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Post by skyship on May 12, 2011 23:27:47 GMT -5
I am bringing this over from LB for all to see from our dear friend "Sunny" who posted this.
Dr. William Tarver Harvey, 73, of San Antonio, Texas, and a native of Liberty, 73, died May 4, 2011, of a heart attack in San Antonio.
A memorial service will be held at the U.S. Air Force Academy in the near future.
Dr. Harvey was the son of Cordelia Williams and Thomas Elijah “E.T.” Harvey.
He earned his undergraduate degree and wings at the U.S. Air Force Academy. Soon after, he became a physician, earning his M.D. degree from Case Western Reserve University. He spent much of his career with NASA, focusing on the medical challenges to humans in space.
A seasoned traveler, in his time he saw the entire world. After retirement, he embarked on another career — researching formerly unknown infectious diseases. His achievements gained him a listing in Who’s Who in America, among other honors.
Dr. Harvey was preceded in death by his parents; his sister, Patricia; and by his brother, Thomas E. Jr.
Survivors include his wife, Pat Dotson Harvey of San Antonio; a nephew, Thomas Elijah Harvey III; an aunt, Mary Alice Harvey of Liberty; and numerous cousins.
Memorial donations may be made to the Cordelia Williams Harvey Scholarship Fund, Copiah-Lincoln Foundation, P.O. Box 649, Wesson, MS 39191, or to the charity of the donor’s choice.
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skyship
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Post by skyship on May 12, 2011 23:33:03 GMT -5
Also on same thread Sharedelight posted statements written by Dr. Harvey. ============================.
Friday January 18, 2008 Dr. Harvey's Latest Statements Re Morgellons and Mutant Worms
The latest Washington Post article on Morgellons has interesting statements from Dr. Harvey as followup to my previous post.
William Harvey, 70, who serves as chairman of the MRF board, has taken those theories one step farther. He says he became interested in Morgellons research after successfully battling chronic fatigue syndrome and made it his mission to find cures for such unexplained illnesses.
He wouldn't be specific, explaining that he first wants the results of his research to appear in a top-notch, peer-reviewed journal such as the Lancet. "This may be the story of the century," he says. A semi-retired doctor in Colorado Springs who spent most of his career working in space medicine for the Johnson Space Center, Harvey says he may have found not only why Morgellons patients would both scratch and act strange, but also what could be the "genesis of probably most chronic human illnesses," such as autism, obesity, chronic fatigue and bipolar disorder.
It all boils down to this: mutant worms.
Harvey hypothesizes that a type of nematode, a wormlike parasite that lives in the soil as well as in the guts or lungs of about half the animals on the planet, mutated somewhere in the 1970s in Southeast Asia and jumped from animals to humans. The parasite is easily spread through the fecal-oral route if someone, for example, is out working in the garden, fails to wash his or her hands thoroughly and then eats an orange. Or it gets into the lungs by inhaling sputum or by kissing. The worm then takes up residence in the colon, Harvey theorizes, and the body's immune system holds it in check.
But when the immune system falters, the worms swarm in the body. That's what happens, Harvey hypothesizes, after a human is infected with a strain of bacteria first reported in 1986, Chlamydophila pneumonia. These bacteria like to live in immune cells, Harvey says, and they feast on those cells' energy. With the host's immune system compromised, the mutant nematodes begin reproducing exponentially, Harvey suspects. They burrow a hole in the wall of the colon, then usually travel at night through the bloodstream or the lymphatic system or crawl in hordes between the layers of the skin, like other species of nematodes are known to do, to the parts of the body with the most blood flow: the face, head and nose. There, a cranial nerve leads right into the brain. A pileup of worms could jam blood and oxygen flow to the brain, Harvey says. "That may explain the psychological symptoms," including the hallucinations, he says.
