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Post by kammy on Dec 8, 2010 16:40:03 GMT -5
Project Title Dimethylsulphoniopropionate (DMSP) production of sub-tropical coccolithophores in response to environmental forcing www.biolsci.monash.edu.au/postgrad/current/larsen.htmlAbstract My research, Larsen (2005), indicates the climate system is potentially very sensitive to changes in the flux of ultraviolet (UV) light, via its affect on the marine production of the biogenic trace gas dimethyl sulphide (DMS). DMS is derived from its precursor dimethylsulphoniopropionate (DMSP) which is produced by marine phytoplankton, notably the coccolithophores (Class: Prymnesiophyceae). DMS is emitted from the oceans into the atmosphere where it is oxidised to sulphates, forming the principal source of cloud condensation nuclei (CCN) over the subtropical oceans. These CCN are of major climatic importance as they determine the number of cloud droplets and so radiative properties of maritime clouds. From a climatic perspective, the oceans between the sub-tropics, 30S-30N absorb more than 60% of the incoming solar energy. Within these oceans, the dominant DMSP producers are expected to be the coccolithophores of which the three most important are Emiliania huxleyi, Gephyrocapsa oceanica and Umbellosphaera irregularis , Hagino and Okada (2004). Work to date has focussed on E. huxleyi, to which my study will contribute. No literature exists on the DMSP content, let alone the environmental factors which affect it, for either G. oceanica or U. irregularis . As both species dominate the sub-tropical flora over a larger area than E. huxleyi , and may well be more competitive in a globally warming world, it is vital to understand the role they play. I have proposed that greenhouse warming will increase the sensitivity of the DMS-climate system to natural UV (Larsen, 2008). At the same time, surface UVB levels may also rise in the tropics from changes in the stratospheric circulation (Jiang et al. 2007). In either case, my hypothesis predicts reduced DMS/DMSP production and so a positive feedback to greenhouse warming."
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Post by kammy on Dec 8, 2010 17:16:19 GMT -5
Condensation nuclei are also called Hygroscopic and Hydrophobic nuclei science.jrank.org/pages/45064/condensation-nuclei.html"Microscopic atmospheric particles that attract water droplets, that may then coalesce to form a raindrop. Condensation nuclei (hygroscopic nuclei) come from pollen, salt from sea spray, dust from volcanic eruptions and soil erosion, and particulate air pollution ( C. Park 2001 ). Many small condensation nuclei cause many small droplets to remain liquid well below normal freezing point, and small ice nuclei may increase the rainfall rate from single clouds, which is the principle of cloud seeding ( Graf ( 2004 ) Science 303, 5662). See hygroscopic nuclei. Various stock photos of Hygroscopic nuceli:
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Post by kammy on Dec 8, 2010 17:24:28 GMT -5
Stock photos of Hydrophobic nuceli:
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Post by kammy on Dec 8, 2010 18:16:17 GMT -5
Now I'm looking to see if it has been documented that the DMS(P) can be taken in by humans to cause disease: Searching on: "intracellular dimethylsulfoniopropinate uptake by humans" www.csa.com/discoveryguides/dimethyl/abstracts-f.php"Here we show evidence for cleavage of DMSP in response to environmental cues among different strains of the haptophyte Emiliania huxleyi (Lohmann) Hay et Mohler and the dinoflagellate Alexandrium spp. (Halim). Sparging or shaking live cells of either taxon increased dimethyl sulfide (DMS), especially in dinoflagellates, known to be very sensitive to shear stresses. Additions of polyamines, known triggers of exocytosis in some protists, also stimulated DMSP cleavage in a dose-responsive manner. Toxins from marine and freshwater phytoplankton create health hazards for both aquatic and terrestrial species and can significantly affect human activities and the economic vitality of local communities. A reasonable case can be made that phytoplankton metabolites such as dimethyl sulfide (DMS) link interaction webs that span hundreds to thousands of kilometers and connect production from oceanic phytoplankton to desert cacti and coyotes via zooplankton, fishes, and sea birds. The ecosystem-wide and potentially global consequences of aquatic chemical cues is an underappreciated topic that warrants additional attention. Here we report results that, together with those in the literature, show that DMSP and its breakdown products (DMS, acrylate, dimethylsulphoxide, and methane sulphinic acid) readily scavenge hydroxyl radicals and other reactive oxygen species, and thus may serve as an antioxidant system, regulated in part by enzymatic cleavage of DMSP. In support of this hypothesis, we found that oxidative stressors, solar ultraviolet radiation, CO sub(2) limitation, Fe limitation, high Cu super(2+) and H sub(2)O sub(2) substantially increased cellular DMSP and/or its lysis to DMS in marine algal cultures. Oceanic phytoplankton, atmospheric sulphur, cloud albedo and climate. Charlson, RJ; Lovelock, JE; Andreae, MO; Warren, SG Nature, vol. 326, no. 6114, pp. 655-661, 1987 The major source of cloud-condensation nuclei (CCN) over the oceans appears to be dimethylsulphide, which is produced by planktonic algae in sea water and oxidizes in the atmosphere to form a sulphate aerosol. Because the reflectance (albedo) of clouds (and thus the Earth's radiation budget) is sensitive to CCN density, biological regulation of the climate is possible through the effects of temperature and sunlight on phytoplankton population and dimethylsulphide production. To counteract the warming due to doubling of atmospheric CO sub(2), an approximate doubling of CCN would be needed." *No, this is not what I'm looking for but a very informative page. I'm starting to notice that every category has a 'budget', sulfur budget... radiation budget... carbon emission budget... this is a scary thought - when has our governments ever been able to stay within a budget? I am also noticing that the information about certain factors that can help the scientists has not been documented or easily accessed by them. I wonder what effect oil spills, volcanoes erupting, other industrial pollution has on the algae blooms?
