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Post by skyship on Mar 25, 2010 1:34:03 GMT -5
It appears that Karl Marx the man who believed Communism was the answer to everything, seemed to suffer from a skin disease. Sounds like Morgellon's to me! "Summary From an analysis of the original correspondence, it has been possible to establish that Karl Marx’s incapacitating skin disease was hidradenitis suppurativa, not ‘boils’ as was universally assumed at the time and since; the psychological effect of this illness on the man and his work appears to have been considerable."... a quote from a biography: inside this link: .."" Marx’s condition fits neatly into traditional Chinese medical diagnosis, which takes account of emotional as well as physical states. From this perspective Marx stole fire from the heavens and could not handle the energy. Marx’s s igns and symptoms are of classic liver/fire ones. The rambling speech, insomnia, mental illness, hot head, boils and gastric fever, sore throat, infected mouth and so forth (described below) are consistent with excess heat/fire. In Chinese medicine fire comes from the liver; the headaches, distension in the right upper liver, red eyes and so on, aggravated in the spring, all contribute to that picture. It seems he was genetically disposed to this sort of illness, but his condition was definitely exacerbated by an unhealthy lifestyle, by smoking, drinking and overwork.""...... more here: Note the similarities assets0.pubget.com/pdf/17986303.pdfskyship
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Post by skyship on Mar 25, 2010 2:19:51 GMT -5
I am thinking heat shock protein issues? "Prokaryotic and Eukaryotic Heat Shock Proteins in Infectious Disease Summary:Publisher: Springer | ISBN: 9048129753 | edition 2009 | PDF | 316 pages | 2,9 mb Prokaryotic and Eukaryotic Heat Shock Proteins in Infectious Disease provides the most current review of the literature relating to the role and influence of heat shock (stress) proteins on the establishment, progression and resolution of infectious disease. Written by leaders in the field of heat shock proteins (HSP) and their biological and immunological properties, the contributors provide a fascinating insight into the complex relationship between, and the involvement of prokaryotic and eukaryotic HSP in disease states. It has been known for some considerable time that heat shock proteins from prokaryotic organisms are immunodominant molecules that are intimately involved in the induction of potential protective inflammatory responses, and this aspect of HSP biology is updated herein. In addition to regulating heat shock protein gene expression, the t ranscription factor HSF1 also appears to play an important role in regulating immune responses to infection. Heat shock proteins are now known to influence infectious disease processes in a number of diverse ways: they are involved in the propagation of prions, the replication and morphogenesis of viruses, and the resistance of parasites to chemotherapy. These proteins also appear to be important mediators of bacteria-host interactions and inflammation, the latter via interactions with cell surface molecules and structures such as Toll-like receptors and lipid rafts. Heat shock proteins can be expressed on the surface of infected cells, and this is likely to provide a target for the innate immune response. Elevated levels of circulating HSP are present in infectious diseases and these proteins might therefore regulate inflammatory responses to pathogenic challenge on a systemic basis. Heat shock proteins are also implicated in the impact of genital tract infections on the reproductive outcome, as well as in the local and systemic consequences of periodontal disease."bit.ly/b2lcmm========================= aqt,,,,,,,,,,,,,,,,here seems to be the succession of it. The things we have discussed! But, what if they are not working?
a summary: will bold the things that may pertain:
============================ The anti-sigma factors. KT Hughes, K Mathee A mechanism for regulating gene expression at the level of transcription utilizes an antagonist of the sigma transcription factor k nown as the anti-sigma (anti-sigma) factor. The cytoplasmic class of anti-sigma factors has been well characterized. The class includes AsiA form bacteriophage T4, which inhibits Escherichia coli sigma 70; FlgM, present in both gram-positive and gram-negative bacteria, which inhibits the flagella sigma factor sigma 28; SpoIIAB, which inhibits the sporulation-specific sigma factor, sigma F and sigma G, of Bacillus subtilis; RbsW of B. subtilis, which inhibits stress response sigma factor sigma B; and DnaK, a general regulator of the heat shock response, which in bacteria inhibits the heat shock sigma factor sigma 32. In addition to this class of well-characterized cytoplasmic anti-sigma factors, a new class of homologous, inner-membrane-bound anti-sigma factors has recently been discovered in a variety of eubacteria. This new class of anti-sigma factors regulates the expression of so-called extracytoplasmic functions, and hence is known as the ECF subfamily of anti-sigma factors. The range of cell processes regulated by anti-sigma factors is highly varied and includes bacteriophage phage growth, sporulation, stress response, flagellar biosynthesis, pigment production, ion transport, and virulence.www.heatshock.net/showabstract.php?pmid=9891799skyship
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Post by skyship on Mar 25, 2010 3:15:32 GMT -5
AsiA form bacteriophage T4
Escherichia coli sigma 70
FlgM, present in both gram-positive and gram-negative bacteria,
flagella sigma factor sigma 28; SpoIIAB
sporulation-specific sigma factor, sigma F and sigma G, of Bacillus subtilis;
RbsW of B. subtilis
stress response sigma factor sigma B
DnaK
heat shock sigma factor sigma 32
ECF subfamily of anti-sigma factors
bacteriophage phage growth, sporulation, stress response, flagellar biosynthesis, pigment production, ion transport, and virulence.
Okay, I got some work to do.
skyship
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