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Post by skyship on Nov 12, 2014 2:27:15 GMT -5
I do not know if visitors on this forum have seen this report, but, the images are accurate when you get to the main forms formed by the filaments which could be nano flowers, bulbs, seeds, amphiphiles, micelles, buckyballs on outside of filament forming more filaments. If sea pansies which are really coral, are used, they appear to have pointed masses on their forms. Sea pansies also have bioilluminence. When lesions dry up plaque is everywhere, so coral could form the plaque as the points calcify. These points are what often contain shards, which look often like calcium plates. Once they are pulled out like the "little hairs" in France, pertaining to our good intelligent alchemist, Sir Thomas Browne. In one of his searches for urns, he heard noises from beneath the area where workers were digging for ancient urns. So, could their be a link between Hollow Earth People and their collections of microbes, archaea, and in this case what was found in Brazil. Also Ca, Fl, and Tx where sea coral, red algae(rhodophyta), blue green algae(cyanobacteria), and an archaean proctista. Erasmus Darwin persisted in saying that life began in the ocean. Etiology of Yan (Morgellons) Disease: A New Plant-Like Infectious Organism from Brazil and Its Skin Manifestationswww.la-press.com/etiology-of-yan-morgellons-disease-a-new-plant-like-infectious-organis-article-a3224 The discussion on this topic: images: which are accurate as far as what I have found on deep layers into the lesions. If you download the pdf, you will see the images. I will try to put them here if possible. www.la-press.com/etiology-of-yan-morgellons-disease-a-new-plant-like-infectious-organis-article-a3224
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Post by skyship on Nov 12, 2014 19:50:24 GMT -5
images from the above report which are very similar to mine: 23years and counting. 4os and 50s when we were born. Alien tech forefront. We were the selected targeted because we had the integrated alien tech from the originial aliens........................ IMO............. images from the report from Brazil: Hollow earth tech......come on people,........get real>........ Attachments:
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Post by skyship on Nov 12, 2014 19:57:54 GMT -5
Best I can do with the pictures folks, the Morgellons bastards who do not want us to discover this are controlling the images and the information. ............ so inside the Altnernate news folks we also have liars. and deceivers to save their own arses............. more images: attachment id="574" thumbnail="1"]
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Post by skyship on Nov 12, 2014 23:12:15 GMT -5
The above are the original, then the BioApi came on the scene as evidenced by Len on AQTs thread Artificial Intelligence. But, the organic material was used first, 2nd stage was BioApi, so no one would discover the real Morgellon beginning, and remember the boys in Lanquedoc who got this who would go to under the earth cities to gather the materials. Do you not think at opportune times that Hollow Earth did not send up some specimens for experiment on humans from the "evil force" under earth which also fights the giants, our allies under earth. As above, so below, they are in communications as always for thousands of years.
Wake up folks~!~!~!~!~! skyship
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Post by skyship on Dec 9, 2014 15:31:17 GMT -5
Alien tech and nano, fits right in there with the AI nanotube seeds. So if look for nanotube seeds, roots, fibers and tubular masses, these could all be from gmo seeds which now have nanoseeds in them to help them grow faster. ? Again agrobacterium is the catalyst in the seeds maybe? those tubular masses could be the masses we expel. ==================================== The mechanism of pleural inflammation by long carbon nanotubes: interaction of long fibres with macrophages stimulates them to amplify pro-inflammatory responses in mesothelial cells "Carbon nanotubes have been found to cause a range of pathogenic effects, oxidative stress [3] inflammation and NLRP3 inflammasome activation [4], fibrosis [5,6] and genotoxicity [7]. " www.particleandfibretoxicology.com/content/9/1/8---------------------------- image: compare to the Yan pictures: full screen: www.particleandfibretoxicology.com/content/9/1/8/figure/F3Figure 3. Uptake of CNT by macrophages. Light micrographs (A-G) of THP-1 cells untreated (D) or treated with the CNT panel. NPCB (A), NTshort (E), NTtang1 (B) and NTtang2 (F) appear as aggregates within the cells (white arrows). However NTlong1 (C) and NTlong2 (G) appear to be protruding from the cells (white arrows). Scale bar indicates 20 μm. SEM images (H-K) show an untreated THP-1 cell (H) and THP-1 cells treated with NPCB (I), NTshort (J) and NTlong2 (K). No particles can be seen associated with the cell surface or protruding from the cells in I or J however the fibre from the NTlong2 (K) sample appear on the surface of the cells and also protruding from the cell. Scale bar indicates 10 μm.