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Post by kammy on Dec 8, 2010 18:41:38 GMT -5
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Post by lilsissy on Dec 9, 2010 0:58:06 GMT -5
Need some time to chew this over, catching up to do.
Jen
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Post by kammy on Dec 9, 2010 6:56:31 GMT -5
This abstract is over my head, the images at Figure 3 are very similar to typical Morgellons, Figure 2 is comparable to the photo I posted above of the particles in blood: Fast Release of Lipophilic Agents from Circulating PEG-PDLLA Micelles Revealed by in ViWo Fo¨rster Resonance Energy Transfer Imaging www.kinam.com/Articles/PMicelles%20Papers/Chen%2008%20Langmuir.pdfand they are calling the micelles in Figure 3 - 'Fret' micelles. Ok, for those of you with medical knowledge... some areas to look in?: Fret micelles Interaction of lipid micelles with blood serum lipoproteins Dysfunctional HDL Cholesterol micelles Ferritin and Ferruginous Micelles in Normal Erythroblasts We need to figure out how do the micelles from the ocean that get into the clouds... that end up in our bodies - where do they go in the human body and what do they do there? lol I'm just assuming they congregate first in the intestines and lungs. ?? Here's where I'm stuck, Jen. Kat mentioned that abnormal micelle formation or production might be seen with the HDL or testing for Diabetes. I saw where 1 in 4 Americans is predicted to have Diabetes in the future... well, here you go. And, I wanted to mention that my good cholesterol (is that HDL or LDL?) has been below normal for many years. You know how doctors were so concerned with following it and now seem to not consider those numbers as important as a few years ago? I'm seeing ZERO, so far, that's written on the micelles from nature, clouds, the atmosphere - and how they directly affect human health. The literature is vague and says fallout can cause certain diseases but I haven't found specifically how it enters the body, etc... still looking.
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Post by kammy on Dec 9, 2010 7:17:55 GMT -5
URACIL I don't have the medical background to put this together that way, I'm the what's happening in nature person. I'm trying to point you gals who are more knowledgeable in this area to look in this direction - around here is what is causing our disease. The best photos of what is happening based on my microscopic observations is involving uracil. I'm just pointing... in my laymen's terms - like the photo of the Cern particles you posted that turned into a white, fuzzy fungus - our particles, micelles are stacking in a chain and also are branching out with filaments to create a fiber network inside of us. Without looking, I think uracil is a product of the liver? Here's a stock photo of uracil: Look at this one below where it has formed a fiber! uracilandlife.org/images/1_4.jpg Here's a site with some good photos: uracilandlife.org/foto_en.htmWhen I put in a search for 'testing for malfunction of uracil', I get many avenues... anyone?: www.google.de/#hl=de&source=hp&q=testing+for+uracil+malfunction&aq=f&aqi=&aql=&oq=&gs_rfai=&fp=533a7c6ffd2d1c5d
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Post by kammy on Dec 9, 2010 7:35:45 GMT -5
I believe that we've been hit by a shotgun blast with many different pellets, over many years that can be adding to our disease. I compared what is happening to a firing squad... one sector is doing this to bring us harm, another is doing something different. In this way, no one company, industry leader, family, and with all major countries participating - noone has to take the responsibility of maiming the planet and the human race. 'It wasn't my bullet that killed that man... it was the guy next to me firing.'