It may explain why Pam Winkler took herself to the emergency room recently. She said that a huge bump had appeared on the side of her skull in the middle of the night. By morning, she said, the bump was gone, but she could feel crawling all over her face. She wasn't making it up, she swore. And she put her stepsister, with whom she's been living since she got out of the state hospital, on the phone. "I can see them. They're moving down from her head to her eye," said Karen DeWeese. "They're about one and a half inches long and a half-inch wide. They look like bubbles under the skin." The ER doctor later found nothing.
The fibers, according to Harvey's theory, are really the hard shells, which he calls cuticles, that these worms shed at five stages as they grow from egg to larvae to adult. The red fibers are the males, he says. Blue fibers are female. "Using a 2,000-power microscope, you can see inside them," he says. "They look like little stovepipes to me. I can tell the blue ones are female because there's a kink in the middle for the sexual organs and some kind of pouch. And we have pictures of them laying thousands of eggs."
lymebusters.proboards.com/index.cgi?board=rash&action=display&thread=14812
LB posted but original is out there somewhere.
The Pulmoni lung worm.....gi worm, skin worm..........? Maybe the Watani Worm, it is red, found in an Ice Meteorite.
skyship
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Post by skyship on May 12, 2011 23:47:27 GMT -5
Here are some papers:Morgellons disease * William T. Harvey, MD, MPH Affiliations o Corresponding Author InformationReprint requests: William T. Harvey, MD, MPH, Medical Advisory Board, Morgellons Research Foundation, PO Box 4154, Baltimore, MD 21094-4154 www.eblue.org/article/PIIS0190962207001958/fulltext========================= An article describing Dr. Harvey, ALS, Lymes etc and treatments. www.enjoyyourlife.ch/images/harvey.pdf========================== ‘Lyme disease’: ancient engine of an unrecognized borreliosis pandemic?q W. T. Harvey, P. Salvato Diversified Medical Practices, Houston, Texas, USA www.ilads.org/files/harvey.pdf==========================
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Post by skyship on May 13, 2011 2:06:57 GMT -5
"I think we are a looking at a major problem that has been unrecognized in humanity right now." -- Dr. William Harvey NASA www.bibliotecapleyades.net/ciencia/ciencia_kerala07.htm#Morgellons%20-%20Weird%20Alien%20Bug%20Hits%20Thousands%20In%20US==================== "Ancient Engine of An Unrecognised 'Borreliosis' Pandemic? A former NASA physician and epidemiologist based in Houston also believes there is an infectious bacteria at the heart of this problem. Dr. William Harvey is the current chairman of the NASA Education Advisory Committee. He has documented more than 565 of these (Borreliosis) cases in Texas and says 94% of (those with Morgellons' skin lesions) have tested positive for the bacteria associated with Lyme disease, or Borreliosis. "I think we are a looking at a major problem that has been unrecognized in humanity right now." Harvey co-authored a published medical study concluding the bacteria Borrelia burgdorferi, associated with Lyme disease, could be at the heart of a widely unknown misdiagnosed infection. In 2003 Harvey published his research in the medical journal Medical Hypotheses. His article 'Lyme Disease: Ancient Engine of an Unrecognized Borreliosis Pandemic', suggests that the bacteria associated with Lyme disease is much more widely distributed. "The yet-unrecognized form appears to have a broader clinical presentation, wider geographic distribution, and vastly greater prevalence," Harvey wrote in his report. He says research suggests it attacks the immune system in a specific way rendering it susceptible to these unusual organisms. "The lab tests that we do are predictably showing certain immune damage and it is consistent from patient to patient to patient to patient." Harvey believes the bacteria is the bigger problem. But the so-called parasites, which have yet to be clinically proven in a controlled laboratory setting, do have highly unusual characteristics as seen through a scanning electron microscope. Harvey says some of the "filaments" have been confirmed as the infectious yeast Candida tropicalis and that doctors can easily see the physical symptoms in people who are branded "delusional." He says when patients complain that "fibers" are coming out of their skin sores, physicians should investigate. "All the doctors have to do is buy a 30X hand-held microscope from Radio Shack and look," Harvey said. "The facts speak for themselves." ......................... ========================= The Ultimate Chemtrail Truth. A Review...... early reports on Morgellons. www.bibliotecapleyades.net/ciencia/ciencia_morgellons08.htm#The_Ultimate_Chemtruth_Skyship
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Post by aqt on May 13, 2011 15:13:50 GMT -5
not a coincidence and so very sad.