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Post by skyship on Dec 9, 2014 15:50:54 GMT -5
Hollow earth theory or not, does not matter, because this is the ai used to make seeds grow, and do those seeds continue to grow, keeps the fruit, veg human from degrading as most organic food will? So the nano replicator could over-ride just the seed, be in roots be in the end product, the continuing replicating, cloning seed inside the human body, just like a real seed, but has added growth. Don't eat the seeds in tomatoes, etc......... ============ Peritonitis along with pleural(lung)proof in abdominal cavity (mice) same for human? Fibromyalgia is related to Morgellons, connective filaments. Carbon nanotubes introduced into the abdominal cavity of mice show asbestos-like pathogenicity in a pilot study Carbon nanotubes1 have distinctive characteristics2, but their needle-like fibre shape has been compared to asbestos3, raising concerns that widespread use of carbon nanotubes may lead to mesothelioma, cancer of the lining of the lungs caused by exposure to asbestos4. Here we show that exposing the mesothelial lining of the body cavity of mice, as a surrogate for the mesothelial lining of the chest cavity, to long multiwalled carbon nanotubes results in asbestos-like, length-dependent, pathogenic behaviour. This includes inflammation and the formation of lesions known as granulomas. This is of considerable importance, because research and business communities continue to invest heavily in carbon nanotubes for a wide range of products5 under the assumption that they are no more hazardous than graphite. Our results suggest the need for further research and great caution before introducing such products into the market if long-term harm is to be avoided. www.nature.com/nnano/journal/v3/n7/full/nnano.2008.111.htmlmore info: sherlock.nature.com/arls/Query?src_jrnl=Nature%20Nanotechnology&src_pp=423-428&family=0&src_vol=3&query=http://xml.nature.com/nnano/journal/v3/n7/xml/nnano.2008.111.xml&src_date=2008&type=LE&src_title=Carbon%20nanotubes%20introduced%20into%20the%20abdominal%20cavity%20of%20mice%20show%20asbestos-like%20pathogenicity%20in%20a%20pilot%20study&prod=NNANO&showall=research============================= Ming Zheng of Dupont Central R&D, Delaware, and Anand Jagota from Lehigh University in Bethlehem, US, developed the technique after discovering a few years ago that DNA could wrap onto carbon nanotubes to form barrel-like structures. www.freerepublic.com/focus/f-news/2288661/posts
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Post by skyship on Dec 9, 2014 16:09:16 GMT -5
Nanoparticle seeds in Pesticides? vermiculite? Lets take Lettuce: Article Impact of non-functionalized and amino-functionalized multiwall carbon nanotubes on pesticide uptake by lettuce (Lactuca sativa L.). ABSTRACT Abstract The effect of non-functionalized and amino-functionalized multiwall carbon nanotube (CNT) exposure, as well as the impact of CNT presence on coexistent pesticide accumulation, was investigated in lettuce (Lactuca sativa L.). Lettuce seeds were sown directly into CNT-amended vermiculite (1000 mg L(-1)) to monitor phytotoxicity during germination and growth. During growth, lettuce seedlings were subsequently exposed to chlordane (cis-chlordane [CS], trans-chlordane [TC] and trans-nonachlor [TN]) and p,p'-DDE (all at 100 ng/L) in the irrigation solution for a 19-d growth period. CNT exposure did not significantly influence seed germination (82-96%) or plant growth. Similarly, pesticide exposure had no impact on plant growth, total pigment production or tissue lipid peroxidation. After 19 d, the root content of total chlordane and p,p'-DDE was 390 and 73.8 µg g(-1), respectively; in plants not exposed to CNTs, the shoot levels were 1.58 and 0.40 µg g(-1), respectively. The presence and type of CNT significantly influenced pesticide availability to lettuce seedlings. Non-functionalized CNT decreased the root and shoot pesticide content by 88% and 78%, respectively, but amino-functionalized CNT effects were significantly more modest, with decreases of 57% in the roots and 23% in the shoots, respectively. The presence of humic acid completely reversed the reduced accumulation of pesticides induced by amino-functionalized CNT, likely due to strong competition over adsorption sites on the nanomaterial (NM). These findings have implications for food safety and for the use of engineered NMs in agriculture, especially with leafy vegetables. www.researchgate.net/publication/261518427_Impact_of_non-functionalized_and_amino-functionalized_multiwall_carbon_nanotubes_on_pesticide_uptake_by_lettuce_%28Lactuca_sativa_L.%29