They are using the ultimate weapon against us - how nature and the human body work naturally. Microscopic micelle formation in nature and in the body is the weapon.
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Post by kammy on Dec 9, 2010 7:53:27 GMT -5
Micelle to fiber technology exists and especially in synthetic and nano applications. Here is a brief description of micelle to silk fibers from insects in nature: www.nature.com/nature/journal/v424/n6952/full/nature01809.html"Here we report the identification of emulsion formation and micellar structures from aqueous solutions of reconstituted silkworm silk fibroin as a first step in the process to control water and protein–protein interactions. These micelles subsequently aggregated into larger ‘globules’ and gel‐like states as the concentration of silk fibroin increased, while maintaining solubility owing to the hydrophilic regions of the protein interspersed among the larger hydrophobic regions." Fabrication of Magnetic Spider Silk and Other Silk-Fiber Composites Using Inorganic Nanoparticles onlinelibrary.wiley.com/doi/10.1002/(SICI)1521-4095(199807)10:10%3C801::AID-ADMA801%3E3.0.CO;2-I/abstract
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Post by kammy on Dec 9, 2010 8:14:36 GMT -5
Looking briefly at uracil malfunction: tinyurl.com/2vdfjvs"Introduction Ultraviolet light is known to cause mutations in the URA3 gene of certain strains of yeast (Lab Manual). This gene is the third in a biosynthetic pathway responsible for producing uracil. Uracil is a building block of RNA (Lab Manual). If uracil is unable to be synthesized, RNA cannot be produced and therefore DNA replication cannot proceed. If an organism cannot synthesize its own uracil due to a mutation in one or more genes controlling its biosynthesis (one of which being URA3) and is not provided with uracil on its growth media, the organism will not survive (Lab Manual). Therefore an organism with a URA3 mutation is an auxotroph (Lab Manual)." What about searching on 'uracil mutation'?
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Post by kammy on Dec 9, 2010 8:22:53 GMT -5
www.web-books.com/MoBio/Free/Ch7F.htm"Mutations may be caused by external factors (UV light, chemical agents, etc.) or spontaneous cellular processes (replication errors, accidental deamination, etc.). The following figure illustrates the deamination process. Conversion of the methylated cytosine (methylcytosine) to thymine may explain the observed CpG islands (more info). Cytosine is one of four bases in DNA molecules. As shown in the above figure, it may be mutated to uracil by deamination. Since uracil is not part of DNA, this mutation can easily be detected and repaired by base excision. Suppose DNA, like RNA, were made up of uracil, then the cytosine to uracil mutation could be corrected only by mismatch repair which is very inefficient. This may explain why DNA chooses thymine, instead of uracil, even though the chemical structure of uracil is simpler than thymine."
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Post by kammy on Dec 9, 2010 8:33:19 GMT -5
Base excision repair en.wikipedia.org/wiki/Base_excision_repair"Base excision repair (BER) is a cellular mechanism that repairs damaged DNA throughout the cell cycle. Single bases in DNA can be chemically damaged by a variety of mechanisms, most commonly deamination, oxidation, and alkylation. Uracil inappropriately incorporated in DNA or formed by deamination of cytosine[2]In addition to base lesions, the downstream steps of BER are also utilized to repair single-strand breaks. End processing enzymes In order for ligation to occur, a DNA strand break must have a hydroxyl on its 3' end and a phosphate on its 5' end. In humans, polynucleotide kinase-phosphatase (PNKP) promotes formation of these ends during BER. This protein has a kinase domain, which phosphorylates 5' hydroxyl ends, and a phosphatase domain, which removes phosphates from 3' ends. Together, these activities ready single-strand breaks with damaged termini for ligation. The AP endonucleases also participate in 3' end processing. Besides opening AP sites, they possess 3' phosphodiesterase activity and can remove a variety of 3' lesions including phosphates, phosphoglycolates and aldehydes. 3' processing must occur before DNA synthesis can initiate because DNA polymerases require a 3' hydroxyl to extend from. Links between BER and cancer Defects in a variety of DNA repair pathways lead to cancer predisposition, and BER appears to follow this pattern. Deletion of BER genes increases the mutation rate in a variety of organisms, predicting that loss of BER could contribute to the development of cancer."
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Post by kammy on Dec 9, 2010 8:44:24 GMT -5
"Uracil inappropriately incorporated in DNA or formed by deamination of cytosine[2]" What would this mean, gals? How would it manifest, how to test for it? Does it mean that instead of CGAT, we might have CGAU or CGATU ?
