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Post by skyship on Feb 2, 2010 17:59:19 GMT -5
What is Calx, why important to Morgellons? ============== Calx is a residual substance, sometimes in the form of a fine powder, that is left when a metal or mineral combusts or is calcinated due to heat. Calx, especially of a metal, is now known as an oxide. According to the obsolete phlogiston theory, the calx was the true elemental substance, having lost its phlogiston in the process of combustion. "Calx" is also sometimes used in older texts on artist's techniques to mean calcium oxide. Etymology Calx is the Latin for heel: the tarsus bone. Calx is also the Latin base for lime, calcium and to tread. 2008, Oxford Pocket Dictionary, Oxford University Press: Oxford, UK.. In popular culture * UK Electronic music artist, Aphex Twin (Richard David James) named some of his tracks after different coloured Calx (green, yellow and blue). en.wikipedia.org/wiki/CalxWhat is Phlogiston? The phlogiston theory (from the Ancient Greek φλογιστόν phlŏgistón "burning up", from φλόξ phlóx "fire"), first stated in 1667 by Johann Joachim Becher, is a defunct scientific theory that posited the existence of a fire-like element called "phlogoism" that was contained within combustible bodies, and released during combustion. The theory was an attempt to explain processes such as combustion and the rusting of metals, which are now understood as oxidation. en.wikipedia.org/wiki/Phlogiston_theoryHistory In 1667, Johann Joachim Becher published his Physical Education, which was the first mention of what would become the phlogiston theory. Traditionally, alchemists considered that there were four classical elements: fire, water, air, and earth. In his book, Becher eliminated fire and air from the classical element model and replaced them with three forms of earth: terra lapidea, terra fluida, and terra pinguis.[1][2] Terra pinguis was the element which imparted oily, sulphurous, or combustible properties.[3] Becher believed that terra pinguis was a key feature of combustion and was released when combustible substances were burned.[1] In 1703 Georg Ernst Stahl, professor of medicine and chemistry at Halle, proposed a variant of the theory in which he renamed Becher's terra pinguis to phlogiston, and it was in this form that the theory probably had its greatest influence.[4] skyship
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Post by skyship on Feb 2, 2010 19:38:22 GMT -5
terra lapidea, terra fluida, and terra pinguis
terra meraculis ?
skyship
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Post by skyship on Feb 2, 2010 19:55:30 GMT -5
Theory
The theory holds that all flammable materials contain phlogiston, a substance without color, odor, taste, or mass that is liberated in burning. Once burned, the "dephlogisticated" substance was held to be in its "true" form, the calx.
"Phlogisticated" substances are those that contain phlogiston and are "dephlogisticated" when burned; "in general, substances that burned in air were said to be rich in phlogiston; the fact that combustion soon ceased in an enclosed space was taken as clear-cut evidence that air had the capacity to absorb only a definite amount of phlogiston. When air had become completely phlogisticated it would no longer serve to support combustion of any material, nor would a metal heated in it yield a calx; nor could phlogisticated air support life, for the role of air in respiration was to remove the phlogiston from the body."[5] Thus, phlogiston as first conceived was a sort of anti-oxygen.