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Post by skyship on Dec 9, 2014 18:27:48 GMT -5
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Post by skyship on Dec 9, 2014 19:22:41 GMT -5
Now, Mayo just woke up or are now trying to come clean? And you thought the nanotoxic gym socks were bad. Researchers from the Mayo Clinic College of Medicine have found nanoparticles in kidney stones, gall stones, and in the hardened arteries which can lead to heart attacks. All of these conditions are caused by calcium build up, and researchers believe nanoparticles may be the seeds that set the calcium deposits growing. io9.com/376643/nanoparticles-causing-heart-attacks-kidney-stones
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Post by skyship on Dec 9, 2014 19:32:07 GMT -5
Lilys proof cosmetics: One of the questions physiologists want to explore is whether nanoparticles can cause diseases such as atherosclerosis, kidney stones, gall stones and periodontal disease. Dr. Lieske is investigating how nano-sized crystals in the kidney can lead to the development of kidney stones. Dr. Miller has been studying the link between atherosclerosis (hardening of the arteries) and nanoparticles which calcify within the arteries. A fuller audio interview with the researchers is available at www.lifelines.tv. (Episode 7) New technology: promise and peril? Nanotechnology presents intriguing possibilities and some troubling unknowns. The technology is already applied in commercial products as disparate as flame resistant materials and cosmetics. In addition, the technology holds promise in the development of medications that can target precise areas of the body, such as a tumor. nanotechwire.com/news.asp?nid=5755The shards we get are plaque and calcification. black, red and white twisted filaments are surrounding the shard plate like material we remove. The nanofilaments can interact with DNA as shown in the dna wrapping around the carbon nanotubes in above post.
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Post by skyship on Dec 9, 2014 21:06:18 GMT -5
We have to take the nodules into consideration, like the tubular masses of Yan's descriptions. Below are the credentials and papers of the two working on nanoparticle calcifications. ======================== Virginia Miller, PhD www.mayo.edu/research/faculty/miller-virginia-m-ph-d/bio-00077492================ Calcifying nanoparticles promote mineralization in vascular smooth muscle cells: implications for atherosclerosis.Hunter LW1, Charlesworth JE2, Yu S3, Lieske JC4, Miller VM5. Abstract BACKGROUND: Nano-sized complexes of calcium phosphate mineral and proteins (calcifying nanoparticles [CNPs]) serve as mineral chaperones. Thus, CNPs may be both a result and cause of soft tissue calcification processes. This study determined if CNPs could augment calcification of arterial vascular smooth muscle cells in vitro.METHODS: CNPs 210 nm in diameter were propagated in vitro from human serum. Porcine aortic smooth muscle cells were cultured for up to 28 days in medium in the absence (control) or presence of 2 mM phosphate ( positive calcification control) or after a single 3-day exposure to CNPs. Transmission electron-microscopy was used to characterize CNPs and to examine their cellular uptake. Calcium deposits were visualized by light microscopy and von Kossa staining and were quantified by colorimetry. Cell viability was quantified by confocal microscopy of live-/dead-stained cells and apoptosis was examined concurrently by fluorescent labeling of exposed phosphatidylserine. RESULTS:
CNPs, as well as smaller calcium crystals, were observed by transmission electron-microscopy on day 3 in CNP-treated but not P-treated cells. By day 28, calcium deposits were visible in similar amounts within multicellular nodules of both CNP- and P-treated cells. Apoptosis increased with cell density under all treatments. CNP treatment augmented the density of apoptotic bodies and cellular debris in association with mineralized multicellular nodules.
CONCLUSION:
Exogenous CNPs are taken up by aortic smooth muscle cells in vitro and potentiate accumulation of smooth-muscle-derived apoptotic bodies at sites of mineralization. Thus, CNPs may accelerate vascular calcification.
