? wing like from moth/butterfly Wnt gene different type cell protocells, or progenitors were used.
I do not remember him mentioning a viral like particle.
the progenitor was in a petri dish.
sporulation was part of the video shots, so the arbuscular of the fungi forming hyphae, and the released spores.
All biological parts it seems.
Why removed? Evidence?
skyship
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Post by skyship on Apr 8, 2010 7:59:20 GMT -5
For those who know the guts of computers, know the guts of man.Computational Models for the Formation of Protocell StructuresIt is reasonable to assure that molecules on the primordial Earth were at least as complex as those commonly found in interstellar space. Finding molecules that are necessary to form living systems in interstellarspace suggests that such molecules also existed on prebiological Earth. While six of the most abundant elements in the universe (H, C, N, O, S, P) are precisely those necessary to form living systems, a large number of organic and inorganic molecules have also been identified in different galaxies. Twelve of the most numerous ones “can be considered as the prebiological precursors of essentially all the biochemical compounds present in living systems.========== takes 18 amino acids from our bodies to make this.....notice the 6, 12, 18........ numbers.......============================== ...."ultraviolet light or electrical discharge from lightning, for instance, consistent with conditions believed to have been present on the prebiotic Earth"..... ================= So, ultraviolet light, from global dimming, the Aerosol operations? Ultraviolet light forms dimers, which take place in skin. With global warming scheme, operation...... it is being created, and those who can survive the onslaught of the dimer formations will make it. Pure radiant energy comes from sun, without it more fungus grows, and global dimming causes the ultraviolet rays, rather than pure sunshine.
The sun is silent, so why the operations continue is beyond me. No rays have been seen coming off from it lately. Unless, more ultraviolet radiation is needed to fulfill the operational plenary purpose.======================= ....." For example, Fox, et. al produced a proteinoid from thermal copolymerization of amino acids in 1958 [6], and Hargreaves, et. al conducted experiments to synthesize various lipids from glycerol, fatty acid, aldehyde and phosphate under conditions believed to be similar to that on the prebiotic Earth"........ ========================= So, so far we have Haldane, Oparin, Fox and now Hargreaves.........====================== ..."If life did come from a gradual chemical and biological evolution, it is conceivable that some mech anism created a closed compartment enclosing a complex mixture of simple (organic and inorganic) and macromolecular organic compounds, prior to the formation of the first living cell. It is generally agreed that structures similar to cell membranes provided such closed environments within which bio-chemical reactions crucial for life could take place. The major components of contemporary cell membranes are lipids, primarily in the form of phospholipids. Together with protein, they divide the intracellular substance from the external environment. Lipids are water-insoluble organic substances."......... ....."They may be considered amphiphilic molecules that contain nonpolar and polar regions, where the term amphiphilic means that they incorporate both a hydrophobic tail and a hydrophilic head within a single lipid molecule. The attractive interaction between nonpolar molecules in water is referred to as a hydrophobic effect. The special properties of amphiphilic molecules provide them the ability to self-associate or self-assemble into small molecular aggregates such as monolayers, micelles, bilayers, vesicles and biological membranes".... ========================= amphilphilic molecules: STAR Grant R829005 - The Influence of Amphiphilic Molecules on the Environmental Fate and Transport of Pharmaceuticals (2001 - 2004) Project Purpose:This work will evaluate transport processes affecting pharmaceutical movement in the environment, and will study the influence of amphiphiles (e.g., surfactants, phospholipids) on the fate and transport of pharmaceuticals in the environment. Project Description(s):This work will examine the influence of amphiphiles on the fate and transport of pharmaceuticals through a combination of batch and column adsorption and desorption experiments involving environmentally relevant pharmaceuticals and amphiphiles. Pharmaceuticals and amphiphiles will be used in concentrations covering the range of likely environmental conditions, from ultra-low concentrations (ng/L) of each, through moderate concentrations (mg/L or higher) which might be observed directly at an infiltration source (e.g., a confined animal feeding operation, etc.). Experimental results will be coupled with transport simulation (tracking both surfactants and pharmaceuticals) to assess the potential impact of amphiphiles on the migration of pharmaceuticals. Project Tasks: Task 1. Batch Adsorption/Desorption Experiments; Task 2. Column Transport Experiments with Surfactants; Task 3. Column Transport Experiments with Class II Amphiphiles/Vesicles; and Task 4. Development of a coupled transport simulator. www.epa.gov/ppcp/projects/star-grant4.html=================== tinyurl.com/ykjqddrskyship