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Post by kammy on Dec 9, 2010 9:39:00 GMT -5
DNA Repair users.rcn.com/jkimball.ma.ultranet/BiologyPages/D/DNArepair.html"Importance DNA in the living cell is subject to many chemical alterations (a fact often forgotten in the excitement of being able to do DNA sequencing on dried and/or frozen specimens ). If the genetic information encoded in the DNA is to remain uncorrupted, any chemical changes must be corrected.
A failure to repair DNA produces a mutation.
The recent publication of the human genome has already revealed 130 genes whose products participate in DNA repair. More will probably be identified soon.
Agents that Damage DNA
Certain wavelengths of radiation ionizing radiation such as gamma rays and x-rays ultraviolet rays, especially the UV-C rays (~260 nm) that are absorbed strongly by DNA but also the longer-wavelength UV-B that penetrates the ozone shield . Highly-reactive oxygen radicals produced during normal cellular respiration as well as by other biochemical pathways.
Chemicals in the environment many hydrocarbons, including some found in cigarette smoke Link to description of a test measuring the mutations caused by the hydrocarbon benzopyrene.
some plant and microbial products, e.g. the aflatoxins produced in moldy peanuts Chemicals used in chemotherapy, especially chemotherapy of cancers
Repairing Damaged Bases
Damaged or inappropriate bases can be repaired by several mechanisms: Direct chemical reversal of the damage Excision Repair, in which the damaged base or bases are removed and then replaced with the correct ones in a localized burst of DNA synthesis. There are three modes of excision repair, each of which employs specialized sets of enzymes. Base Excision Repair (BER) Nucleotide Excision Repair (NER) Mismatch Repair (MMR)
Direct Reversal of Base Damage
Perhaps the most frequent cause of point mutations in humans is the spontaneous addition of a methyl group (CH3-) (an example of alkylation) to Cs followed by deamination to a T. Fortunately, most of these changes are repaired by enzymes, called glycosylases, that remove the mismatched T restoring the correct C. This is done without the need to break the DNA backbone (in contrast to the mechanisms of excision repair described below)."
"that remove the mismatched T restoring the correct C."
TGAT instead of CGAT ?
(Wondering... so, if our DNA has been mutated, is this a precursor to cancer?)
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Post by kammy on Dec 9, 2010 11:04:54 GMT -5
This is interesting. I was just getting ready to close that uracil page I had open uracilandlife.org/foto_en.htm and I see the 1st photo: uracilandlife.org/images/01.jpgI don't know where they got this specimen, it doesn't say but it reminded me of this same stuff that grew our artifacts that came out of my tap water. This is a microscopic photo at 100x running my bathroom sink water straight into the Petri dish, it looks very similar. Another bullet in their gun?: tinypic.com/r/k4iez9/7Kat wrote an article on the two different types of uracil... let me see if I can find her blog article? If I remember, there are two types of uracil, one is natural and the other man-made and one is spelled with a 'c' and the other a 's' - urasil. Just thinking - they are overloading, overpowering our natural uracil with man made to cause our CGAT - 'T' to turn into a 'U'? Piles and piles of micelles in my tap water... I said, 'what the hay?'... tinypic.com/r/k4iez9/7tinypic.com/r/14j3o8w/7
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Post by kammy on Dec 9, 2010 11:38:34 GMT -5
I just scrolled down this page again, it's giving us the best clues for the medical terminology of what's happening inside the body: uracilandlife.org/foto_en.htm4.4 Blood samples taken from a patient who was diagnosed as cancer of bowels: see the development of hypha and cellulose micelles from prochlorons. uracilandlife.org/images/4_7.jpg4.7 Fibril and cellulose vesicles in a BSE preparation. This image 4.6 shows the best of what's happening:uracilandlife.org/images/4_6.jpg"4.6 A preparation from a patient who was diagnosed as thyroid hypertrophy: see the fibrille, nodule and reticular cellulose vesicles." You see the hyphae 'hairs' coming out around the micelles? This is representative of our white, fuzzy 'fungus', this is how it is created. Terminology to look at:hypha and cellulose micelles prochlorons Fibril and cellulose vesicles fibrille, nodule and reticular cellulose vesicles hypertrophy
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Post by kammy on Dec 9, 2010 11:58:13 GMT -5
(Very Suspect) Prochlorons microbewiki.kenyon.edu/images/e/e8/Cell.jpg"Bacteria; Cyanobacteria; Prochlorales; Prochloraceae Description and Significance Species of the Prochloron genus are oxyphotobacteria, or oxygenic photosynthetic prokaryotes. They are most prominent in tropical waters as symbionts with ascidians, commonly known as sea squirts. These organisms' unique photosynthetic morphology make them interesting to scientists who study the evolution of the features in photosynthesizers. Prochlorons are distributed within the tissues of didemnid ascidians, mainly in the cloacal cavities under the upper tunic of the organism (see above image). Ascidians, or "sea squirts", are primarily known as a nusciance to humans. These sessile tunicates are found on most solid marine surfaces, including rocks, reefs, and algae."