They are willing to do whatever it takes to keep their secret
what they don't know is
we won't let them.
I'm so sorry.
aqt
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Post by gail roberts on Aug 9, 2013 0:51:52 GMT -5
not a coincidence and so very sad. They are willing to do whatever it takes to keep their secret what they don't know is we won't let them. I'm so sorry. aqt
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Post by Fl4sh52 on Aug 11, 2013 11:30:16 GMT -5
Sky, it's funny how most research and articles are done in Asia drug research and such are heavy throughout Korea and the wetlands and jungles. If Dr. Harvey is correct 1970's time wise the Vietnam war? Hmmmm.....
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Post by Fl4sh52 on Aug 11, 2013 11:51:30 GMT -5
I don't think they will recognize the problem until they are ready. Doesn't an epidemic kick off the Marshall law thing? Just sayin! Here's a funny story in my area they stopped visitors from going to a fossil site . Reasoning it would cause e dirt to become airborne releasing a spore or better known as valley fever. Here's the thing though people believe anything..... If they are scared. Sky, correct me if I'm wrong here but doesn't the breeze, wind, rain, traffic also kick up dust more so than a few people picking up fossils of shark teeth. The medical establishment can't be all to blame for the coverup. The Dr's who do care and take time to try and find answer are just cut-off with the standard insurance company answer of No! Not covered. Then to the the surprise of the office wham an audit or threat of an insurance company pulling contract. The insurance companies have all been merged saw that coming about 10 yrs ago. There were numerous insurance companies offering decent coverage. Now there are less than maybe 30-40 if lucky but they are somehow all connected. Controlling coverage and services. Medical to pharmaceutical. Same with drug companies they have all merged now there are only about 10 large pharma co. All the others are subsidiaries or co- market drugs. Generic companies are owned by the big pharma companies. So in essence they get paid for brand and generic production sales. Still they play the game of lawsuits to protect patents so the generic cant reach market or I should say can't reach market by other companies driving price down. It's slight of hand tricks. Here is a good one if the government was so gung ho for recycling why don't they make the stock bottles that medication comes in recyclable? Anthore thing. There are places with cancer clusters where all of a sudden it had to be tainted water. But people forget to look up they think jet engines fuel is normal, but don't think to keep looking up watching the chemtrails fall for hrs. Breathing and absorbing through clothes and skin. It is all a game of smoke and mirrors.
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Post by skyship on Aug 12, 2013 12:36:47 GMT -5
A nematode very common in Asian areas is this which many do not talk of. They are not pinworms: " Strongyloides stercoralis is the scientific name of a human parasitic roundworm causing the disease of strongyloidiasis. Its common name is threadworm. In the UK and Australia, however, the term threadworm can also refer to nematodes of the genus Enterobius, otherwise known as pinworms.[1]" "Persistence of infection is the first of these important features. Because of autoinfection, humans have been known to still be infected up to 65 years after they were first exposed to the parasite (e.g., World War II or Vietnam War veterans). Once a host is infected with S. stercoralis, infection is lifelong unless effective treatment eliminates all adult parasites and migrating autoinfective larvae."......"Main article: Strongyloidiasis Many people infected are usually asymptomatic at first. Symptoms include dermatitis: swelling, itching, larva currens, and mild hemorrhage at the site where the skin has been penetrated. If the parasite reaches the lungs, the chest may feel as if it is burning, and wheezing and coughing may result, along with pneumonia-like symptoms (Löffler's syndrome). The intestines could eventually be invaded, leading to burning pain, tissue damage, sepsis, and ulcers. In severe cases, edema may result in obstruction of the intestinal tract, as well as loss of peristaltic contractions.[9] Strongyloidiasis in immunocompetent individuals is usually an indolent disease. However, in immunocompromised individuals, it can cause a hyperinfective syndrome (also called disseminated strongyloidiasis) due to the reproductive capacity of the parasite inside the host. This hyperinfective syndrome can have a mortality rate close to 90% if disseminated.