KEYWORDS:
biomineralization; hydroxyapatite; protein-mineral complex; vascular calcification
www.ncbi.nlm.nih.gov/pubmed/24920905
========
Karen, Lil Sis and I talked of this hydroxyapatite before, as well as AQT and phosphorylation. this phosphorus is inorganic so how did it become the nanoseed?able to attach to soft tissue. I now have smooth muscle contractions when in relaxed position after muscle is used. these filaments like amyloid hardens into the shards while other filaments wrap around them. Inside the shards we remove we often find the morgellon filaments which are not organic. They appear polymer and soft plastic, or rubber and pointed at one end which makes them the long needle like CNTs.
===================================
Smooth muscle is found within the walls of blood vessels (such smooth muscle specifically being termed vascular smooth muscle) such as in the tunica media layer of large (aorta) and small arteries, arterioles and veins. Smooth muscle is also found in lymphatic vessels, the urinary bladder, uterus (termed uterine smooth muscle), male and female reproductive tracts, gastrointestinal tract, respiratory tract, erector pili of skin, the ciliary muscle, and iris of the eye. The structure and function is basically the same in smooth muscle cells in different organs, but the inducing stimuli differ substantially, in order to perform individual effects in the body at individual times. In addition, the glomeruli of the kidneys contain smooth muscle-like cells called mesangial cells.
en.wikipedia.org/wiki/Smooth_muscle_tissue
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Post by skyship on Dec 9, 2014 22:08:47 GMT -5
erector pili of skin are called Arrector pili of skin The arrector pili muscles are small muscles attached to hair follicles in mammals. Contraction of these muscles causes the hairs to stand on end[1] - known colloquially as goose bumps.Each arrector pili is composed of a bundle of smooth muscle fibres which attach to several follicles (a follicular unit), and is innervated by the sympathetic branch of the autonomic nervous system. The contraction of the muscle is therefore involuntary - stresses such as cold, fear etc. may stimulate the sympathetic nervous system and thus cause contraction, but the muscle is not under conscious control.Contraction of the muscles has a number of different purposes. Its principal function in the majority of mammals is to provide insulation: air becomes trapped between the erect hairs, helping the animal retain heat. Erection of the porcupine's long, thick hairs causes the animal to become more intimidating, scaring predators. Pressure exerted by the muscle may cause sebum to be forced along the hair follicle towards the surface, protecting the hair. The effectiveness of the muscles' action in humans has been questioned, as humans have relatively little body hair to allow for thermal insulation. en.wikipedia.org/wiki/Arrector_pili_muscleNote the connection to Panniculitis which can be caused by microorganisms, nanoparticles etc. the water side of the janus particle leaks out. The serum......... in bad cases of leaking lesions. The sclerosing of the hair follicle, it becomes white and often plates can be seen in the calcified shard areas of the skin. So those little muscles next to the hair follicles replacing them with nanoparticle or nanotube filaments. Nanowires. I, just found a twisted helical of moving nanowires, they looked ribbon like. These next to the destroyed hair follicle. I have images which I will be putting in a report. Also am trying to repeat the Yan experiment, if possible. dementia, when gets in brain? calcified muscle, veins, arteries etc. nanoparticles, amyloid and smooth muscle destruction. shards, amyloids, plaque and calcificaton all the same thing whether in brain, heart, arteries, gut, or skin. Created by nanoseeds?
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Post by kritters on Dec 9, 2014 22:14:25 GMT -5
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Post by kritters on Dec 9, 2014 22:21:11 GMT -5
erector pili of skin are called Arrector pili of skin The arrector pili muscles are small muscles attached to hair follicles in mammals. Contraction of these muscles causes the hairs to stand on end[1] - known colloquially as goose bumps. Each arrector pili is composed of a bundle of smooth muscle fibres which attach to several follicles (a follicular unit), and is innervated by the sympathetic branch of the autonomic nervous system. The contraction of the muscle is therefore involuntary - stresses such as cold, fear etc. may stimulate the sympathetic nervous system and thus cause contraction, but the muscle is not under conscious control. Contraction of the muscles has a number of different purposes. Its principal function in the majority of mammals is to provide insulation: air becomes trapped between the erect hairs, helping the animal retain heat. Erection of the porcupine's long, thick hairs causes the animal to become more intimidating, scaring predators. Pressure exerted by the muscle may cause sebum to be forced along the hair follicle towards the surface, protecting the hair. The effectiveness of the muscles' action in humans has been questioned, as humans have relatively little body hair to allow for thermal insulation. ahhh... the arrestor pili....viagra research, maybe?