Joseph Black's student Daniel Rutherford discovered nitrogen in 1772 and the pair used the theory to explain his results. The residue of air left after burning, in fact a mixture of nitrogen and carbon dioxide, was sometimes referred to as "phlogisticated air", having taken up all of the phlogiston. Conversely, when oxygen was first discovered it was thought to be "dephlogisticated air", capable of combining with more phlogiston and thus supporting combustion for longer than ordinary air.[6]
skyship
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Post by skyship on Feb 2, 2010 21:40:45 GMT -5
Very interesting: Other significant scientific discoveries. Continuing his studies of the atmosphere, Priestley discovered ammonia (alkaline air), sulfur dioxide (vitriolic acid air), silicon tetrafluoride (fluor acid air), nitrogen (also discovered by Daniel Rutherford in 1772), and a gas later identified as carbon monoxide. Of striking significance was his observation that light was important for plant growth and that green plants gave off "dephlogisticated air" (really oxygen). These observations became fundamental to the systematic work on photosynthesis by the Dutch physician Jan Ingenhousz (begun in 1779) and by the Swiss clergyman-naturalist Jean Senebier during the next two decades. Drawing then on his earlier knowledge of electricity, Priestley decomposed ammonia by sparking with electric sparks and noticed the formation of dew when mixtures of hydrogen and oxygen are exploded. In 1779, for reasons not entirely clear, Priestley left Shelburne's employ and settled in the industrial town of Birmingham as minister of the New Meeting congregation. He received a small annuity from Shelburne, and friends raised a subscription to defray the expenses of his experiments. The years at Birmingham were the happiest and busiest of his life. He wrote books on religion and theology and published sermons, tracts, and catechisms. In his History of the Corruptions of Christianity (1782), he rejected most of the fundamental doctrines of Christianity, including the Trinity, predestination, and the divine verbal inspiration of the Bible and traced them to their historical sources of error. This work aroused another storm of protest. By the time of the French Revolution he had acquired a reputation as the antagonist of all establishments, both political and religious. Priestley regularly took part in meetings of Birmingham's Lunar Society, which met monthly at the full moon. Flourishing independently of the Royal Society of London, this organization in 1766-91 actively promoted science and its applications to industry and crafts. There, Priestley was the honoured colleague of the naturalist Erasmus Darwin, the pottery manufacturer Josiah Wedgwood, and the inventor of the steam engine, James Watt. Concluding his scientific work, he observed that "calces" (oxides) are changed to the metallic state (reduced) when heated in hydrogen, but he did not notice that water is also produced, and he gave a phlogistic interpretation. During these years, Priestley was widely known as the defender of the principles of the French Revolution and an ardent advocate of civil and religious liberty. He angered the antirevolutionary populace by publicly disagreeing with the Reflections on the Revolution in France (1790), written by the British statesman Edmund Burke, who opposed the Revolution. On July 14, 1791, on the second anniversary of the fall of the Bastille prison in Paris, an outbreak of mob violence occurred in Birmingham, in the course of which Priestley's house, library, and laboratory were destroyed. He was driven from Birmingham, never to return. For the next two years Priestley resided at Hackney, near London, where he taught at the New College (Hackney College); but the progress of the French Revolution, the execution of Louis XVI in 1793, and the declaration of war against France the same year excited further rancour against him. His three sons had emigrated to the United States in August 1793; Priestley and his wife followed in April 1794, taking up residence in Northumberland, Pa. He was welcomed by various learned societies, including the American Philosophical Society, and offered the chair of chemistry at the University of Pennsylvania, but he refused to accept any public office. During his years in America, Priestley continued his literary and religious activities, but he was hampered in his scientific work by the difficulties of communicating with his friends in England. He published in the United States the last four volumes of his six-volume A General History of the Christian Church (vol. 1-2, 1790-1803, and vol. 3-6, 1802-03). He was a friend and correspondent of two leaders of the American Revolution, John Adams and Thomas Jefferson, both of whom held views on religion similar to his own. The Doctrines of Heathen Philosophy Compared with Those of Revelation appeared after his death. It had been written at the suggestion of Jefferson, who felt, however, that "it did not do justice to the undertaking," adding that Priestley "felt himself pressed by the hand of death." He died at Northumberland in 1804. (S.R.) en.wikipedia.org/wiki/The_Doors_of_Perception
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Post by skyship on Feb 2, 2010 22:29:57 GMT -5
Something about the Lunar Society and the Philisophical Societies in both England and US in the way back machine.
anyway it was Priestly link to E. darwin and Wedgewood that has me intrigued.
and that is when plastics seems to come into play.