[10][11]Immunosuppressive drugs, such as those used for tissue transplantation (especially corticosteroids) can increase the rate of autoinfection to the point where an overwhelming number of larvae migrate through the lungs, which in many cases can prove fatal. In addition, diseases such as human T-lymphotropic virus 1, which enhance the Th1 arm of the immune system and lessen the Th2 arm, increase the disease state.[11] Another consequence of autoinfection is the autoinfective larvae can carry gut bacteria back into the body. About 50% of people with hyperinfection present with bacterial disease due to enteric bacteria. Also, a unique effect of autoinfective larvae is larva currens due to the rapid migration of the larvae through the skin. Larva currens appears as a red line that moves rapidly (more than 5 cm or 2 in per day), and then quickly disappears. It is pathogonomic for autoinfective larvae and can be used as a diagnostic criterion for strongyloidiasis due to S. stercoralis."........ en.wikipedia.org/wiki/Strongyloides_stercoralisI have seen the larva migrans. It is painful. also the intestinal issues. It also has filaria, the smaller worms. Many symptoms of Morgellons similar. 3 things often connected to Morgellons: Chlamydia forms Candida tropicalis Nematode (or filaments) All 3 Dr. H mentions. I have thought about this gut bacteria being carried throughout the body. These are nasty creatures carrying this crap. Then in the MEast wars, along comes oncho and leishmaniasis (just another form of Chagas like disease) All areas of war. Africa was loaded with this. Sand areas or jungle areas. So, why or why are Americans told this does not exist here? Most Morgellons were found in water areas Fl, Ca, Tx. So, where does the worm originate other than soil? What kind of soil? Swamps? Jungles? etc? The Labs? "Chemoattractant This parasite depends on chemical cues to find a potential host. I t uses sensor neurons of class AFD to identify cues excreted by the host.[15]S. stercoralis is attracted to nonspecific attractants of warmth, carbon dioxide, and sodium chloride. Urocanic acid, a component of skin secretions in mammals, is a major chemoattractant. Larvae of S. stercoralis are strongly attracted to this compound.[7] This compound can be suppressed by metal ions, suggesting a possible strategy for preventing infection."How about "manna"? or its real name Gold powder? cure for many things? S
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Post by skyship on Aug 12, 2013 12:48:34 GMT -5
Circled form: fragment of the worm. I am pulling forms out like this. So, I do think Dr. H. had a good hypothesis. This is at 400px. upload.wikimedia.org/wikipedia/commons/thumb/9/94/Strongyloides_-_very_high_mag_-_2.jpg/400px-Strongyloides_-_very_high_mag_-_2.jpg"Strongyloidiasis is a human parasitic disease caused by the nematode (roundworm) Strongyloides stercoralis, or sometimes S. fülleborni. It can cause a number of symptoms in people, principally skin symptoms, abdominal pain, diarrhea and weight loss. In some people, particularly those who require corticosteroids or other immunosuppressive medication, Strongyloides can cause a hyperinfection syndrome that can lead to death if untreated. The diagnosis is made by blood and stool tests. The drug Ivermectin is widely used in the treatment of strongyloidiasis. It is thought to affect 30–100 million people worldwide, mainly in tropical and subtropical countries. Worldwide efforts are aimed at eradicating the infection in high-risk groups. Strongyloidiasis was first described in France in 1876."........"History The disease was first recognized in 1876 by the French physician Louis Alexis Normand, working in the naval hospital in Toulon; he identified the adult worms, and sent them to Arthur Réné Jean Baptiste Bavay, chief inspector for health, who observed that these were the adult forms of the larvae found in the stool. In 1883 the German parasitologist Rudolf Leuckart made initial observations on the life cycle of the parasite, and Belgian physician Paul Van Durme (building on observations by the German parasitologist Arthur Looss) described the mode of infection through the skin. The German parasitologist Friedrich Fülleborn described autoinfection and the way by which strongyloidiasis involves the intestine. Interest in the condition increased in the 1940s when it was discovered that those who had acquired the infection abroad and then received immunosuppression developed hyperinfestation syndrome.[8]"....