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Post by skyship on Dec 9, 2014 22:28:41 GMT -5
No no that would be the Erector one. Men and their toys~! or their best friends!~!~ LOL.......
But, this temp issue could determine why we sometimes do not realize when we are cold or hot~! We either get too cold, even though the goosebumps would tell us we are cold, or the sweating would tell us we are hot. With Morgellons temperature of skin plays a big part, in my opinion. But, if follicular hairs are destroyed and come out with the filaments, you can see the difference. The follicular hair is empty of melanin. It is hollow.
Skyship
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Post by skyship on Dec 9, 2014 23:39:26 GMT -5
So dandruff is caused by microbe, now? Malassezia globosa what it looks like: Malassezia_globosa images duckduckgo: TScientists from P&G Beauty & Grooming have successfully sequenced the complete genome for Malassezia globosa (M. globosa), a naturally occurring fungus responsible for the onset of dandruff and other skin conditions in humans. Results of the genome sequencing were published the online edition of Proceedings of the National Academy of Sciences (PNAS). Dandruff and seborrheic dermatitis affect more than 50 percent of the human population. Despite the role of Malassezia in these and other common skin diseases, including eczema, atopic dermatitis and psoriasis, little was known about the fungus at the molecular level until this study. In addition, understanding of Malassezia's genetic make-up may help scientists reevaluate the parameters that have historically been used to classify fungal organisms. From www.pgbeautygroomingscience.com/dandruff-genome.html The other person involved in the nanoparticle calcification from Mayo is John C. Lieske, M.D. His credentials: www.mayo.edu/research/faculty/lieske-john-c-m-d/bio-00027820=====================
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Post by kritters on Dec 10, 2014 0:36:40 GMT -5
you are the best, Sky!!!
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Post by skyship on Dec 10, 2014 0:46:53 GMT -5
Krits, The brain keeps thinking outside the box but pulling in the gem scientists. The real Dudes. "Hey Dude" as my lost friend "Cheetah" used to say. I am trying to tap their dude mentality. They gotta be real cowboys~! another image of the fungi Malassezia globosa----looks like some kind of yeast to me~!~!~!~! So fungi and algae are at the core of these nanoseeds.the fungi malassia globosa could be the the fungi and then there are 3 algaes: according to the Yan report Algae could be filament and it is viral as well. There is now viral algae found in human throats. Causing many problems. Fungi can be attacked with Mucinex, but Algae in human throats? www.morgllns.org/jblog/?m=201401
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Post by skyship on Dec 10, 2014 1:56:50 GMT -5
Rhodophyte which is red algae, cryptomonad and glaudocystophyta algae as listed by Yan dude.
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Post by skyship on Dec 12, 2014 5:04:19 GMT -5
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Post by skyship on Dec 14, 2014 0:26:50 GMT -5
Look at the clubbed ends on these filaments~! These came from my microscope at 10X. These are real. Why would I make this up? I have lost much, as many of us have. You think I am doing this for fun?
The hairs when these smaller root filaments latch on are destroyed. The melanin is stolen. So, can we see that some transformation is going on here? Why would real keratin be so colorful?
I am frustrated with shills who come on here and insult to get us to shut up~! I am not afraid of you crappers, because you are no different than paid spies, disinformants with rude "no care" attitudes.
The rest of us here have not been informed of this great find by these Morgellon shills. You know who I am talking about. Us citizen scientists will do the experiments to find out. We will find a way.
Carnicom took as his own, Freeman's findings, but does not give Freeman the credit when he found Cross Domain Bacteria, We still remember what Woese was looking for, at least some of us do.
Anyhow, Krits, we keep on doing what we do~! Tally Ho~!~!~!
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Post by skyship on Dec 14, 2014 1:05:34 GMT -5
Remember Karen and Lil Sis talking of p53? this was proven at U of Mi. This is observation by one of the top univ in the country. Why would they lie about this? This report is not from U of Mi but is significant.