So what would phlogistics have to do with plastics?=============================== Macrophages contribute to both initiation and resolution of inflammatory processes. These cells of diverse functional activity participate as both effectors and affectors in host tissue development, homeostasis and response to injury. Survival and function of the macrophage is dependent on colony stimulating factors granulocyte colony stimulating factor (GM-CSF) and macrophage colony stimulating factor (M-CSF). These colony stimulating factors play a significant role in defining the phenotype and activity of macrophages through regulating the expression of transcription factors PU.1 and peroxisome proliferator activator receptor gamma (PPAR?). Recent data suggests that alveolar macrophages are not passive participants in immune responses and that the status of “macrophage activation” defines the phenotype of the ensuing innate and adaptive responses. The status of alveolar macrophage activation, including cytokine production, phagocytosis, and antigen presentation, orchestrates the intensity and duration of the immune response. www.bentham.org/ebooks/9781608050208/contents.htmj=================== PU 1 and peroxisome proliferator activator receptor gamma (PPAR
focusing on the PU.1, seems a spleen virus was contrived:======================= SPLEEN FOCUS FORMING VIRUS PROVIRAL INTEGRATION ONCOGENE SPI1; SPI1Alternative titles; symbols ONCOGENE SPI1SFFV VIRUS-INDUCED MURINE ERYTHROLEUKEMIA ONCOGENE, MOUSE, HOMOLOG OF HEMATOPOIETIC TRANSCRIPTION FACTOR PU.1Gene map locus 11p11.2TEXT CLONING Moreau-Gachelin et al. (1988) reported the characterization of a putative oncogene isolated from a murine erythroleukemia induced by the acute leukemogenic retrovirus spleen focus forming virus (SFFV). The genomic locus was called Spi1, for SFFV proviral integration. Rearrangements due to SFFV integration were found in 95% of the erythroid tumors studied. A 4.0-kb mRNA was detected in rearranged tumors. The evolutionary conservation of the SPI1 locus was examined in cellular DNAs from various vertebrate species. EcoRI fragments that hybridized with a probe from the locus were present in DNA digests from birds and several mammals, including man. 30 PubMed Neighbors Ray et al. (1990) reported that the SPI1 gene encodes a 216-amino acid protein showing 85% identity with the murine counterpart. GENE STRUCTURE Ray et al. (1990) identified 5 exons in the SPI1 gene.MAPPING Van Cong et al. (1989, 1990) mapped the SPI1 oncogene to chromosome 11 by somatic cell hybrid analysis and regionalized it to 11p11.22 by in situ hybridization. GENE FUNCTION Spi1 (and Sfpi1) is also known as PU.1. It is an ETS-domain transcription factor essential for the development of myeloid and B-lymphoid cells. In myeloid cells, PU.1 is thought to regulate transcription of both FMS (164770) and CD18/11B (600065) proteins that are central to the osteoclast phenotype. The similarities between macrophages and osteoclasts, and the critical role PU.1 plays in generating myeloid cells (which are osteoclast progenitors), suggested to Tondravi et al (1997) that failure to express PU.1 might preclude development of osteoclasts, leading to arrested bone resorption and osteopetrosis. The investigators showed that PU.1 mRNA progressively increases as marrow macrophages assume the osteoclast phenotype in vitro. The association between PU.1 and osteoclast differentiation was confirmed by demonstrating that PU.1 expression increased with the induction of osteoclastogenesis by either 1,25-dihydroxyvitamin D3 or dexamethasone. 30 PubMed Neighbors www.ncbi.nlm.nih.gov/entrez/dispomim.cgi?id=165170==================== Direct interaction of hematopoietic transcription factors PU.1 and GATA-1: functional antagonism in erythroid cellsMalignant transformation usually inhibits terminal cell differentiation but the precise mechanisms involved are not understood. PU.1 is a hematopoietic-specific Ets family transcription factor that is required for development of some lymphoid and myeloid lineages. genesdev.cshlp.org/content/13/11/1398.full======================= functional antagonism in erythroid cells:..............wouldn't that be our blood cells??ohhhhhhhhhhhhhthank you thank you COLD SPRING HARBOR!
CHanging our blood without our knowledge, what did they do first use the phlogistic theory, burn those old ancient suckers, so you can infiltrate those plastic pieces of junk?
making O blood are we?