en.wikipedia.org/wiki/Strongyloidiasis
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Post by skyship on Aug 12, 2013 13:09:27 GMT -5
Strongyloides stercoralis: there but not seen ...."Strongyloides stercoralis is a nematode endemic in humid, tropical regions (1, 2) including Africa, Southeast Asia, and Latin America (3). It is also endemic in southeastern United States and southern Europe, although most cases in the US occur in immigrants and military veterans who have lived in endemic regions (2). A second species of Strongyloides, S. fuelleborni can cause human strongyloidiasis but is less common and mainly found in Africa and Papua New Guinea (4). S. stercoralis is unique in its ability to replicate in the human host permitting ongoing cycles of autoinfection. Strongyloidiasis can consequently persist for decades without further exposure to exogenous infection (2, 4). The estimated prevalence of strongyloidiasis is between 50 to 100 million infections worldwide; however, the accuracy of these estimates is uncertain due to the poor sensitivity of screening methods (2, 5)."..... ..."Chronic S. stercoralis infections can be asymptomatic or cause cutaneous, gastrointestinal and/or pulmonary symptoms (4). In patients with concurrent Human T-cell-lymphocytic virus 1 (HTLV-1) infection or those on corticosteroid therapy, autoinfection can go unchecked and large numbers of invasive Strongyloides larvae may disseminate widely and cause hyperinfection, which can be fatal"....... So we have the: Virus:HTLV-1 ( this has been associated with Morgellons) Bacteria: Chlamydia (Carnicom) Yeast: Candida tropicalis Nematode: Dr. Harvey Protomyxozoa (Dr. Fry): trypanasomatid, myxotricha (symbiosis)? Myxotricha: has 3 forms of spirochaetes, archean form? extremophiles.(Lynn Margulis) Naval related. from the oceans, termite gut, or your local tree? Wood ticks?
Evidently they like damp places. black fungi.==== Pathogenesis Strongyloides has a complex biology with two separate life cycles, the free living cycle and the parasitic cycle (8, 9). Filariform larvae in the soil infect the human host by penetration of intact skin to begin the parasitic cycle. The larvae enter circulation, are transported to the lungs, penetrate alveolar spaces, ascend the bronchial tree, are swallowed and reach the small bowel (2, 4, 9). There the parthenogenetic females (i.e., capable of reproducing without males) embed in the duodenal mucosa and lay embryonated eggs that hatch in situ, releasing the rhabditiform larvae in the intestinal wall (4). The larvae migrate into the lumen and are either passed into feces or mature into filariform larvae, which can infect the intestinal mucosa or skin of the perianal region to restart the parasitic cycle (2, 4). Rhabditiform larvae passed into feces can become infectious filariform larvae directly or go through a free-living cycle of development in the soil. This adaptability allows for the parasite's survival in the absence of mammalian hosts www.ncbi.nlm.nih.gov/pmc/articles/PMC2948977/
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Post by skyship on Aug 12, 2013 13:17:21 GMT -5
Vectors of war? Rapid detection of Opisthorchis viverrini and Strongyloides stercoralis in human fecal samples using a duplex real-time PCR and melting curve analysis. Janwan P, Intapan PM, Thanchomnang T, Lulitanond V, Anamnart W, Maleewong W. Source Department of Parasitology, Faculty of Medicine, Khon Kaen University, Khon Kaen 40002, Thailand. Abstract Human opisthorchiasis caused by the liver fluke Opisthorchis viverrini is an endemic disease in Southeast Asian countries including the Lao People's Democratic Republic, Cambodia, Vietnam, and Thailand. Infection with the soil-transmitted roundworm Strongyloides stercoralis is an important problem worldwide. In some areas, both parasitic infections are reported as co-infections. A duplex real-time fluorescence resonance energy transfer (FRET) PCR merged with melting curve analysis was developed for the rapid detection of O. viverrini and S. stercoralis in human fecal samples. Duplex real-time FRET PCR is based on fluorescence melting curve analysis of a hybrid of amplicons