Its receptor, IGF-IR, is synthesized in mesenchymal as well as in epithelial cells. Gene expression has been demonstrated for the dermal papilla and cells of the hair matrix (Hodak et al, 1996;Rudman et al, 1997). In humans, IGF-binding protein 3 is mainly expressed in the dermal papilla of hair- producing follicles (Batch et al, 1996). Further reports reveal the capability of keratinocytes to produce insulin-like growth factor binding protein 3 (Igfbp3) (Wraight et al, 1994). In mouse, IGFBP3 protein has been localized to the zone of follicular apoptosis at the onset of the regression phase of the hair cycle, which has been interpreted as a direct effect of p53 expression (Botchkarev et al, 2000;Botchkarev et al, 2001).........[/quote] Either way you look at this, this mitogen is responsible, for variations abnormalities in dermal papilla cells and hair follicles, but it is the p53 that makes the decision. more on the p53. Mutant p53 Aggregates into Prion-like Amyloid Oligomers and Fibrilswww.ncbi.nlm.nih.gov/pmc/articles/PMC3431633/=========================== Anyone who gets dementia generally has been diagnosed as having diabetes, alz and other prion, amyloid, plaque, calcifications in brain, skin, gut etc. Calcifications with oligomers intermingled in? the seed monomer?
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Post by skyship on Dec 14, 2014 1:42:59 GMT -5
We see the forms as described by Yan. Here others think this is GMO related and it could be? Because we refuse to look at the filaments as they are and appear in the skin, we overlook many things. After all, retinoid, and monsanto, and micelles, janus particles were all patented by the Univ of Pa. So, there could be more than one source of these filaments. here we examine GMO....Monsanto, Bayer, Syngenta etc..... Perplexing fiber disease - GMO driven? Dr. Rima Laibow, medical director for the Natural Solutions Foundation, suspects Morgellons is caused by genetically modified organisms. After studying the skin fibers, it was discovered that they contain DNA of both fungus and bacterium used extensively in GMOs. The fibers consist of cellulose and cannot be made nor broken down by the human body. According to Laibow, "...GM technology apparently has, like Professor Frankenstein found, a way to animate the non-living. These fibers twist and twine, grow and divide. In short, living beneath the skin of people, they form parasitic lesions out of what should be non-living material but which, through the horror of genetic modification, has taken on the characteristics of a living thing." A letter received by Laibow documented how GM 'bio-active textiles' may actually be the cause of Morgellons. A handful of university labs have created textiles with genetically manipulated e-coli bacteria, chemicals, nematodes and proteins infused into their structure. The technology was then sold to textile manufacturers for broad scale applications. The fibers found in the fabric have striking similarities to the fibers found in the skin of individuals suffering from Morgellons. Both are composed of cellulose, have 'helical coil type tendencies' (a genetically manipulated trait), autofluoresce (also seen in genetic manipulation) and contain DNA of genetically modified bacteria. Learn more: www.naturalnews.com/037867_GMO_Morgellons_disease_mysterious_fibers.html#ixzz3Lqs8wWWz
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Post by skyship on Dec 14, 2014 1:54:10 GMT -5
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Post by skyship on Dec 14, 2014 2:10:36 GMT -5
p53, oligomers and GMO nanoseeds:
This goes a bit deeper than what Phantasm has shown due to lack of following the scientists who created such things as Morgellons to all of a sudden find they are wrong. Gotta hit the top to find what is at the bottom...... Follow the "fleur de lis", the real one, folks. ==================== DiscoveryIn 1975–1977, Elizabeth Blackburn, working as a postdoctoral fellow at Yale University with Joseph Gall, discovered the unusual nature of telomeres, with their simple repeated DNA sequences composing chromosome ends. Their work was published in 1978.[3] Elizabeth Blackburn, Carol Greider, and Jack Szostak were awarded the 2009 Nobel Prize in Physiology or Medicine for the discovery of how chromosomes are protected by telomeres and the enzyme telomerase.[4] Nevertheless, in the 1970s there was no recognition that the telomere-shortening mechanism normally limits cells to a fixed number of divisions, or animal studies suggesting that this is responsible for aging on the cellular level and sets a limit on lifespans.[5][6]However, in the early 1970s, Russian theorist Alexei Olovnikov first recognized that chromosomes could not completely replicate their ends. Building on this, and to accommodate Leonard Hayflick's idea of limited somatic cell division, Olovnikov suggested that DNA sequences are lost every time a cell/DNA replicates until the loss reaches a critical level, at which point cell division ends.[7][8] However, Olovnikov's prediction was not widely known except by a handful of researchers studying cellular aging and immortalization.[9] It remained for a privately funded collaboration from biotechnology company Geron to isolate the genes for the RNA and protein component of human telomerase in order to establish the causal role of telomere shortening in cellular aging and telomerase reactivation in cell immortalization.[10]Today, it is established that telomeres protect a cell's chromosomes from fusing with each other or rearranging—abnormalities that can lead to cancer—and so cells are destroyed when their telomeres are consumed. Most cancers are the result of "immortal" cells that have ways of evading this programmed senescence.[5]Another technique, referred to as single telomere elongation length analysis (STELA), was developed in 2003 by Duncan Baird. This technique allows investigations that can target specific telomere ends, which is not possible with TRF analysis. However, due to this technique's being PCR-based, telomeres larger than 25Kb cannot be amplified and there is a bias towards shorter telomeres. Telomere length is associated with the general health of an individual as well as certain diseases, beyond cancer.[57][58] While multiple companies offer telomere length measurement services,[59][60] the utility of these measurements for widespread clinical or personal use has been questioned by prominent scientists without financial interests in these companies.[61][62] Nobel Prize winner Elizabeth Blackburn, who was the co-founder of one of these companies and has prominently promoted the clinical utility of telomere length measures,[63] resigned from the company in June 2013 "owing to an impending change in the control of Telome Health".[64]en.wikipedia.org/wiki/Telomere================================ One must go to the top Phantasm. Get with it~!..........these nobel winners are now falling, resigning and the thinking person can see why.
