This is the one Rense mentioned on show. One World Whey.
The glutathione that repairs cells.
I wonder if this would help us clean up the debris around the cells?
"What Are The Benefits?
* Boosts the immune system * Possesses anti-aging properties * Helps reduce body fat * Improves the building of lean muscle * Increases energy levels * Enhances the feeling of youth * Boosts sports performance * Speeds recovery from exercise"
We have been able to retain the complete range of biological activity and composition of the health-giving whey protein factors as they are found in nature (i.e., lactalbumins, lactoglobulins, lactoferrins, glycomacropeptides, amino peptides, etc.) so that the resulting protein is perfectly balanced, undamaged, and abundant in life-promoting growth factors and amino acids - such as cystine - and other essential nutrients that are commonly lost or degraded in conventional processing. Cystine is an amino acid that breaks down into two molecules of cysteine. Cysteine then combines with richly available amino acids glycine and glutamate inside the cell to produce glutathione. Glutathione is one of the body's most important antioxidants. It is essential to the liver and entire body for the detoxification of heavy metals or any other toxins. Glutathione binds to heavy metals inside the cells and elsewhere and carries them out of the body. It is also essential for the immune system, and it delays muscular fatigue during exercise. What this means to you is not only enhanced lean muscle growth, enhanced immunity and youthful cellular regeneration, but also improved digestion and assimilation, along with the opportunity to remain healthier and younger - longer.
So, this goes further down in identifying what this is.
It appears that the zinc finger is involved.
Now we are still on 1q42.3
"Interferon regulatory factor 2-binding protein zinc finger; pfam11261" /db_xref="CDD:192729"
Now lets see if we can narrow this down some more.
pfam11261; the zinc finger binder.
pfam11261: IRF-2BP1_2 Interferon regulatory factor 2-binding protein zinc finger IRF-2BP1 and IRF-2BP2 are nuclear transcriptional repressor proteins and can inhibit both enhancer-activated and basal transcription. They both contain N-terminal zinc finger represented in this family and C-terminal RING finger domains.
Interferon Regulatory Factor 2 binding protein 2= also called MGC72189, involves gluco kinase.
So far it is a Havana pseudogene, involves the zinc finger ( ZFC3)
We sought to identify an essential component of the TEAD4/VGLL4 transcription factor complex that controls vascular endothelial growth factor A (VEGFA) expression in muscle. A yeast 2-hybrid screen was used to clone a novel component of the TEAD4 complex from a human heart cDNA library. We identified interferon response factor 2 binding protein 2 (IRF2BP2) and confirmed its presence in the TEAD4/VGLL4 complex in vivo by coimmunoprecipitation and mammalian 2-hybrid assays. Coexpression of IRF2BP2 with TEAD4/VGLL4 or TEAD1 alone potently activated, whereas knockdown of IRF2BP2 reduced, VEGFA expression in C(2)C(12) muscle cells. Thus, IRF2BP2 is required to activate VEGFA expression.
==================== Notice it is called a Network++++++++++++
Phosphotransfer networks and cellular energetics
Intracellular energy transfer
Following the discovery of adenine nucleotides, and their role in cellular energetics as a key link between spatially separated energy transducing processes, the energy transfer concept through the ‘adenylate wire’ was proposed (Lipmann, 1941). Fritz Lipman, the author of this concept, was among the ﬁrst to notice the analogy between the energy-carrying adenine nucleotide system and the electrical circuit. Indeed, basic principles of energy transfer, in terms of the rate and efficiency, apply equally to both industrial and metabolic networks (Peusner, 1974; Jeong et al., 2000).
Now this lecture was 1953 and these people were affiliated with Cold Spring Harbor( fomer eugenics school) now epigenetics school.
the adenylate wire is this: receiver and transporter.......
poly ADP-ribose transferase-1 (PARP-1) as an ATP sensor receiving this energy source by way of a specific adenylate kinase ATP wire (AK) from mitochondrial ATP synthase (F0F1)
..."Regulation of malignant phenotype and bioenergetics by a π-electron donor-inducible mitochondrial MgATPase.
E Kun, J Mendeleyev, E Kirsten, A Hakam, AM Kun, A Fekete, PI Bauer, Z Dunai, R Mihalik
The recognition of poly ADP-ribose transferase-1 (PARP-1) as an ATP sensor receiving this energy source by way of a specific adenylate kinase ATP wire (AK) from mitochondrial ATP synthase (F0F1), and directly regulating cellular mRNA and DNA synthesis, was the first step towards the identification of an effect by PARP-1 that is of fundamental significance.