======================= so subtle, so cold!' more later. skyship
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Post by skyship on Feb 2, 2010 22:51:06 GMT -5
myeloid cells (which are osteoclast progenitors) WOESE found his progenitor didn't he? Progenitor: Main Entry: pro·gen·i·tor Pronunciation: \prō-ˈje-nə-tər, prə-\ Function: noun Etymology: Middle English, from Anglo-French progenitour, from Latin progenitor, from progignere to beget, from pro- forth + gignere to beget — more at kin Date: 14th century 1 a : an ancestor in the direct line : forefather b : a biologically ancestral form 2 : precursor, originator <progenitors of socialist ideas — Times Literary Supplement> <progenitor cells> www.merriam-webster.com/dictionary/progenitorcheck out link to Woese from Carnicom's link. the feb 2010 reference: skyship
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Post by skyship on Feb 2, 2010 23:04:09 GMT -5
PU.1 is a hematopoietic-specific Ets family transcription factor that is required for development of some lymphoid and myeloid lineages. ets family transcription factor? What the h............ is Lineage switch?///////////////lkjofjoperovm alsjf...........lsjdlflel....... ====================== Lineage switch induced by overexpression of Ets family transcription factor PU.1 in murine erythroleukemia cell PU.1 is an Ets family transcription factor essential for myelomonocyte and B-cell development. We previously showed that overexpression of PU.1 in murine erythroleukemia (MEL) cells inhibits growth and erythroid differentiation and induces apoptosis of the cells. In an effort to identify target genes of PU.1 concerning these phenomena by using a messenger RNA differential display strategy, we found that some myeloid-specific and lymphoid-specific genes, such as the osteopontin gene, are transcriptionally up-regulated in MEL cells after overexpression of PU.1. bloodjournal.hematologylibrary.org/cgi/content/short/97/8/2300---------------------- tumors in the old blood cells, so incoming progenitors take over. Lets see what Woese has to say about that?................... ------------------ used Salmonella, Brucella and Agrobacterium......................... ----------------- mic.sgmjournals.org/cgi/reprint/138/1/199.pdfskyship IT IS THE MEL 18 that changes the A and B blood lines to O............................ changes the antigen on the A and B............ So used phlogistic theory to burst the old blood cells?
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Post by skyship on Feb 2, 2010 23:32:11 GMT -5
Phlogistics and artificial blood cell progenitors Oh yeah............Mr. Molecule himself knew nothing of this? the stuff we have don't match, not perfect enough, heh? ========================= 2.1.1 Hemoglobin Formulations When tetrameric hemoglobin is freed from the red cell it loses effectiveness in three ways. First, it dissociates to dimers that are rapidly cleared from circulation by the mononuclear phagocytic system (10-30 minute half life) and by the kidneys (1 hour half life). Second, it binds O2 more tightly, reducing deliverability of O2 during tissue hypoxia. Third, during storage, hemoglobin may be oxidized to useless methemoglobin due to the absence of the protective enzyme methemoglobin reductase normally present in red cells. Efforts to modify hemoglobin to increase intravascular dwell time have followed many pathways. Hemoglobin (in solution) has been cross-linked (either internally or with a macromolecule), polymerized, modified by recombinant DNA techniques, or microencapsulated. Encapsulation is most promising, given that all vertebrate hemoglobin is contained in cells to maintain its stability, preserve function, and protect the host from toxicity. Chang [24] reported making synthetic lecithin-cholesterol microcapsules containing hemoglobin as early as 1957. Hunt [25] reported liposome encapsulation of hemoglobin in 1983; his 0.1-1.5 micron "neohemocytes" proved nontoxic to rat kidneys of rats but were cleared from circulation with a half-life of 5.8 hours. Liposome-encapsulated hemoglobin (0.4-micron microspheres) can be stored dry up to 3 months without loss of function [26], and can sustain life in rats transfused isovolemically down to red cell hematocrits of only 4% [27]. Taking a different approach, Suslick used ultrasound to weld roughly 1 million hemoglobin molecules into 2-micron hollow-core microspheres that carry 50% more oxygen per unit volume than natural red cells; the product, which remains 80% effective after 6 months' storage, is in preliminary animal and human trials [28]. www.foresight.org/nanomedicine/Respirocytes.htmlmore later: skyship
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