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Post by kammy on Dec 9, 2010 12:10:04 GMT -5
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Post by kammy on Dec 9, 2010 13:46:16 GMT -5
Prochlorons are distributed within the tissues of didemnid ascidians Prochloron didemni microbewiki.kenyon.edu/index.php/Prochloron_didemniProchloron didemni are photosynthetic cyanobacteria with a twist. They carry out photosynthesis similar to eukaryotic plants, with two reaction centers in noncyclic electron flow.(7) However, unlike cyanobacteria, P. didemni have chlorophyll a and b and lack phycobiliprotiens.(2) Photosynthesis takes place in the thylakoids, with the thylakoids concentrated toward the periphery of the cell.(7) The cell wall of Prochloron didemni consists of peptidoglycan and muramic acid of the A1 γ type. Approximately 40% cross-linkage exists in its cell wall with a network of five to eight layers and a thickness of 12nm.(9) Ecology Prochloron didemni is an obligate symbiont that lives in the ascidian Lissoclinum patella. L. patella along with P. didemni can be found in tropical waters, such as the reefs in Hawaii and the Great Barrier Reef in Australia.(2,7) Lissoclinum patella - suspect
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Post by kammy on Dec 9, 2010 13:57:41 GMT -5
Alright - let me look at hypha and cellulose micelles as per post 93 to go by: reference.findtarget.com/search/biopolymer/"Biopolymers are polymers produced by living organisms. Cellulose, starch, chitin, proteins, peptides, DNA and RNA are all examples of biopolymers, in which the monomeric units, respectively, are sugars, amino acids, and nucleotides. Cellulose is both the most common biopolymer and the most common organic compound on Earth. About 33 percent of all plant matter is cellulose. E.G. The cellulose content of cotton is ~ 90 percent and that of wood is ~ 50 percent. Cellulose Many polymer researchers believe that polymer chemistry had its origins with the characterization of cellulose. Cellulose was isolated for the first time some 150 years ago. Cellulose differs in some respects from other polysaccharides produced by plants, the molecular chain being very long and consisting of one repeating unit (cellobiose). Naturally, it occurs in a crystalline state. From the cell walls, cellulose is isolated in microfibrils by chemical extraction. In all forms, cellulose is a very highly crystalline, high molecular weight polymer, which is infusible and insoluble in all but the most aggressive, hydrogen bond-breaking solvents. Because of its infusibility and insolubility, cellulose is usually converted into derivatives to make it more user friendly in processing. Some fungi can secrete enzymes that catalyze oxidation reactions of either cellulose itself or the lower molecular weight oligomers produced from the enzymatic hydrolysis of cellulose. Of these, the peroxidases can provide hydrogen peroxide for free radical attack on the C2–C3 positions of cellulose to form ‘aldehyde' cellulose, which is very reactive and can hydrolyze to form lower molecular weight fragments. Bacteria also secrete enzymes, some of which form complexes that act jointly in degrading cellulose to form carbohydrate nutrients which the microorganisms utilize for survival."
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Post by kammy on Dec 9, 2010 14:21:46 GMT -5
It may seem like I'm jumping around quite a bit - however, I'm trying to stay in one place, it just happens to be a big place... lol I'm trying not to venture out too far from what is happening in the ocean that could possibly be ending up in our bodies via the DMS micelle evaporation route to the clouds and then rain. As I stated before, I believe that 'others' can tag along with the DMS cell that's where looking at possible marine fungi, bacteria, organisms comes in. Back to - Fibril and cellulose vesicles: highered.mcgraw-hill.com/classware/ala.do?isbn=0072464631&alaid=ala_661120 "Membrane-Assisted Transport In exocytosis, a vesicle formed by the Golgi apparatus fuses with the plasma membrane as secretion occurs; insulin leaves insulin-secreting cells by this method. During endocytosis, cells take in substances by vesicle formation as plasma membrane pinches off by either phagocytosis, pinocytosis, or receptor-mediated endocytosis. In phagocytosis, cells engulf large particles (e.g., bacteria), forming an endocytic vesicle. Phagocytosis is commonly performed by ameboid-type cells (e.g., amoebas and macrophages). When the endocytic vesicle fuses with a lysosome, digestion of the internalized substance occurs." *Ok, I'm going to stop here and note that Cellulose needs to be looked into further in the fiber production along with the membrane-assisted transport systems and their roles.