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Post by skyship on Dec 14, 2014 3:45:00 GMT -5
telomerase.asu.edu/sequences_telomere.htmlHere is the data base, so you wonder why we have so many different variations of Morgellons, it was programmed that way and if we look at Szostak we know these are artificial protocells, we were the original protocell recipients. Kurzweil and his gang at Google including former Darpa wench made this happen. The microchip.........evidence one who worked at a microchip factory in TX is now dead. Please do not underestimate where we will be looking. If Blackburn resigned because of ethics, we understand, or was it because the science was not only misleading, but wrong. When p53 can select death over life, or immoral life then we know it is in the selection process which is all part of the Zionist Bolshevik revolution in science. We are not dummies here, our brains are still functioning, without brainwashing of new tech, tv, and the lying zionists who we have overlooked. They have infiltrated every avenue, every discipline in science and in academeia. We must stand up to these liars. They have mislead many down the path of AI. And this AI is an in vented AI, the real stuff we must still seek, as truth seekers.
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Post by skyship on Dec 14, 2014 3:55:08 GMT -5
So, the telomere nobel prize winners are coming unglued. Precisely, what needs to happen and the source Weizemann Institute. These people are killers. Making p53 destruct rather than extend the cell. p53 is the core.
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Post by skyship on Dec 14, 2014 18:32:54 GMT -5
www.ishitech.co.il/0202ar3.htmOn the other hand upon serum stimulation, when Akt becomes active and enhances cell survival, phosphorylation occurs at an Akt consensus site (serine 166) within the Mdm2 protein, a key regulator of p53 function. www.nature.com/onc/journal/v21/n8/abs/1205181a.htmlSo, Mdm2 protein regulates whether p53 increases cell survival or kills the cell (apoptosis). This involves phosphorylation.
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Post by skyship on Dec 14, 2014 20:08:03 GMT -5
Mdm2 protein, Mdm1, 2, 3, 4? Monoubiquitination is involved in receptor endocytosis, virus budding, transcription, DNA repair, and caspase recruitment in apoptosis, while polyubiquitination generally causes protein degradation (8, 9). Because a chain of at least four ubiquitin molecules is believed to be necessary for efficient proteosomal degradation (15), other proteins must aid in polyubiquitination of p53. At least one report indicates that p300/CBP cooperates with MDM2 in polyubiquitination and degradation of p53. mcr.aacrjournals.org/content/1/14/993.full one report indicates that p300/CBP cooperates with MDM2 in polyubiquitination and degradation of p53
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Post by skyship on Dec 16, 2014 3:48:29 GMT -5
I am not taling about nanobacteria, I am talking about oligomers which are nano seed in monomer and root into the nerve, muscle, and bone. Graphical abstract Glycosyl-nucleoside lipids (GNLs) were previously described as a new supramolecular tool for nucleic acids delivery. Here we describe new glycosyl-nucleoside amphiphiles featuring fluorinated hydrophobic chains (GNFs). These non-toxic fluorocarbon amphiphiles self-assemble into supramolecular structures capable of stabilizing hydrogels. www.sciencedirect.com/science/article/pii/S0040403909023284
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