generated from two genera of DNA elements: the 162 bp pOV-A6 DNA sequence specific to O. viverrini and the 244 bp 18S rRNA sequence specific to S. stercoralis, and two pairs of specific fluorophore-labeled probes. Both O. viverrini and S. stercoralis can be differentially detected in infected human fecal samples by this process through their different fluorescence channels and melting temperatures. Detection limit of the method was as little as two O. viverrini eggs and four S. stercoralis larvae in 100 mg of fecal sample. The assay could distinguish the DNA of both parasites from the DNA of negative fecal samples and fecal samples with other parasite materials, as well as from the DNA of human leukocytes and other control parasites. The technique showed 100% sensitivity and specificity. The introduced duplex real-time FRET PCR can reduce labor time and reagent costs and is not prone to carry over contamination. The method is important for simultaneous detection especially in areas where both parasites overlap incidence and is useful as the screening tool in the returning travelers and immigrants to industrialized countries where number of samples in the diagnostic units will become increasing. www.ncbi.nlm.nih.gov/pubmed/21537984
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Post by skyship on Aug 12, 2013 13:19:56 GMT -5
Detection of Opisthorchis viverrini in infected bithynid snails by real-time fluorescence resonance energy transfer PCR-based method and melting curve analysis. Intapan PM, Thanchomnang T, Lulitanond V, Pongsaskulchoti P, Maleewong W. Source Department of Parasitology, Faculty of Medicine, Khon Kaen University, Khon Kaen, Thailand. pewpan@kku.ac.th Abstract A real-time fluorescence resonance energy transfer (FRET) PCR combined with melting curve analysis was developed for the detection of Opisthorchis viverrini in experimentally infected bithynid snails, its first intermediate hosts. The test is based on the fluorescence melting curve analysis of a hybrid between an amplicon from the pOV-A6-specific probe sequence, a 162-bp repeated sequence specific to O. viverrini and specific fluorophore-labeled probes. The real-time FRET PCR could detect as little as a single cercaria artificially introduced in a pool of 30 non-infected snails. The O. viverrini-infected snails were discriminated from non-infected snails and from genomic DNA of other parasite DNAs by their melting temperatures. Sensitivity and specificity of this method were both 100%. Melting curve analysis is a sensitive alternative for the specific detection of O. viverrini-infected snails; it is rapid, allows a high throughput, and can be done on small samples. The assay not only has a high potential for epidemiological surveys of O. viverrini-infected bithynid snails, but also for the detection of cercariae infestations of natural waterways when monitoring transmission sites. www.ncbi.nlm.nih.gov/pubmed/18512076
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Post by skyship on Aug 12, 2013 13:21:14 GMT -5
O. viverrini-infected bithynid snail The ecology of the Bithynia first intermediate hosts of Opisthorchis viverrini. Petney T, Sithithaworn P, Andrews R, Kiatsopit N, Tesana S, Grundy-Warr C, Ziegler A. Source Department of Ecology and Parasitology, Karlsruhe Institute of Technology, Kornblumenstrasse 13, Karlsruhe, Germany. Petney@kit.edu Abstract Opisthorchiasis, together with its associated cholangiocarcinoma, is one of the most important human parasitic diseases on continental Southeast Asia. A great deal of epidemiological data from humans is available on this disease, particularly from the northeast of Thailand, however, only limited information is available on those aspects of the life cycle relating to its Bithynia (Gastropoda) and cyprinid fish intermediate hosts. Here we review the information which is available on the Bithynia hosts of Opisthorchis viverrini. Only one major ecological study has been carried out at one site on a single species of Bithynia. We show not only that detailed ecological studies are required to clarify the epidemiology of opisthorchiasis, but also that the taxonomic status of the Bithynia species transmitting O. viverrini requires clarification. www.ncbi.nlm.nih.gov/pubmed/21821148