The molecular target of AK-ATP is Arg 34 of the Zn finger I of PARP-1, which is also a site for cation-π interactions as a target of π-electron donors. We now identify this π-electron receptor site as the second active center of PARP-1 which by interaction with a π-electron donor-inducible MgATPase reversibly controls a malignant vs. non-malignant phenotype through energizing the NADH➝NADP+ transhydrogenase, a reaction which is the metabolic connection of PARP-1 to cell function. The specific enzyme-inducing action of the π-electrons is executed by the PARP-1 -topoisomerase I - DNA complex of the nuclei regulating both the nature and the quantity of cellular enzymes that constitute cell-specific physiology.
Well .... well.... welll....I think that acetone, acetate, acetyl justs keeps popping up its little head every once in a while, just to tease us. but. it just might be related to amyloid after all, especially knowing rubber could make acetyl, so "rubber genes" in the making................
It is that CoA.................. acetylation..................
One of the encodes of epigenetics. Coding is not just in DNA. Histones......again........ changing the chromosome, began right at chromosome 1.
Last Edit: Jul 20, 2011 23:02:56 GMT -5 by skyship
THis sort of sums up a lot in this 1q11.42.3: CoA issue...
A human skin multifunctional O-acyltransferase that catalyzes the synthesis of acylglycerols, waxes, and retinyl esters
Abstract Acyl-CoA-dependent O-acyltransferases catalyze reactions in which fatty acyl-CoAs are joined to acyl acceptors containing free hydroxyl groups to produce neutral lipids. In this report, we characterize a human multifunctional O-acyltransferase (designated MFAT) that belongs to the acyl-CoA:diacylglycerol acyltransferase 2/acyl-CoA:monoacylglycerol acyltransferase (MGAT) gene family and is highly expressed in the skin. Membranes of insect cells and homogenates of mammalian cells overexpressing MFAT exhibited signiﬁcantly increased MGAT, acyl-CoA:fatty acyl alcohol acyltransferase (wax synthase), and acyl-CoA:retinol acyltransferase (ARAT) activities, which catalyze the synthesis of diacylglycerols, wax monoesters, and retinyl esters, respectively.
Furthermore, when provided with the appropriate substrates, intact mammalian cells overexpressing MFAT accumulated more waxes and retinyl esters than control cells. We conclude that MFAT is a multifunctional acyltransferase that likely plays an important role in lipid metabolism in human skin.—
Now, lets not get excited here, this was just the beginning..........Notice the: membranes of insect cells, homogenates of mammalian cells, acyl-CoA:fatty acyl alcohol acyltransferase (wax synthase) acyl-CoA:retinol acyltransferase (ARAT) synthesis of diacylglycerols, wax monoesters, and retinyl esters the lipid metabolism in human skin
=================================================== I do believe that "monoesters" are the formers of monomers, dimers, oligomers, protofibrils, tubules....So, this is a hardened resin, waxy, like amyloids. So, goes back to Bence Jones and before. Bence Jones crystal tapes forms look like tapes. By the way, so far 23 types of amyloid have been determined. Just to let the amyloid folks know.
Thankyou Lil sis, for putting that blood journal article up, because it tells us what is happening to the blood.
The 1953 Lipmann lecture, lists issues (organs) involved with this CoA, from clostridium butylicum or Proteus Morganii..................what do these have to do with the formation of strands?, They build them, called SAM. Self assembling monomers, they knew this in 1953............ Now, the newer form is self assembling molecules, or self assembling microtubules, self assembling you name it!
Sam or same thing................... bottom up nano, to get the electrical receiver into the human body. A lot here, but how and why and what was the purpose, or motive?
Last Edit: Jul 20, 2011 22:48:39 GMT -5 by skyship
membranes of insect cells, homogenates of mammalian cells, acyl-CoA:fatty acyl alcohol acyltransferase (wax synthase) acyl-CoA:retinol acyltransferase (ARAT) synthesis of diacylglycerols, wax monoesters, and retinyl esters the lipid metabolism in human skin
Examples of proto-oncogenes include RAS, WNT, MYC, ERK, and TRK. The MYC gene is implicated in Burkitt's Lymphoma, which starts when a chromosomal translocation moves an enhancer sequence within the vicinity of the myc gene. The myc gene codes for widely used transcription factors. When the enhancer sequence is wrongly placed, these transcription factors are produced at much higher rates. Another example of an oncogene is the Bcr-Abl gene found on the Philadelphia Chromosome, a piece of genetic material seen in Chronic Myelogenous Leukemia caused by the translocation of pieces from chromosomes 9 and 22. Bcr-Abl codes for a receptor tyrosine kinase which is constitutively active, leading to uncontrolled cell proliferation.
These are cancer producing recombinant genes. So, is related to blood cancer......... RAS, WNT, MYC, ERK, and TRK.