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Post by kammy on Dec 10, 2010 9:17:33 GMT -5
A duplicate post from the stream thread, and to have everything in one place - as I feel it's important in trying to convey what it is I'm trying to get across: This molecule/micelle of DMSO is the same as what is exploding and creating our 'fungus' and fiber network, except I believe we have a DMS(P) version: Stock Photo of DMSOHuman lesion 100x, these micelles 'explode' the filaments out of them creating a spider web-like network: tinypic.com/r/2myxoas/7Baraka captured it the best in his "Morgellons Forming" video: www.youtube.com/watch?v=LPodyogcOzg
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Post by kammy on Dec 10, 2010 9:18:51 GMT -5
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Post by kammy on Dec 10, 2010 9:21:46 GMT -5
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Post by kammy on Dec 10, 2010 9:23:40 GMT -5
Duplicate post: What is it about D.M.S.O. that you find interesting kammy , am I missing something. I know it opens up the barriers in the body. I have some, Jen I believe that is a old horse lineament that is help full for arthritis but if you have a parasite it can help it travel deeper in the body , I have heard. Jen I don't know for sure if DMSO is a good product to treat external lesions with or not? Possibly with something else, I suspect it might be. I'm pretty sure that by itself it is not going to be very effective, it's just a carrier. If its relative DMS is the main culprit carrier, then what effect would adding DMSO have?... I don't know. (Fight fire with fire, like cures like?) I'm thinking (so this is being said without the research) this overabundance of micelle production is happening in the gut lining, we need something to soothe the lining, neutralize or raise the PH in the gut... drinking baking soda, vinegar, salt water/C...? Carnicom did PH testings... I can't remember the results... have to look. I think it's forming the fibers under our skin and not necessarily in the gut. The micelles are traveling through the blood and ending up trapped places under the skin because they trying to exit the skin's pores. Something (uracil?) a stress of some kind causes them to start their explosions such as in Baraka's video or start stacking to form fibers (free radicals?) as Carnicom and I have captured, and that if enough congregate in one place and can't get out the pores - we get lesions. And since the head has the largest pores, that's why we're having scalp involvement, they are more concentrated there in trying to escape. Maybe we need to sweat them out? We're probably breathing them out too... we need to exercise more to rid them. Drink more water, regular bowel movements... detox. I see these what I have referred to as 'carbon balls'/micelles are like cells themselves - like cancer cells that are reproducing wildly and out of control, the more we allow them to do this internally - the sicker we get. Our disease is an over-abundance of these micelles that can't be rid quick enough, remember they start out nano in size, Carnicom talks about barely seeing them at 10,000x but they grow to larger sizes to where one can easily fill up a lens at 100x! These environmental micelles are naturally in the air we breathe, our water and food - their characteristic is that of a 'carrier', or readily picks up hitchhikers. Our environment is being negatively altered with every new bit of pollution we encounter to put more in our environment or to cause our natural, internal micelle production processes to go haywire or to be corrupted. Our internal micelles have picked up something bacterial/fungal/parasitical from the environment or from association with incoming micelles from the environment and have now incorporated these pathogens into them. Or, we have two different forms of micelles inside of us now, a toxic environmental one or more and the natural that are separate of each other. If the micelle mixture in the environment lessens, hopefully our internal loads will lessen also, as I hope these micelles that are formed naturally can be corrected to preform like they used to. We're trying to figure out what our internal micelle production processes are in the other thread so that we can lead doctors to test in certain areas. (I'm thinking we need at the least - Gastroenterology or Internal Medicine.)