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Post by skyship on Aug 12, 2013 13:26:51 GMT -5
Benthos: www.stroudcenter.org/research/projects/schuylkill/taxa/images/taxon28.jpgbithynid snail and benthos, in the waters? or out? in soil? the snail the carrier or are certain water bugs, midges (blood worms)? the mutated worms? Land snails? carriers. some symbiosis going on? ============== "Operculum (Snails) by Matt Law BithyniaOperculumCorporationStTaunton.jpg Bithynia tentaculata operculum from a medieval ditch in Taunton, Somerset, UK. Photo by Matt Law An operculum is a hard permanent seal attached to the foot of some gastropod molluscs (often known as operculates) which can seal the mouth of the shell when the snail is inactive. By doing this, the snail can minimise moisture loss (Evans 1972, 92). Opercula are made of conchiolin,although in some species it is partly calcified (Graham 1971, 12). They are built up in concentric rings or spiral increments as the shell grows (Kerney & Cameron 1979, 16). Opercula have the potential to be well preserved archaeologically, and may even survive in higher numbers than their associated shell. Penkman et al. (2011) used amino acid racemisation of British Bithynia tentaculata opercula to develop a terrestrial correlation for the deep sea foraminiferid oxygen-isotope derived Marine Isotope Stage (MIS) chronology of the Quaternary. Opercula rather than shells were chosen because they have greater stability than the aragonitic shells. References Evans, J.G., 1972, Land Snails in Archaeology. London: Seminar Press Graham, A., 1971. British Prosobranchs. Synopses of the British Fauna (New Series) No. 2. London: Academic Press Kerney, M.P., and Cameron, R.A.D., 1979. A Field Guide to the Land Snails of Britain and North-west Europe. London: Collins Penkman, K.E.H., Preece, R.C., Bridgland, D.R., Keen, D.H., Meijer, T., Parfitt, S.A., White, T.S. and Collins, M.J., 2011. A chronological framework for the British Quaternary based on Bithynia opercula.Nature, 2011, 476, pp. 446-449." www.wikiarc.org/Operculum-SnailsAll those skeletons they are finding in UK? King Tut? archeology? NO....the truth is there...............WE CAN't HAVE IT~!
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Post by skyship on Aug 12, 2013 13:36:52 GMT -5
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Post by skyship on Aug 12, 2013 13:40:11 GMT -5
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Post by skyship on Aug 12, 2013 13:49:10 GMT -5
" Another important interaction is between B. tentaculata and the waterbirds of the Upper Mississippi River(UMR), specifically Lake Onalaska. During the 2006 spring migration, an estimated 22,000-26,000 birds died on the Upper Mississippi River National Wildlife and Fish Refuge (Sauer, et al. 2007). The majority of the bird deaths were lesser scaup, and American coots, but others were also affected such as the northern pintail, American wigeon, northern shoveler, blue winged teal, mallard, American black duck, gadwall, redhead, ring-necked duck, bufflehead, tundra swan, hering gull, and the ruddy duck (Sauer, et al. 2007). Cause of death was found to be from infection by two trematode parasites, Cyanthocotyle bushiensis and Sphaeridiotrema globulus (Sauer, et al. 2007). And sure enough, our friend the faucet snail is the vector passing trematode infection to the waterbirds that eat this snail. B. tentaculata is the first and second intermediate host in the trematode life cycle (Sauer, et al. 2007). " ...." The trematode, Sphaeridiotrema globulus, causes infected birds to be lethargic, have bloodstained vents and have lesions and inflammation in their small intestine (Cole and Friend 1999). S. globulus feeds on blood, resulting in severe blood loss and the bird most often dies from hypovolemic shock (Cole and Friend 1999). Cyanthocotyle bushiensis is found in the lower intestine and cecae of infected waterbirds where it causes hemorrhage and plaque formation eventually blocking the intestines and weight loss (Cole and Friend 1999). The bird most often dies from vascular leakage causing dehydration (Cole and Friend 1999). This parasitic infection is so detrimental to these waterbirds because only a small number of trematodes is needed to cause death."