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Post by skyship on Apr 8, 2010 8:40:06 GMT -5
continue......... ......."since a computer simulation can be built upon our understanding of biological systems and hypotheses, it provides persuasive support for the underlying biological theories. Also, a well-defined computational model can be used as a tool to investigate the properties and dynamics of more complex biological structures."....... ....."Due to the geometrical packing properties of lipid molecules, lipids tend to form micelle structures when they are cone shaped, i.e. when they are single-chained lipids (surfactants) with large head-group areas and relatively thin tails [12]. However, in these simulation experiments, the surfactants have large tails and small head-group areas, and thus should form inverted micelles."....... ......."Basic model and the interacting forces In an aqueous environment, hydrophobic effects drive phospholipid particles closer to each other, with their tails pointing inward, forming structures like micelles where water has been “squeezed out”. In order to simulate hydrophobic effects, we define each particle in our model to be an amphiphilic structure having a polar hydrophilic head group and a non-polar hydrophobic tail. These lipid-like particles are the basic elements of our computational model. The head of a particle is defined as a sphere of radius r. The tail is represented as a thin, inflexible rod of length L"....... ================= sounds like they are describing a bacteriophage here............ ================== ...."Based on hydrophobicity and other physical and chemical properties of lipids, we define seven interparticle interaction forces (Figure 3). These forces, described below from a top-level, intuitive viewpoint, exist between every individual pair of particles in the system. In the force definitions the particles are treated as structured rigid body rather than single points, and heads and tails play different roles in these definitions."..... ......"Discussion In this paper we presented a new computational model for simulations of the self-assembly of lipid-like particles into well-formed micelle-like protocell structures in an aqueous environment. By adopting a structural particle model and using a set of simple forces as an approximation of the inter-molecular interactions between lipid molecules, this model provides a reasonable reflection of protocell and reversed micelle formation while remaining reasonably tractable computationality. The effectiveness and efficiency of this model was demonstrated both by successful simulations of the formation of such as micelles from a pool of randomly distributed lipid-like particles, and the formation of reversed micelle structures in an oil environment containing small water drops."....... ==================== sounds like if you are making a simulation model, it might be used for something?..... ================ ....." Our next objective is to simulate the formation of monolayer structures in an oil-water interface environment, and develop computer programs for a three-dimensional simulation. This model may also be used to study, at the microscopic level, the selfassembly of different protocell structures in the evolutionary process and the impact of environmental conditions on the formations of these structures. It may also be useful as a tool to investigate the effects of changes in the environment (such as pH values and temperature in the solvent, the density of the lipids, etc.) on the formation of lipid aggregates, as well as the dynamic (fluid-like) property of those aggregates. This model may be further generalized to simulate the formation of other, more complex structures of amphiphilic components. Along this line, we are currently exploring the possibility of further extending this model to simulate the formation of membrane-like bilayer structures. We hope that further progress of this research will provide substantial insight into the process of protocell structure formations, and provide a useful tool for such studies."... ================= Incidently................ ================ ..."Acknowledgment: This work was supported in part by NASA award NAGW-2805."..... ================ Note references and scientists tinyurl.com/ykjqddrskyship
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Post by lilsissy on Apr 8, 2010 21:14:30 GMT -5
reversed micelle structures in an oil environment containing small water drops.".......
Can these be used for reverse transcription of D.N.A. sequences ?
Anybody know?