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Post by kammy on Dec 10, 2010 11:03:59 GMT -5
I'm looking again to see if anyone else SCREWED UP ;D and left a microscopic photo of DMS(O)(P) out there for us to go by: Search criteria - 'DMSO microscopic photos': Interesting that this abstract would show up?: In vitro gene transfer using human papillomavirus-like particles nar.oxfordjournals.org/content/26/5/1317.fullThese HPV-16 look similar to the unidentified 'flower-looking' micelle just above that is more in a viral form that Carnicom showed: Here's the Carnicom photo from above that I'm referring to: -------------------------------------------- HIGH RESOLUTION SEM OF INTRINSIC MUSCLE FIBERS OF ANTERIOR THIRD RAT'S TONGUE Here's an abstract that shows the micelles in Figure 2: www.scielo.cl/scielo.php?pid=S0717-95022003000400006&script=sci_arttext"Fig 1 Light microscopy showing the muscle fiber bundles of the anterior third of the rat tongue. Collagen fibers in the lamina propria (\) and muscle fiber groupings are seen (arrows). Azo carmin X88 Fig 2 Muscle cells containing periphery nuclei are clearly shown. (arrows). HE X585 Fig 3 Polarized light microscopy view showing collagen fiber bundles surrounding the muscle fibers (perimysium) (arrow). Picro-sirius X220. Fig 4 Polarized light microscopy view showing a network of variable spaces (\) and revealing collagen fibers in the endomysium (arrows). Picro-sirius X585." * Figure 2 is dead ringing pointing to the micelle calling them "periphery nuclei", another name for our micelle might be periphery nuclei? This abstract goes on to talk about the: "The collagen fiber network that covers the muscle fibers form trabeculae delineating the original spaces of each muscle fiber..." that we were just looking at in fiber formation. What's 'trabeculae'? "Fig. 11. Muscle fiber surface freeze-cracked with DMSO. Image under HRSEM reveals the intracellular structures arranged in parallel directions. X.800. Fig. 12. Sarcoplasm freeze-cracked with DMSO. Mitochondria (small arrows) are vertically arranged between the bundles of myofilaments (large arrows). X5,800. Fig. 13. Muscle fiber surface freeze-cracked with DMSO and macerated in diluted solution of osmium. Shows the internal cytoplasmic structures and the basement membrane (\) containing a network of collagen fibers. X16.200. Fig. 14. Basement membrane displaying a meshwork structure with thin and thick collagen fibrils in a three-dimensional SEM image (arrow). X18.000. Fig. 15. At high magnification, the basement membrane shows a sponge-like structure and a few collagen fibrils (arrows). X110.000. Fig. 16. Image under HRSEM reveals a number of large and small mitochondria (M). X18.000." *In Figure 14 you can see an evident micelle right next to the arrow: "Fig. 14. Basement membrane displaying a meshwork structure with thin and thick collagen fibrils in a three-dimensional SEM image (arrow). X18.000." They are calling what I am calling the hyphae of the white, fuzzy 'fungus' - "thin and thick collagen fibrils".WE NEED TO LOOK CLOSER AT COLLAGEN FIBER FORMATION... thinking - is it now that our naturally produced collagen now has a 'fungal/bacterial' aspect to it? I am not sure what they are looking at here in Figure 19, cristae mitochondriales? "Fig. 19. At high magnification. cristae mitocondriales are projected from the internal layers of the membrane The lamellae of cristae mitochondriales are clearly visible (arrows). X81.000. At high magnifications, many spherical-shaped particles were observed attached to the surface of the mitochondrial cristae, corresponding to the inner membrane, which contains ATPase (Tanaka, 1989)." periphery nuclei? trabeculae? thin and thick collagen fibrils? cristae mitochondriales? anyone?
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Post by kammy on Dec 10, 2010 12:01:19 GMT -5
Search criteria still - 'DMSO microscopic photos': mcr.aacrjournals.org/content/8/9/1217.abstract"Although autophagy is generally considered a prosurvival mechanism that preserves viability, there is evidence that it could drive an alternative programmed cell death pathway in cells with defects in apoptosis." ------------------------------- Here's a before and after of DMSO showing no visible micelles before and yes, afterward. www.bli.uci.edu/images/dmso.jpg---------------------------------------------- Interesting: www.gmicheli.it/momu.html"Chromatin Nuclear content of Xenopus laevis oocytes. Preparation: dispersal of chromatin in low ionic strength buffer, followed by fixation in sucrose/glutaraldehyde and adsorption on activated carbon films ('Miller spread'). TEM imaging upon rotary shadowing with platinum. Upper panel: beaded chromatin fibers (the beads, representing nucleosomes, have a diameter of about 120 Å). Lower panel: 'Christmas tree'-like arrangement of nascent ribosomal RNA transcripts along a portion (about 8 kb) of an rDNA repeat unit." *I've got photos of something very much like this happening: "Heteroduplex. Replication fork TEM imaging upon cytochrome-c/formamide spreading, uranyl acetate staining and rotary shadowing with platinum. Large panel: molecular hybrid (heteroduplex) between the antibiotic resistance transposons Tn21 and Tn1935. Tn1935 contains the entire sequence of Tn21, as well as two additional regions (0.97 kb and 2.7 kb, separated by a 7.8 kb stretch), corresponding to the genes for resistance to ampicillin and kanamycin, which appear as single-stranded DNA loops. Inset: replication fork in replicating DNA from Carassius auratus. The single stranded region at the fork point is about 120 Å long (35 bp)." Questions: Chromatin? beaded chromatin fibers (the beads, representing nucleosomes)? Heteroduplex? Carassius auratus?