........ ..." Trematode Life Cycle: (Cole and Friend 1999) Trematode eggs are released into the water through the feces of infected birds. These eggs become free-swimming and infect the primary intermediate host, Bithynia tentaculata and continue to develop. After some development, the parasite is called cercariae and is released from B. tentaculata. This free-swimming cercariae becomes encysted on as metacercariae on or in the second Creative Commons Copyright intermediate host, which is also B. tentaculata. When waterbirds consume these snails, they become infected and the trematode life cycle continues. ".. bioweb.uwlax.edu/bio203/2010/schmidt_ama3/interactions.htmUsed to kill the mollusks? ???killed the birds~!~!~!~! this is a trematode, not a nematode. but the mutations? for control of mollusks? DOE/Genomes to Life? project ? bioengineering the oceans. ?? Save the environment: contaminate the people? birds, water, OOPS~! ============= bioweb.uwlax.edu/bio203/2010/schmidt_ama3/interactions.htm
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Post by skyship on Aug 12, 2013 14:09:47 GMT -5
more from link: ......"Cause of death was found to be from infection by two trematode parasites, Cyanthocotyle bushiensis and Sphaeridiotrema globulus (Sauer, et al. 2007). And sure enough, our friend the faucet snail is the vector passing trematode infection to the waterbirds that eat this snail. B. tentaculata is the first and second intermediate host in the trematode life cycle (Sauer, et al. 2007). "... ==================== On another angle: "Keywords: strongyloides stercoralis; S. fulleborni; Cochin China Summary Strongyloides stercoralis might be described as the “military worm”. It was first described by a military physician in soldiers returning from war and in recent times much work on diagnosis, immunology and epidemiology has been conducted on veterans of World War II and Vietnam. The organism was first described in 1876 in French soldiers returning from Cochin China (now Vietnam) who were suffering from intractable diarrhea. onlinelibrary.wiley.com/doi/10.1002/0470842504.ch18c/summaryI think we are going into the jungle, and we are finding some stuff~! I have almost 3,000 images of just the organisms. a few filaments, telechelic polymers, spheres, charged particles, new carbon (fullerenes), ...... A lot of symbiosis going on: all on my arm, it is localized, but that is okay, because knowing this is chronic, which I have thought a long time, I can deal with this. But, we see eventually, what is wrong. The Missing Neglected Piece is Adam~! I will post those when I can download. Hidden biology eventually surfaces and then we can find the synthetic mimics. Going back some years to move forward again. the 20 foot leap, then back to the smallest measurement, the evolutionary intermediates, E. Darwin. Origin of "evolutionary life" the beginning of the circle, of manmade created forms/patterns/images/but the truth will speak for itself. Is The Missing Neglected Piece Adam~! All for the sake of "selfish gene"? or is it selfish Dawkins? Woese (the 3 kingdoms) or Margulis (Symbiosis)? www.adamthemissinglink.com/
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Post by skyship on Aug 12, 2013 15:03:58 GMT -5
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Post by Williegaf on Sept 12, 2020 17:09:43 GMT -5
Hi, here on the forum guys advised a cool Dating site, be sure to register - you will not REGRET it bit.ly/32hIGxJ
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Post by StephenNit on Sept 19, 2020 9:43:52 GMT -5
Hi, here on the forum guys advised a cool Dating site, be sure to register - you will not REGRET it bit.ly/2RA7I5l
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Post by JustinTox on Oct 17, 2020 9:50:35 GMT -5
Hi, here on the forum guys advised a cool Dating site, be sure to register - you will not REGRET it bit.ly/3lZrYtZ
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