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Post by lilsissy on Apr 8, 2010 21:53:46 GMT -5
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Post by skyship on Apr 9, 2010 17:24:32 GMT -5
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Post by aqt on Apr 10, 2010 11:30:35 GMT -5
lilsissy www.ncbi.nlm.nih.gov/pubmed/12927829sky, that is the Syd Fox I researched with the protocell is there something different in your link that I didn't find? what of that link did you want us to see?? thanks for all aqt
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Post by aqt on Apr 10, 2010 11:34:01 GMT -5
Inclusion Bodies en.wikipedia.org/wiki/Inclusion_bodiesInclusion bodies are nuclear or cytoplasmic aggregates of stainable substances, usually proteins. They typically represent sites of viral multiplication in a bacterium or a eukaryotic cell and usually consist of viral capsid proteins. Inclusion bodies can also be hallmarks of genetic diseases, as in the case of Neuronal Inclusion bodies in disorders like Frontotemporal dementia and Parkinson's disease Protein inclusion bodies are classically thought to contain misfolded protein. However, this has recently been contested, as green fluorescent protein will sometimes fluoresce in inclusion bodies, which indicates some semblance of the native structure and researchers have recovered folded protein from inclusion bodies
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Post by aqt on Apr 10, 2010 11:40:54 GMT -5
When genes from one organism are expressed in another the resulting protein sometimes forms inclusion bodies. This is often true when large evolutionary distances are crossed: a cDNA isolated from Eukarya for example, and expressed as a recombinant gene in a prokaryote risks the formation of the inactive aggregates of protein known as inclusion bodies. While the cDNA may properly code for a translatable mRNA, the protein that results will emerge in a foreign microenvironment. This often has fatal effects, especially if the intent of cloning is to produce a biologically active protein. For example, eukaryotic systems for carbohydrate modification and membrane transport are not found in prokaryotes. The internal microenvironment of a prokaryotic cell (pH, osmolarity) may differ from that of the original source of the gene. Mechanisms for folding a protein may also be absent, and hydrophobic residues that normally would remain buried may be exposed and available for interaction with similar exposed sites on other ectopic proteins. Processing systems for the cleavage and removal of internal peptides would also be absent in bacteria. The initial attempts to clone insulin in a bacterium suffered all of these deficits. In addition, the fine controls that may keep the concentration of a protein low will also be missing in a prokaryotic cell, and overexpression can result in filling a cell with ectopic protein that, even if it were properly folded, would precipitate by saturating its environmentARE WE DEVELOPING INCLUSION BODIES WHEN THE GENES FROM ONE ORGANISM ARE EXPRESSED IN ANOTHER......INCLUSION BODIES WE ARE EXPRESSING GENES FROM OTHER ORGANISMS... Like the woman with the cyanobacteria growing out of her spinal lesions and the folks who have the insects.....they are expressing the insect gene one organism expressing the genes of another. aqt en.wikipedia.org/wiki/Inclusion_bodies
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Post by skyship on Apr 10, 2010 16:00:27 GMT -5
Yes, exactly and I have done extensive research on Fox, it does not stop there, he found how earth began, and life itself, but he did not make the protocell that changes our very cells, one has to go into the molecular approach, however, our problem is the missing MICROBIOLOGY sources. From Fox I found more, If you can look up Margulis protocells, or even Woese's protocells, he seems to put the baculovirus as the progenitor, but what did they actually do? The only link I can find to baculovirus is Bac20 which relates to viral, which causes mutation into the as messenger RNA to DNA, the viral is what unfolds the DNA, along with the prions.
Who actually was given the power to change our DNA?
That is my question.
I believe a total of 12 genes have been altered, replaced, or destroyed.
Finding these is the issue.
Replace of body parts, fibronectin, actin, cytoskeleton remodeling, has made us simple the base for architecture design.
This is where you either integrate or you stay natural.
Your choice.
skyship
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Post by skyship on Apr 10, 2010 16:06:16 GMT -5
I chose to stay natural, and it won't be easy.
The AMA is only going to perpetuate the dna link to everything, when it is the cell itself, viruses, the drugs themselves have the power to cause pancreatitis like Imurol, many others, ......
One has to make the choice of natural, or modified drugs which modify dna itself.
There are Rna viruses, there are DNA viruses, and there are bacteriophages which carry dna changes in their heads, which through viral vector, integrated into our DNA. by MUTATION
So, much is based on DNA, when it is the viral component that has control
From viral they went to nanotubes. which operatie any damn way they want to.
Depends on how programmed.
They are artificial.
Skyship
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