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Post by skyship on Dec 10, 2010 12:25:08 GMT -5
The notochord is important, is related to what was used, the electric organ from a sting ray, This was used in studies in 40s and 50s, then the studies went further and I remember the studies some of them on other notochords, and the tunicate was one of them.
Thank you for this info Kammy, it is making sense.You mentioned this, and I know the issue with me involves muscular dystrophy, so I do know the dystrophin issue well, but, that one photo (5) shows what I have been pulling out, and it has meandering spike like features, that branch off the main fiber. They are like spikes in cactus, about the only way to describe them.
trabeculae, shows in spleen, related to lymphs?www.histol.chuvashia.com/atlas-en/lymphoid-en.htmahhhhhhhhhhh so it is mysterious?Transverse & Longitudinal Sections of a Selaginella Stem revealing the Mysterious Trabeculae!www.botany.hawaii.edu/faculty/webb/BOT201/Selaginella/Selaginella-3.htm===================== Apical cell, just as I thought, you covered this and came very close, in the studies on Superbug.
Botany, relationship to human extracellular spaces, but there also was a spleen reorganization from the cytoskeleton reconstitution, which again involves the histones,
neurospora crassa has an apical complex called the Spitzenkorper, that could be the apical former. Will check into that.
n. crassa was supposed to have been used in lab studies only, but, if they knew the mobile fiber was in this and could form, then this may have been used to create the Human Cell Less System based on spores, in otherwords the outside system of cells would be made of spores, not cells, does that make sense? But, this secondary system would be what is used to control the real human dna. or chromosomes.
It appears in the transition 2 parallel chromosome systems are going on thereby providing a source to integrate foreign dna. by way of spleen?
Not sure, will follow your lead here.
More on the trebaculae: found in other areas than spleen, but, spleen seems to be center.
========================== = A trabecula (plural trabeculae. From Latin for "small beam.") is a small, often microscopic, tissue element in the form of a small beam, strut or rod, generally having a mechanical function, and usually but not necessarily composed of dense collagenous tissue.
On histological section, a trabecula can look like a septum, but in three dimensions they are topologically distinct, with trabeculae being roughly rod or pillar-shaped and septa being sheet-like. Trabeculae are usually composed of dense fibrous tissue, i.e. mainly of collagen, and in most cases provide mechanical strengthening or stiffening to a soft solid organ, such as the spleen. They can be composed of other materials, such as bone or muscle.When crossing fluid-filled spaces, trabeculae may have the function of resisting tension (as in the flipper) or providing a cell filter (as in the eye.)
Multiple perforations in a septum may reduce it to a collection of trabeculae, as happens to the walls of some of the pulmonary alveoli in emphysema.
========== Examples of trabeculae * trabeculae of bone * trabeculae of corpora cavernosa of penis * trabeculae of corpus spongiosum of penis * trabecular meshwork of the eye * trabeculae of spleen * trabeculae carneae * septomarginal trabecula * cardiac trabeculae en.wikipedia.org/wiki/TrabeculaSkyship
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Post by skyship on Dec 10, 2010 12:37:42 GMT -5
Chromatin? beaded chromatin fibers (the beads, representing nucleosomes)? Heteroduplex? Carassius auratus?from your list Kammy.The oligonucleiotide is the substituting nucleosome, I believe, in the histone 3 and 4 changes, that is where the beads (nuclei) have been substituted.
This brings in Lil sissy's S phase, where cell is altered through the 4 stages. THAT IS WHERE SYNTHESIS takes place.
G1, G2, S phase. will see if can find the complete form:
==== en.wikipedia.org/wiki/File:Cell_Cycle_2.svg======== Phases The cell cycle consists of four distinct phases: G1 phase, S phase (synthesis), G2 phase (collectively known as interphase) and M phase (mitosis). M phase is itself composed of two tightly coupled processes: mitosis, in which the cell's chromosomes are divided between the two daughter cells, and cytokinesis, in which the cell's cytoplasm divides in half forming distinct cells. Activation of each phase is dependent on the proper progression and completion of the previous one. Cells that have temporarily or reversibly stopped dividing are said to have entered a state of quiescence called G0 phase.en.wikipedia.org/wiki/Cell_cycle========= DNA replication occurs at the S phase.
Skyship
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