RAS: Harvey Ras oncogene Image HRAS.jpg
Cancer occurs when the growth and differentiation of cells in a body tissue become uncontrolled and deranged. While no two cancers are genetically identical (even in the same tissue type), there are relatively few ways in which normal cell growth can go wrong. One of these is to make a gene that stimulates cell growth hyperactive; this altered gene is known as an 'oncogene'.
Ras is one such oncogene product that is found on chromosome 11. It is found in normal cells, where it helps to relay signals by acting as a switch. When receptors on the cell surface are stimulated (by a hormone, for example), Ras is switched on and transduces signals that tell the cell to grow. If the cell-surface receptor is not stimulated, Ras is not activated and so the pathway that results in cell growth is not initiated. In about 30% of human cancers, Ras is mutated so that it is permanently switched on, telling the cell to grow regardless of whether receptors on the cell surface are activated or not.
* tch 1 Overexpression Inhibits Osteoblastogenesis by Suppressing ... Among the Wnt proteins, Wnt 1, 3, 3a, 7a, and 8 activate the canonical Wnt/β-catenin ... cloned into the retroviral vector pLPCX (Clontech, Palo Alto, CA) for the creation ... www.jbc.org/content/281/10/6203.full * Related Searches for creation of WNT protein o Wnt Protein Hair Krt17 o Cancer Signaling Pathways o Canonical Pathway o Wnt Proteins Promote Bone Reg… o Signaling Pathways o Wnts * Wnt antagonists and their use in the diagnosis and treatment of ...
Additionally, while the creation of Fc fusions is generally known as one technique ... A “Wnt protein” is a ligand of the Wnt signaling pathway component which binds to ... ip.com/patapp/US20080299136 * * Access : Creation of genome-wide protein expression libraries ...
Creation of genome-wide protein expression libraries using random activation of gene expression ... I want to purchase this article. Price: US$32. In order to purchase this ... nature.com/nbt/journal/v19/n5/full/nbt0501_440.html * advantages of protein - thoughts.com conversation engine
various samples of protine on the market, whey protein ... benefit from, because it aids promote the creation of ... It is perfect to folks who are want to lose weight and ... www.thoughts.com/prestonriver717/advantages-of-protein * Kelly Baggett-Creation of a Bodybuilder | Dr. Squat - Dr. Fred ...
Here is what Kelly Baggett wrote in his article "creation of a bodybuilder" ... The rest of your diet is open to virtually whatever you want. Protein builds the muscle ... drsquat.com/content/kelly-baggett-creation-bodybuilder * Modular protein nanostructures.html - 2009.igem.org
Approach to creation of modular protein nanostructures. Larger natural proteins are with exception of fibrils composed of folded domains. If we want to prepare the material ... 2009.igem.org/Modular_protein_nanostructures.html * Protein therapy speeds bone healing in mice - FierceBiotech Research
Creation of an Sfrp1 lacZ knock-in allele that is null for Sfrp1 function ... A secreted frizzled related protein, FrzA, selectively associates with Wnt-1 protein ... www.ncbi.nlm.nih.gov/pmc/articles/PMC2435376 =============================================== abnormal plasma cells in the bone marrow and overproduction of monoclonal immunoglobulin (IgG, IgA, IgD, or IgE) or Bence-Jones proteins (monoclonal globulin protein found in the blood)
For about a decade, the model proposed by Fearon and Vogelstein has been the paradigm of the genetic alterations involved in the development of colorectal carcinoma. During this time, much information has become available on the function of the key genes in this model, as well as on their interactions. This review examines the impact of this new knowledge on the Vogelstein model. It is concluded that the model as such still stands and with a few modifications could even be strengthened in that, contrary to the original proposal, the order of genetic events seems to be essential. Crucial molecular events include derangement of the Wnt- and defects in the transforming growth factor β (TGFβ)-signalling pathways, which exert a synergistic effect on the cell cycle. Finally, with loss of p53 function, several checks and balances are disrupted, which paves the way to gross chromosomal aberrations and aneuploidy.
So, as long as the models used can be tracked down, then the cancer results will have a CAUSE and it certainly is not from "The First GREAT CAUSE!" This is from a model..
This makes me think that this new artificial transhuman LIFE was created from ART, somebodies' vision of perfection, rather than ART created from LIFE. Humans were already a great piece of ART, Priceless! Diverse! Individual! Unique! Because they knew what "The First GREAT CAUSE was! And they knew who created this "FIRST GREAT CAUSE"! It is self-evident!
tash: Hi skizit, I have watched all your videos on youtube and cant thank you enough for all you have educated me on. I cry for you alot and a bit for me. I was wanting to send you photos of what is raining down everyday here in Australia in hope you can tell me
Dec 11, 2019 23:28:22 GMT -5
tash: not sure where to send them as hush mail and rocket mail bounced back
Dec 11, 2019 23:30:03 GMT -5